ALCOHOL-INDUCED HYPOGLYCEMIA AND COMA CAUSED BY ALCOHOL SPONGING

PEDIATRICS ◽  
1970 ◽  
Vol 46 (3) ◽  
pp. 445-447
Author(s):  
Malcolm H. Moss
Keyword(s):  
Blood Glucose ◽  
Elevated Temperature ◽  
Ethyl Alcohol ◽  
Alcohol Intoxication ◽  
Severe Hypoglycemia ◽  
Male Infant ◽  
Blood Alcohol ◽  
Acute Alcohol Intoxication ◽  
Tepid Water

The inhalation of ethyl or isopropylalcohol vapor during sponge bathing may result in alcohol intoxication and coma. Ingestion of these alcohols may cause hypoglycemia in children or adults. The present case is the first reported in which alcohol-induced hypoglycemia developed following inhalation of alcohol. A six month-old male infant became comatose following sponge bathing with ethyl alcohol. He was found to have acute alcohol intoxication (blood alcohol 220 mg per 100 ml) and severe hypoglycemia (blood glucose 22 mg per 100 ml). The administration of 50% glucose in water intravenously resulted in prompt recovery. Alcohol sponging to lower elevated temperature is rarely necessary. Cooling with tepid water is effective and considerably less hazardous; the addition of alcohol is not necessary.

THE INFLUENCE OF VITAMIN C ON EOSINOPHIL RESPONSE TO ACUTE ALCOHOL INTOXICATION IN RATS

1960 ◽  
Vol XXXV (IV) ◽  
pp. 585-593 ◽  
Author(s):  
T. P. J. Vanha-Perttula
Keyword(s):  
Vitamin C ◽  
Ethyl Alcohol ◽  
Intraperitoneal Injection ◽  
Alcohol Intoxication ◽  
Albino Rats ◽  
Acute Alcohol Intoxication ◽  
Alcohol Administration ◽  
Initial Values

ABSTRACT The effect of ethyl alcohol on the circulating eosinophil cells has been studied in female albino rats. An intoxicating dose of alcohol caused a marked depletion of circulating eosinophils which was most clearly evident four hours after the administration of the alcohol. The initial values were not reached before 24 hours had elapsed. Intraperitoneal injection of vitamin C 12 hours prior to the alcohol administration very effectively prevented this eosinopenic reaction. The mechanism of regulation of the eosinophil cells in the circulation has been discussed in the light of previous results and of those obtained in this study.

scholarly journals Neuropsychological Profile of Acute Alcohol Intoxication during Ascending and Descending Blood Alcohol Concentrations

2005 ◽  
Vol 31 (6) ◽  
pp. 1301-1309 ◽  
Author(s):  
Tom A Schweizer ◽  
Muriel Vogel-Sprott ◽  
James Danckert ◽  
Eric A Roy ◽  
Amanda Skakum ◽  
...  
Keyword(s):  
Alcohol Intoxication ◽  
Blood Alcohol ◽  
Acute Alcohol Intoxication ◽  
Neuropsychological Profile

Acute Neuropsychiatric Complications of Chronic Alcoholism

1968 ◽  
Vol 114 (506) ◽  
pp. 85-92 ◽  
Author(s):  
H. G. Morgan
Keyword(s):  
Ethyl Alcohol ◽  
Alcohol Intake ◽  
Alcohol Intoxication ◽  
Chronic Alcoholism ◽  
Acute Alcohol Intoxication ◽  
Secondary Complications ◽  
Neuropsychiatric Complications ◽  
Symptoms And Signs

The effects of ethyl alcohol in man may be classified into two main groups. The first, consisting of all those symptoms and signs that can be directly attributed to the action of alcohol itself, includes all the features of acute alcohol intoxication. The second group, with which this study is concerned, consists of a wide variety of conditions which do not appear to result directly from the action of alcohol and which may be regarded as secondary complications. They occur only when excess alcohol intake has occurred over a prolonged period.

