A rare case of benign omentum teratoma

Introduction. Mature teratomas (benign cystic teratomas or dermoid cysts) are among the most common ovarian tumours; however, teratomas of the omentum and mesentery are extremely rare. Teratoma in the intraperitoneal cavity is uncommon and atypical, and it is even more uncommon in adulthood. Case Outline. An 82-year-old female was admitted to our department with clinical signs of abdominal tumour. The ultrasound scan and preoperative laboratory tests were done. Explorative laparotomy revealed tumour with torsion on its pedicle at the greater omentum. After removal of the mass and the incision a tooth and hair were found, characteristics of teratoma. Conclusion. The excision was very effective and also definitive treatment for this case. The patient recovered well and was discharged 3 days later. The patient probably carried the tumour all her life asymptomatically until admission.

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Feline Acromegaly: A Review of the Syndrome

Journal of the American Animal Hospital Association ◽

10.5326/0410292 ◽

2005 ◽

Vol 41 (5) ◽

pp. 292-297 ◽

Cited By ~ 22

Author(s):

Charles A. Hurty ◽

Bente Flatland

Keyword(s):

Laboratory Tests ◽

Disease Onset ◽

Clinical Signs ◽

Definitive Diagnosis ◽

Definitive Treatment ◽

Financial Investment ◽

Insulin Resistant ◽

Gh Secretion ◽

Diabetes Mellitus Diagnosis ◽

Serum Gh

Acromegaly is characterized by chronic excessive growth hormone (GH) secretion by the pituitary gland. Feline acromegaly is most commonly caused by a functional pituitary tumor. Definitive diagnosis can be difficult because of the gradual disease onset, subtle clinical signs, unavailability of relevant laboratory tests, and client financial investment. The most significant clinical finding of acromegaly is the presence of insulin-resistant diabetes mellitus. Diagnosis is currently based upon brain imaging and measurement of serum GH and/or insulin-like growth factor-1 concentrations. Definitive treatment in cats is not well described, but radiation therapy appears promising.

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BiodentineTM Full Pulpotomy in Mature Permanent Teeth with Irreversible Pulpitis and Apical Periodontitis

Healthcare ◽

10.3390/healthcare9060720 ◽

2021 ◽

Vol 9 (6) ◽

pp. 720

Author(s):

Xuan Vinh Tran ◽

Lan Thi Quynh Ngo ◽

Tchilalo Boukpessi

Keyword(s):

Root Canal ◽

Periodontal Ligament ◽

Clinical Signs ◽

Apical Periodontitis ◽

Permanent Teeth ◽

Definitive Treatment ◽

Capping Agent ◽

Root Canal Therapy ◽

Pulp Capping ◽

Irreversible Pulpitis

Vital pulp therapy, including direct pulp capping and partial and full pulpotomy, is primarily indicated for immature or mature permanent teeth with reversible pulpitis. Mature permanent teeth with irreversible pulpitis are frequently treated with root canal therapy. This report presents two cases of full pulpotomy using BiodentineTM in mature permanent teeth with irreversible pulpitis and acute apical periodontitis. The periapical radiograph illustrated a deep carious lesion extended to the pulp with apical radiolucency lesion or widened periodontal ligament space. Full pulpotomy with a tricalcium silicate-based cement was chosen as the definitive treatment. After decayed tissue excavation under a rubber dam, the exposed pulp tissue was amputated to the level of the canal orifice with a new sterile bur. BiodentineTM was applied as the pulp capping agent after hemostasis was obtained and for temporary restoration. The clinical signs disappeared quickly after the treatment. After one month, the coronal part of the temporary restoration was removed, and a composite resin was placed over the capping agent as a final restoration. At two-year follow-ups, the teeth were asymptomatic. Radiographs showed healing of the periapical lesion and periodontal ligament. BiodentineTM full pulpotomy of mature permanent teeth with irreversible pulpitis and apical periodontitis can be an alternative option to root canal therapy.

