scholarly journals Brainstem auditory evoked potentials in children with microcephaly

2016 ◽  
Vol 43 (1) ◽  
pp. 28
Author(s):  
Irawan Mangunatmadja ◽  
Dwi Putro Widodo ◽  
Hardiono D Pusponegoro
Keyword(s):  
Hearing Loss ◽  
Evoked Potentials ◽  
Auditory Evoked Potentials ◽  
Clinical Manifestations ◽  
Auditory Brainstem ◽  
Brainstem Auditory Evoked Potentials ◽  
Perinatal Infection ◽  
History Of ◽  
Delayed Development

Background Hearing loss (HL) is commonly found in childrenwith microcephaly. The aim of this study was to reveal hearing lossand auditory brainstem pathways disorders in children with micro-cephaly and other handicaps.Methods There were 194 children who were referred for hearingevaluation. Subjects with history of congenital perinatal infection(TORCH) were excluded. Data were collected from the results ofBrainstem Auditory Evoked Potentials (BAEP) recordings, includ-ing sex, age, clinical manifestations, latency and interlatency be-tween waves I, III, V, and the hearing levels of each ear.Results Moderate to profound HL were found in fourteen ears(58%) of patients with microcephaly. Moderate to profound HL (28%)and endocochlear damage (15%) were found in the ears of pa-tients with microcephaly and delayed speech. Moderate to pro-found HL (39%) and endocochlear damage (11%) were detectedin the ears of patients with microcephaly and delayed develop-ment. Moderate to profound HL (21%) and endocochlear damage(16%) were found in the ears of microcephalic patients with bothdelayed speech and delayed development. Moderate to profoundHL (26%) and endocochlear damage (32%) were detected in theears of patients with microcephaly and cerebral palsy.Conclusion This study revealed the importance of early HL de-tection in microcephalic patients especially those with other handi-caps such as delayed speech, delayed development, and cere-bral palsy

No. 288 Follow-Up of Children With Hearing Loss Diagnosed by Brainstem Auditory Evoked Potentials

PM&R ◽  
2014 ◽  
Vol 6 (8) ◽  
pp. S149
Author(s):  
O. Daniel Páez ◽  
Fernando Ortiz C ◽  
V. Martha ◽  
C. Ortiz ◽  
Fabián J.F. Páez
Keyword(s):  
Hearing Loss ◽  
Evoked Potentials ◽  
Auditory Evoked Potentials ◽  
Brainstem Auditory Evoked Potentials ◽  
Children With Hearing Loss

scholarly journals Microvascular decompression for typical trigeminal neuralgia: Personal experience with intraoperative neuromonitoring with level-specific-CE-Chirp® brainstem auditory evoked potentials in preventing possible hearing loss

2020 ◽  
Vol 11 ◽  
pp. 388
Author(s):  
Luciano Mastronardi ◽  
Franco Caputi ◽  
Guglielmo Cacciotti ◽  
Carlo Giacobbo Scavo ◽  
Raffaelino Roperto ◽  
...  
Keyword(s):  
Hearing Loss ◽  
Trigeminal Neuralgia ◽  
Evoked Potentials ◽  
Posterior Fossa ◽  
Microvascular Decompression ◽  
Auditory Evoked Potentials ◽  
Intraoperative Neuromonitoring ◽  
Brainstem Auditory Evoked Potentials ◽  
Group A ◽  
Group B

Background: Permanent hearing loss after posterior fossa microvascular decompression (MVD) for typical trigeminal neuralgia (TTN) is one of the possible complications of this procedure. Intraoperative brainstem auditory evoked potentials (BAEPs) are used for monitoring the function of cochlear nerve during cerebellopontine angle (CPA) microsurgery. Level-specific (LS)-CE-Chirp® BAEPs are the most recent evolution of classical click BAEP, performed both in clinical studies and during intraoperative neuromonitoring (IONM) of acoustic pathways during several neurosurgical procedures. Methods: Since February 2016, we routinely use LS-CE-Chirp® BAEPs for monitoring the function of cochlear nerve during CPA surgery, including MVD for trigeminal neuralgia. From September 2011 to December 2018, 71 MVDs for TTN were performed in our department, 47 without IONM of acoustic pathways (Group A), and, from February 2016, 24 with LS-CE-Chirp BAEP (Group B). Results: Two patients of Group A developed a permanent ipsilateral anacusia after MVD. In Group B, we did not observe any permanent acoustic deficit after surgery. In one case of Group B, during arachnoid dissection, intraoperative LS-CE-Chirp BAEP showed a temporary lag of V wave, resolved in 5 min after application of intracisternal diluted papaverine (0.3% solution without excipients). Conclusion: MVD is widely considered a definitive surgical procedure in the management of TTN. Even though posterior fossa MVD is a safe procedure, serious complications might occur. In particular, the use of IONM of acoustic pathways during MVD for TTN might contribute to prevention of postoperative hearing loss.

