scholarly journals Structural Brain Lesions in Functional Psychosis

1987 ◽  
Vol 151 (3) ◽  
pp. 420-420
Author(s):  
Sunar Birsoz ◽  
Ali Ihsan Baysal
2020 ◽  
Vol 14 ◽  
Author(s):  
Eirini Papageorgiou ◽  
Nathalie De Beukelaer ◽  
Cristina Simon-Martinez ◽  
Lisa Mailleux ◽  
Anja Van Campenhout ◽  
...  

2013 ◽  
Vol 167 (2) ◽  
pp. 328-334 ◽  
Author(s):  
Aristeidis H. Katsanos ◽  
Panagiotis Korantzopoulos ◽  
Georgios Tsivgoulis ◽  
Athanassios P. Kyritsis ◽  
Maria Kosmidou ◽  
...  

2019 ◽  
Vol 9 (1) ◽  
Author(s):  
Benjamin Rohaut ◽  
Kevin W. Doyle ◽  
Alexandra S. Reynolds ◽  
Kay Igwe ◽  
Caroline Couch ◽  
...  

BMJ Open ◽  
2018 ◽  
Vol 8 (3) ◽  
pp. e019016 ◽  
Author(s):  
Tobias Braun ◽  
Martin Juenemann ◽  
Maxime Viard ◽  
Marco Meyer ◽  
Sven Fuest ◽  
...  

ObjectivesFibre-endoscopic evaluation of swallowing (FEES) to detect dysphagia is gaining more and more importance as a diagnostic tool. Therefore, we have investigated the impact of FEES in neurological patients in a clinical setting.DesignCross-sectional hospital-based registry.SettingPrimary acute care in a neurological department of a German university hospital.Participants241patients with various neurological diseases who underwent FEES procedure.Primary and secondary outcome measuresDysphagia and related comorbidities.Results267 FEES were performed in 241 patients with various neurological diagnoses. Dysphagia was diagnosed in 68.9% of the patients. In only 33.1% of the patients, appropriate oral diet was chosen prior to FEES. A relevant dysphagia occurred more often in patients with structural brain lesions (83.1% vs 65.3%, P=0.001), patients with dysphagia had a longer hospitalisation (median 18 (IQR 12–30) vs 15 days (IQR 9.75–22.75), P=0.005) and had a higher mortality (8.4% vs 1.3%, P=0.041). When the oral diet was changed, we observed a lower pneumonia rate (36% vs 50%, P=0.051) and a lower mortality (3.7% vs 11.3%, P=0.043) in comparison to no change of oral diet. A restriction of oral diet was identified more often in older patients (median 75 years (IQR 66.3–82 years) vs median 72 years (IQR 60–79 years), P=0.01) and in patients with structural brain lesions (86.8% vs 73.1%, P=0.05).ConclusionOn clinical investigation, dysphagia was misjudged for the majority of the patients. FEES might help to compensate this drawback, revising the diet regime in nearly 70% of the patients.


Author(s):  
Vladimir S. Kostià ◽  
Marina Stojanovié-Svetel ◽  
Aleksandra Kacar

ABSTRACT:Background:Symptomatic (secondary) dystonias associated isolated lesions in the brain provide insight into etiopathogenesis of the idiopathic form of dystonia and are a basis for establishing the possible correlation between the anatomy of a lesion and the type of dystonia according to muscles affected.Methods:In 358 patients with differently distributed dystonias, a group of 16 patients (4.5%) was encountered in whom dystonia was associated with focal brain lesions.Results:Of the 16 patients, 3 patients had generalized, 3 segmental and 4 hemidystonia, while the remaining 6 patients had focal dystonia. The most frequent etiologies were infarction in 7, and tumor in 4 patients. These lesions were usually found in the lenticular and caudate nucleus, thalamus, and in the case of blepharospasm in the upper brainstem.Conclusions:Our results support the suggestion that dystonia is caused by a dysfunction of the basal ganglia.


2016 ◽  
Vol 6 (1) ◽  
pp. 51 ◽  
Author(s):  
AaronMatthew McMurtray ◽  
ErinK Saito ◽  
Meera Nagpal ◽  
Amanda Leon ◽  
Bijal Mehta

Author(s):  
Michael Drees ◽  
Neil Kulkarni ◽  
Jorge Vidaurre

AbstractElectrical status epilepticus during sleep (ESES) is an age-related, self-limited epileptic encephalopathy characterized by heterogeneous clinical manifestations and a specific electroencephalographic pattern of continuous spikes and waves during slow sleep. The etiology of ESES is not completely clear, although structural brain lesions, abnormal immunological markers, and genetic mutations have been associated with the syndrome. ESES was first described in 1971 and since then, the diagnostic criteria have changed multiple times. Additionally, inconsistency between authors in how to record and evaluate the electroencephalogram also leads to variability between studies. These inconsistencies hamper objectivity, comparison, and generalization. Because of this, one of the first priorities of physicians treating this condition should be defining the parameters of this disease so that cooperative building can occur.


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