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Report of a Rare Case of Trauma-Induced Thyroid Storm

Ear Nose & Throat Journal ◽

10.1177/014556130208100820 ◽

2002 ◽

Vol 81 (8) ◽

pp. 570-574 ◽

Cited By ~ 16

Author(s):

Neil M. Vora ◽

Fred Fedok ◽

Brendan C. Stack

Keyword(s):

Nervous System ◽

Rare Case ◽

Hepatic Dysfunction ◽

Motor Vehicle ◽

Motor Vehicle Accident ◽

Definitive Treatment ◽

Thyroid Storm ◽

Vehicle Accident ◽

Hyperthyroid State ◽

Life Threatening

Thyroid storm is a potentially life-threatening endocrinologic emergency characterized by an exacerbation of a hyperthyroid state. Several inciting factors can instigate the conversion of thyrotoxicosis to thyroid storm; trauma is one such trigger, but it is rare. Patients with thyroid storm can manifest fever, nervous system disorders, gastrointestinal or hepatic dysfunction (e.g., nausea, vomiting, diarrhea, and/or jaundice), and arrhythmia and other cardiovascular abnormalities. Treatment of thyroid storm is multimodal and is best managed by the endocrinologist and medical intensivist. Initial medical and supportive therapies are directed at stabilizing the patient, correcting the hyperthyroid state, managing the systemic decompensation, and treating the underlying cause. Once this has been achieved, definitive treatment in the form of radioactive ablation or surgery should be undertaken. We describe a case of thyroid storm in a young man that was precipitated by a motor vehicle accident.

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Key-Gaskell syndrome in Brazil: first case report

Arquivo Brasileiro de Medicina Veterinária e Zootecnia ◽

10.1590/1678-6518 ◽

2014 ◽

Vol 66 (4) ◽

pp. 1046-1050

Author(s):

B.B.J. Torres ◽

G.C. Martins ◽

P.E. Ferian ◽

B.C. Martins ◽

M.A. Rachid ◽

...

Keyword(s):

Case Report ◽

Poor Prognosis ◽

Neuronal Degeneration ◽

Clinical Signs ◽

Definitive Treatment ◽

Autonomic Ganglia ◽

Nervous Systems ◽

First Case ◽

The World

Feline dysautonomia is a devastating disease characterized by neuronal degeneration in autonomic ganglia that results in clinical signs related to dysfunction of the sympathetic and parasympathetic nervous systems. The cause is unknown and this disease has a poor prognosis and no definitive treatment. Most reports have been described in few countries around the world, but the prevalence may be underestimated in countries like Brazil. This study describes the progression and clinicopathological changes of dysautonomia in a 17-month-old female Brazilian shorthair cat.

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Trauma surgery

Oxford Assess and Progress: Clinical Surgery ◽

10.1093/oso/9780199696420.003.0021 ◽

2014 ◽

Author(s):

Sebastian Dawson-Bowling ◽

Serena Ledwidge

Keyword(s):

Life Support ◽

Trauma Surgery ◽

Clinical Signs ◽

Tension Pneumothorax ◽

Trauma Team ◽

Definitive Treatment ◽

Advanced Trauma Life Support ◽

Mortality And Morbidity ◽

Golden Hour ◽

The Uk

Appreciation of the ‘golden hour’ for resuscitation, and adoption of principles of the advanced trauma life support (ATLS) system are key factors in improving outcome for the patient with major injuries. Adherence to the strict protocols of the ABCDEs of the primary survey enables the trauma team to identify and deal with life-threatening conditions, prior to definitive treatment of problems with lesser immediacy. The clinician who understands the mechanism of injury will maintain heightened levels of suspicion for clinical signs which point to well-recognized conditions resulting in early mortality and morbidity, for instance, tension pneumothorax, cardiac tamponade, and rising intracranial pressure. This chapter will probe your grasp of the principles of trauma management. You will also be tested on common patterns of thoracic, abdominal, vascular, and cranial injuries. Whilst clinical presentations of civilian trauma have remained consistent in recent years, the impact of military trauma in worldwide theatres of conflict has stimulated numerous advances in the management of trauma. The current impetus for reorganization of trauma services in the UK is tacit acknowledgement of the improvement in outcomes that can be achieved by adherence to recognized protocols in this challenging and demanding field of surgery.

