scholarly journals Can dietary supplements maintain cognition in mid- to late-life?

2019 ◽  
Vol 26 (1) ◽  
pp. 3-7
Author(s):  
Alexander L. W. Smith
Keyword(s):  
Risk Factors ◽  
Dietary Supplements ◽  
Cochrane Review ◽  
Late Life ◽  
Healthy Adults ◽  
Modifiable Risk Factors ◽  
Cognitive Health ◽  
Mineral Supplementation ◽  
Vitamin And Mineral Supplementation

SUMMARYCognitive health, and prevention of its decline to dementia, has risen in prominence with a corresponding exploration of modifiable risk factors to prevent a decline in cognitive health with age. This commentary discusses a new Cochrane review that examines the effect of vitamin and mineral supplementation in maintaining cognitive health in cognitively healthy adults in mid- and late-life. From a heterogeneous body of evidence, the quality of which ranged from very low to moderate, the review draws the conclusions of little or no benefit of supplements.

scholarly journals Vitamin and mineral supplementation for maintaining cognitive function in cognitively healthy people in mid and late life: a Cochrane Review

2019 ◽  
Vol 26 (1) ◽  
pp. 2-2
Author(s):  
Anne W. S. Rutjes ◽  
David A. Denton ◽  
Marcello Di Nisio ◽  
Lee-Yee Chong ◽  
Rajesh P. Abraham ◽  
...  
Keyword(s):  
Cognitive Function ◽  
Cochrane Review ◽  
Late Life ◽  
Healthy People ◽  
Mineral Supplementation ◽  
Vitamin And Mineral Supplementation

scholarly journals Modifiable Risk Factors for Prevention of Dementia in Midlife, Late Life and the Oldest-Old: Validation of the LIBRA Index

2017 ◽  
Vol 58 (2) ◽  
pp. 537-547 ◽  
Author(s):  
Stephanie J.B. Vos ◽  
Martin P.J. van Boxtel ◽  
Olga J.G. Schiepers ◽  
Kay Deckers ◽  
Marjolein de Vugt ◽  
...  
Keyword(s):  
Risk Factors ◽  
Oldest Old ◽  
Late Life ◽  
Modifiable Risk Factors

scholarly journals Association of sex differences in dementia risk factors with sex differences in memory decline in a population-based cohort spanning 20–76 years

2021 ◽  
Vol 11 (1) ◽  
Author(s):  
Kaarin J. Anstey ◽  
Ruth Peters ◽  
Moyra E. Mortby ◽  
Kim M. Kiely ◽  
Ranmalee Eramudugolla ◽  
...  
Keyword(s):  
Risk Factors ◽  
Sex Differences ◽  
Older Women ◽  
Late Life ◽  
Population Based ◽  
Modifiable Risk Factors ◽  
Vascular Risk ◽  
Memory Decline ◽  
Dementia Risk ◽  
With Memory

AbstractSex differences in late-life memory decline may be explained by sex differences in dementia risk factors. Episodic memory and dementia risk factors were assessed in young, middle-aged and older adults over 12 years in a population-based sample (N = 7485). For men in midlife and old age, physical, cognitive and social activities were associated with less memory decline, and financial hardship was associated with more. APOE e4 and vascular risk factors were associated with memory decline for women in midlife. Depression, cognitive and physical activity were associated with memory change in older women. Incident midlife hypertension (β = − 0.48, 95% CI − 0.87, − 0.09, p = 0.02) was associated with greater memory decline in women and incident late-life stroke accounted for greater memory decline in men (β = − 0.56, 95% CI − 1.12, − 0.01), p = 0.05). Women have fewer modifiable risk factors than men. Stroke and hypertension explained sex differences in memory decline for men and women respectively.

scholarly journals Modifiable risk factors of vasospastic angina: a systematic literature review

2021 ◽  
Vol 8 (3) ◽  
pp. 1481
Author(s):  
Abdulmajeed F. Albalawi ◽  
Hamoud T. Alotaibi ◽  
Razan K. Alharbi ◽  
Abdulrahman A. Alghamdi ◽  
Ali A. Alzahrani ◽  
...  
Keyword(s):  
Risk Factors ◽  
Coronary Artery ◽  
Medical Personnel ◽  
Coronary Vasospasm ◽  
Vasospastic Angina ◽  
Modifiable Risk Factors ◽  
Lifestyle Modifications ◽  
Medical Disorders ◽  
Underlying Pathophysiology

Vasospastic angina is caused by transient coronary spasms unrelated to exertion, which may even occur at rest (classically at night) and promptly responds to short acting nitrates. It is thought to be caused by inherent generalised hyper reactivity of the smooth muscle cells of epicardial vessels to various stimuli. Being comparatively less studied, the risk factors for VSA vary considerably from obstructive or atherosclerotic cardiac disease. However, coronary vasospasm can occur in non stenosed arteries, atherosclerotic arteries as well as sub critically stenosed arteries which can result in significant overlap between the risk factors and underlying pathophysiology. 1-14% of AMI can be caused by non-obstructive, or ‘functional’ coronary artery disorders like vasospastic angina but VSA continues to be underdiagnosed and less well understood than obstructive coronary artery disorders. This prompted us to study the existing literature for modifiable risk factors of coronary artery vasospasm so that an emphasis can be made on proper lifestyle modifications and avoidance of vasospastic agents in susceptible individuals. Smoking proved to be the most important risk factor whereas recreational drugs and drugs used for treatment of certain medical disorders have also been shown to associate with coronary vasospasm. Medical personnel, therefore, need to be more vigilant in history taking as well as investigating cardiac chest pain in which traditional investigations end up being normal so that an early diagnosis of vasospastic angina can be made and appropriate steps taken to improve the quality of life of patients.

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