Normal Neonatal Brainstem Auditory Evoked Potentials

1982 ◽  
Vol 91 (5) ◽  
pp. 485-488 ◽  
Author(s):  
Howard H. Zubick ◽  
Marvin P. Fried ◽  
Michael F. Epstein ◽  
Peter Feudo ◽  
Marshall Strome
Keyword(s):  
Hearing Loss ◽  
Evoked Potentials ◽  
Auditory Evoked Potentials ◽  
Large Sample Size ◽  
Brainstem Auditory Evoked Potentials ◽  
American Academy Of Pediatrics ◽  
Sample Data ◽  
Wave Forms ◽  
Normal Neonates ◽  
Total Agreement

Brainstem auditory evoked potentials present a unique opportunity to assess the neuronal and auditory status of the newborn. To date, sample data have been few in number, thereby limiting their interpretive value. The present study was undertaken in an effort to accrue data generated from a large sample size. One hundred fifty-five normal neonates were tested within the first 36 hours of life. No subjects qualified for inclusion in the High Risk Register for Hearing Loss established by the American Academy of Pediatrics. Each ear was tested independently, using click stimuli at a rate of 30.1 per second at 65 dB HTL. Test runs were replicated for purposes of reliability. A run consisted of 2,000 sweeps of data, each being 10 ms in length. Identification of wave forms were assessed by two or more observers. Total agreement was required from all observers for inclusion of wave peaks for analysis. Latencies of onset of stimulus to peak were obtained. Descriptive as well as inferential statistics were computed. Results provide standardized data for comparison.

scholarly journals Age Effects on Neural Representation and Perception of Silence Duration Cues in Speech

2019 ◽  
Vol 62 (4S) ◽  
pp. 1099-1116 ◽  
Author(s):  
Lindsey Roque ◽  
Casey Gaskins ◽  
Sandra Gordon-Salant ◽  
Matthew J. Goupell ◽  
Samira Anderson
Keyword(s):  
Hearing Loss ◽  
Regression Analysis ◽  
Linear Regression ◽  
Evoked Potentials ◽  
Auditory Evoked Potentials ◽  
Linear Regression Analysis ◽  
Auditory Brainstem ◽  
Normal Hearing ◽  
Group Differences ◽  
Cortical Auditory Evoked Potentials

Purpose Degraded temporal processing associated with aging may be a contributing factor to older adults' hearing difficulties, especially in adverse listening environments. This degraded processing may affect the ability to distinguish between words based on temporal duration cues. The current study investigates the effects of aging and hearing loss on cortical and subcortical representation of temporal speech components and on the perception of silent interval duration cues in speech. Method Identification functions for the words DISH and DITCH were obtained on a 7-step continuum of silence duration (0–60 ms) prior to the final fricative in participants who are younger with normal hearing (YNH), older with normal hearing (ONH), and older with hearing impairment (OHI). Frequency-following responses and cortical auditory-evoked potentials were recorded to the 2 end points of the continuum. Auditory brainstem responses to clicks were obtained to verify neural integrity and to compare group differences in auditory nerve function. A multiple linear regression analysis was conducted to determine the peripheral or central factors that contributed to perceptual performance. Results ONH and OHI participants required longer silence durations to identify DITCH than did YNH participants. Frequency-following responses showed reduced phase locking and poorer morphology, and cortical auditory-evoked potentials showed prolonged latencies in ONH and OHI participants compared with YNH participants. No group differences were noted for auditory brainstem response Wave I amplitude or Wave V/I ratio. After accounting for the possible effects of hearing loss, linear regression analysis revealed that both midbrain and cortical processing contributed to the variance in the DISH–DITCH perceptual identification functions. Conclusions These results suggest that age-related deficits in the ability to encode silence duration cues may be a contributing factor in degraded speech perception. In particular, degraded response morphology relates to performance on perceptual tasks based on silence duration contrasts between words.

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