Effect of acute alcohol intoxication on steroid output of rat adrenals in vitro

1963 ◽  
Vol 204 (5) ◽  
pp. 849-855 ◽  
Author(s):  
Harold Kalant ◽  
Rosemary D. Hawkins ◽  
Caroline Czaja
Keyword(s):  
Alcohol Intoxication ◽  
Rate Of Change ◽  
Blood Alcohol ◽  
Acute Alcohol Intoxication ◽  
Significant Elevation ◽  
Stomach Tube ◽  
Adrenal Steroid ◽  
Rat Adrenals ◽  
Blood Alcohol Levels

Ethyl alcohol, 2 g/kg in 20% solution, was given to rats by gavage and the adrenals were removed 1 1/2 hr later and incubated in vitro. Their corticosteroid production was not significantly different from that of glands from water-treated or untreated controls. However, when this dose of alcohol was administered by intraperitoneal injection, a significant elevation in adrenal steroid output was observed. Increasing the dose of alcohol to 4 g/kg by stomach tube also evoked an increased adrenocortical response, but when this total dose was divided into two spaced doses, each one of 2 g/kg by stomach tube, 1 1/2 hr apart, no significant alteration in adrenocortical production was observed, despite higher blood alcohol levels and equally deep intoxication. Thus, although the blood alcohol level attained may influence the activity of the adrenal cortex, the rate of change in blood and tissue alcohol levels also seems to be a significant factor in determining whether adrenal cortical stimulation will occur. None of the treatments appeared to modify the relative proportions of the various steroids released.

scholarly journals Sudden cardiac death and acute drunken state: Autopsy study

2010 ◽  
Vol 138 (9-10) ◽  
pp. 590-594 ◽  
Author(s):  
Vladimir Zivkovic ◽  
Borislav Miletic ◽  
Slobodan Nikolic ◽  
Fehim Jukovic
Keyword(s):  
Sudden Cardiac Death ◽  
Cardiac Death ◽  
Alcohol Intoxication ◽  
Blood Alcohol Concentration ◽  
Alcohol Concentration ◽  
Blood Alcohol ◽  
Acute Alcohol Intoxication ◽  
Natural Death ◽  
Autopsy Study ◽  
Alcohol Abusers

Introduction. Sudden natural death occurs unexpectedly in apparently healthy subjects, or in persons during an apparent benign phase in the course of disease. The most common cause is sudden cardiac death, which is sometimes the first and last manifestation of coronary heart disease. Alcohol directly influences excitation of myocytes, and therefore provokes arrhythmias and possibly, sudden cardiac death. Objective. To establish the frequency of sudden cardiac death in cases of acute alcohol intoxication, to determine blood alcohol concentration at the moment of death, and to determine frequency and level of ethanol intoxication in chronic alcohol abusers, as well as causes of sudden death in those cases. Method. Retrospective autopsy study was performed for a three-year-period. We analyzed cases of sudden natural death, in relation to age and gender, cause of death, and blood alcohol concentration (at least 0.5 g/L). We considered the person to be a chronic alcoholic abuser if gross examination of organs during autopsy showed changes typical for excessive and habitual alcohol consumption. Results. Our sample consisted of 997 cases: 720 men and 277 women, average age 62.0?15.2 years (min=11; max=98). Total of 753 of them died of sudden cardiac death: much more men (?2=167.364; p=0.000), significantly younger than women (t=6.203; p=0.000). We determined acute alcohol intoxication in 73 persons - average blood alcohol concentration 1.85?1.01 g/L (min=0.55; max=3.85), and 61 of them died of cardiovascular diseases (?2=236.781; df=5; p=0.000). Conclusion. In our observed sample, not many persons were under acute alcohol intoxication (around 7%). Most commonly, they were chronic alcohol abusers who died due to exacerbation of chronic heart disease, mildly or moderately intoxicated - the younger, the drunker.

Categories of Alcohol Consumers

2010 ◽  
Vol 24 (4) ◽  
pp. 213-214
Author(s):  
Claude Tomberg
Keyword(s):  
Alcohol Consumption ◽  
Alcohol Dependence ◽  
Alcohol Intoxication ◽  
Blood Alcohol ◽  
Acute Alcohol Intoxication ◽  
Common Pattern ◽  
Classification Schemes ◽  
Social Drinking ◽  
Blood Alcohol Levels ◽  
The Brain

Common patterns of alcohol consumption are described with their main criteria, blood alcohol levels, and diagnostic criteria. Binge drinking, an acute alcohol intoxication pattern of particularly concern, is also described, as it appears to be the most common pattern among teenagers when the brain is not yet mature. A number of classification schemes have been proposed specially for social drinking and alcohol dependence, and discussions still surround about them. The article summarize a consensus in classification.

Sign in / Sign up

Export Citation Format

Share Document