The Central Nervous System and Behaviour. Selected Translations from the Russian Medical Literature under the joint sponsorship of the Josiah Macy, Jr. Foundation and the National Science Foundation

1961 ◽  
Vol 117 (11) ◽  
pp. 1054-b-1054
Author(s):  
C. B. F.
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
National Science Foundation ◽  
Medical Literature ◽  
National Science ◽  
The Central Nervous System ◽  
Russian Medical

scholarly journals Brachial Plexopathy from Metaldehyde Poisoning – A Case Report

2021 ◽  
Vol 26 (4) ◽  
pp. 839-843
Author(s):  
See-Teng Tan ◽  
Guanglin Chen ◽  
Wai Ching, Deanna Lee ◽  
You-Jiang Tan
Keyword(s):  
Magnetic Resonance Imaging ◽  
Central Nervous System ◽  
Nervous System ◽  
Medical Literature ◽  
Subsequent Development ◽  
Brachial Plexopathy ◽  
Resonance Imaging ◽  
Sensorimotor Deficits ◽  
The Central Nervous System ◽  
The Right

We describe the case of a middle-aged female with schizophrenia, who developed acute sensorimotor deficits of the right upper limb within a week of ingesting large amounts of metaldehyde in a suicide attempt. A right-sided brachial plexopathy was diagnosed clinically and supported by electrophysiologic assessments and targeted magnetic resonance imaging scans. Although metaldehyde’s neurotoxicity typically affects the central nervous system, focal involvement of the peripheral nervous system remains unreported in medical literature, and its pathogenic processes await further elucidation. Therefore, we recommend the continued observation for the subsequent development of sensorimotor deficits during the first week of metaldehyde poisoning.

The Cerebral Associations of Raynaud's Disease

1916 ◽  
Vol 62 (259) ◽  
pp. 730-756 ◽  
Author(s):  
Hubert J. Norman
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Medical Literature ◽  
Clinical Material ◽  
Raynaud's Disease ◽  
Disease Complex ◽  
History Of ◽  
The Central Nervous System ◽  
Raynaud’S Disease

It was in 1862 that Maurice Raynaud first made public his description of the condition with which his name has since been associated, and his Thesis for the Doctorate in Medicine has now become a classic in medical literature (1). Although there may be meticulous critics who will dispute his claim to priority in the delineation of the disease-complex known as Raynaud's disease, there can be no doubt that it was he who first succinctly and clearly described the condition. To use a phrase which is apposite in dealing with such a subject—it was Raynaud who first drew a “line of demarcation” between the symptoms characteristic of his syndrome and those typical of gangrenes in general. In his own words, he was dealing with “a very limited corner of the general history of gangrenes.” In the present paper it is proposed to remain within still stricter limitations, yet even there the amount of clinical material is not inconsiderable. There is an additional interest, too, from the fact that Raynaud, especially in his later researches (2), became more and more convinced that the explanation of the peripheral symptoms should be sought in some change in the central nervous system.

Effects of Hyperoxia on Lung Utrastructure

1970 ◽  
Vol 28 ◽  
pp. 26-27
Author(s):  
Gladys Harrison
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Light Microscopy ◽  
Oxygen Effects ◽  
Age Dependent ◽  
The Central Nervous System ◽  
The Subject ◽  
Space Age ◽  
Alveolar Septa ◽  
Extensive Damage

With the advent of the space age and the need to determine the requirements for a space cabin atmosphere, oxygen effects came into increased importance, even though these effects have been the subject of continuous research for many years. In fact, Priestly initiated oxygen research when in 1775 he published his results of isolating oxygen and described the effects of breathing it on himself and two mice, the only creatures to have had the “privilege” of breathing this “pure air”.Early studies had demonstrated the central nervous system effects at pressures above one atmosphere. Light microscopy revealed extensive damage to the lungs at one atmosphere. These changes which included perivascular and peribronchial edema, focal hemorrhage, rupture of the alveolar septa, and widespread edema, resulted in death of the animal in less than one week. The severity of the symptoms differed between species and was age dependent, with young animals being more resistant.

Emigration of neutrophils in the central nervous system

1983 ◽  
Vol 41 ◽  
pp. 788-789
Author(s):  
John L.Beggs ◽  
John D. Waggener ◽  
Wanda Miller ◽  
Jane Watkins
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Osmium Tetroxide ◽  
White Blood Cells ◽  
Arterial Perfusion ◽  
E Coli ◽  
Intracisternal Injection ◽  
The Central Nervous System ◽  
Brain And Spinal Cord ◽  
Interendothelial Junctions

Studies using mesenteric and ear chamber preparations have shown that interendothelial junctions provide the route for neutrophil emigration during inflammation. The term emigration refers to the passage of white blood cells across the endothelium from the vascular lumen. Although the precise pathway of transendo- thelial emigration in the central nervous system (CNS) has not been resolved, the presence of different physiological and morphological (tight junctions) properties of CNS endothelium may dictate alternate emigration pathways.To study neutrophil emigration in the CNS, we induced meningitis in guinea pigs by intracisternal injection of E. coli bacteria.In this model, leptomeningeal inflammation is well developed by 3 hr. After 3 1/2 hr, animals were sacrificed by arterial perfusion with 3% phosphate buffered glutaraldehyde. Tissues from brain and spinal cord were post-fixed in 1% osmium tetroxide, dehydrated in alcohols and propylene oxide, and embedded in Epon. Thin serial sections were cut with diamond knives and examined in a Philips 300 electron microscope.

Mammalian Bone Marrow Acetylcholinesterase

1982 ◽  
Vol 40 ◽  
pp. 44-45
Author(s):  
Ezzatollah Keyhani
Keyword(s):  
Central Nervous System ◽  
Bone Marrow ◽  
Nervous System ◽  
Common Property ◽  
Extracellular Medium ◽  
The Central Nervous System ◽  
The Common ◽  
Mammalian Bone

Acetylcholinesterase (EC 3.1.1.7) (ACHE) has been localized at cholinergic junctions both in the central nervous system and at the periphery and it functions in neurotransmission. ACHE was also found in other tissues without involvement in neurotransmission, but exhibiting the common property of transporting water and ions. This communication describes intracellular ACHE in mammalian bone marrow and its secretion into the extracellular medium.

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