scholarly journals PATHOGENESIS OF PARAINFLUENZA VIRUS (PV)-INDUCED CROUP: ROLE OF DEFICIENT REGULATION OF CELL-MEDIATED IMMUNE RESPONSE AND IGE PRODUCTION

1984 ◽  
Vol 18 ◽  
pp. 267A-267A
Author(s):  
R Welliver ◽  
M Sun ◽  
D Losi
2012 ◽  
Vol 30 (6) ◽  
pp. 500-504 ◽  
Author(s):  
Deepak Kaul ◽  
M. Sasikala ◽  
A. Raina

1998 ◽  
Vol 66 (2) ◽  
pp. 397-402 ◽  
Author(s):  
S. M. Rhind ◽  
H. W. Reid ◽  
S. R. McMillen ◽  
G. Palmarini

AbstractThe relationship between weaning stress-induced changes in stress hormone profiles and immune function was investigated in groups of 10 lambs immunized against adrenocorticotrophic hormone (ACTH; treatment A) or fi-endorphin (treatment B) to reduce the circulating concentrations of cortisol and fi-endorphin respectively. Control animals (treatment C) were immunized against a porcine thyroglobulin carrier protein. Application of weaning stress was associated with significantly elevated plasma cortisol concentrations but no significant increase in fi-endorphin concentrations in C lambs. Immunization against ACTH suppressed the post-weaning increase in cortisol concentration. This was associated with a transient reduction in the lymphocyte stimulation response to keyhole limpet haemocyanin (KLH) antigen in the A animals but there was no effect on the antibody response or interferon-y production by antigen stimulated lymphocytes. There were no significant effects of immunization against fi-endorphin on the capacity to mount antibody or cell-mediated immune responses. It is concluded that weaning stress-induced increases in cortisol did not inhibit the immune response. Since cortisol concentrations and the cell mediated immune response at 8 days after immunization were positively associated it is concluded that these indices are not independent measures of stress.


1995 ◽  
Vol 121 (S1) ◽  
pp. S15-S15
Author(s):  
C. Ferrari ◽  
A. Penna ◽  
A. Bertoletti ◽  
A. Cavalli ◽  
G. Missale ◽  
...  

1980 ◽  
Vol 17 (1) ◽  
pp. 31-37 ◽  
Author(s):  
Steven I. Wolinsky ◽  
James S. Goodwin ◽  
Ronald P. Messner ◽  
Ralph C. Williams

1974 ◽  
Vol 19 (2) ◽  
pp. 77-80 ◽  
Author(s):  
D. M. Weir

The role of chemical messengers is considered in relation to the initiation and amplification of the immune response. The roles of pre-existing tissue factors of the complement, clotting and Kinin systems, are considered and the additional effects of the chemical mediators produced by activated lymphocytes and tissue breakdown products are described. The final expression of the immune response appears to depend on complex inter-relationships between these factors and the cells of the lymphoid tissues. Understanding of their actions might be expected to be of value in manipulating the cell-mediated immune response in various disease states.


Sign in / Sign up

Export Citation Format

Share Document