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164 Dementia, a Familial Affair

Age and Ageing ◽

10.1093/ageing/afz103.97 ◽

2019 ◽

Vol 48 (Supplement_3) ◽

pp. iii17-iii65

Author(s):

Aqueel Qureshi ◽

Ahmed Gabr ◽

Daniel Zulkifli ◽

Elijah Chaila ◽

Margaret O'Connor

Keyword(s):

Family History ◽

Rare Case ◽

Clinical Signs ◽

Final Diagnosis ◽

Clinical Findings ◽

Post Mortem ◽

Mri Findings ◽

Progressive Dementia ◽

Familial Predisposition ◽

Rasmussen’S Encephalitis

Abstract Background In Ireland, there are approximately 55,000 cases of dementia. One of the rare causes of dementia is Creutzfeldt-Jakob disease (CJD), affecting one person per million each year worldwide. It is a rapidly progressive degenerative fatal disorder with an estimated mortality of 70% within one year. In this case report we present a rare case of possible familial-CJD which presented initially as a stroke mimic. Methods A 64-year old female referred via GP with abnormal left arm athetoid movements and in-coordination. MRI brain demonstrated a small sub-acute hyper-intense lesion in the right basal ganglia on diffusion weighted imaging. Her symptoms were atypical for acute stroke, however an alternative cause was not evident. She was monitored closely with early supported discharge. However the left sided athetoid movements worsened and were associated with intermittent myoclonic jerks and dystonic posturing. She was reassessed with a wider differential including focal impaired seizures, Rasmussen’s encephalitis, and CJD. EEG showed periodic lateralizing epileptic discharges, however patient failed to show any response to anti-epileptic treatment and her clinical course was one of rapid deterioration. Clinical findings and subsequent MRI findings showed new areas of hyperintensity supporting CJD. Results Ultimately our patient deteriorated rapidly resulting in an akinetic and abulic state, resulting in death. A final diagnosis of sporadic-CJD was made based on rapid progressive deterioration and findings on MRI as well as confirmation on post-mortem brain pathology. A deeper review of family history revealed a sister who had passed away years prior with rapid progressive neurological illness. Her work-up showed clinical signs and EEG findings supporting CJD, however there was no post mortem to confirm her diagnosis. Conclusion This case highlights the importance of genetics shaping phenotypes and that consideration should always be given to a full relevant family history. It also shows a rare case of rapidly progressive dementia confirmed due to CJD with a likely underlying familial predisposition.

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Sports-Related Concussion: Neurometabolic Aspects

Seminars in Speech and Language ◽

10.1055/s-0039-1679887 ◽

2019 ◽

Vol 40 (05) ◽

pp. 333-343

Author(s):

Russell E. Banks ◽

Delfina C. Domínguez

Keyword(s):

Traumatic Brain Injury ◽

Brain Injury ◽

Mild Traumatic Brain Injury ◽

Laboratory Tests ◽

Clinical Signs ◽

Signs And Symptoms ◽

Time Frame ◽

Neurological Damage ◽

Metabolic Disturbances ◽

Clinical Signs And Symptoms

AbstractConcussion is a transitory brain injury resulting from a blow to the head. Concussion is considered a mild traumatic brain injury (mTBI), which is self-limited. Repetitive mTBI has been associated with chronic, progressive neurological damage. Extreme biochemical changes occur in neuron cells as a result of mTBI. These metabolic disturbances may reflect the symptoms observed in patients who had suffered concussions. However, it has been difficult to correlate clinical signs and symptoms. Currently, there are no laboratory tests to diagnose concussion, though several biomarkers are being investigated. Further studies are needed to elucidate the biochemical details of the metabolic cascade and the associated time frame, which will help determine when an athlete can safely return to the game.

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6.16 Some Advances in Clinical Biochemistry Relating to Trace Metals

BSAP Occasional Publication ◽

10.1017/s0263967x00030305 ◽

1983 ◽

Vol 7 ◽

pp. 149-150

Author(s):

C. H. McMurray ◽

W. J. Blanchflower ◽

P. J. McParland ◽

D. G. O'Neill ◽

D. A. Rice

Keyword(s):

Trace Metals ◽

Blood Plasma ◽

Whole Blood ◽

Laboratory Tests ◽

Blood Clotting ◽

Clinical Biochemistry ◽

Clinical Signs ◽

Cu Deficiency ◽

Phenylene Diamine

The clinical signs of copper (Cu) deficiency are largely non-specific and a number of laboratory tests have been used extensively to assist in diagnosis. Among these are whole blood, plasma and serum Cu and caeruloplasmin (McMurray, 1980). However, for any marker to be useful diagnostically, it is necessary to identify any factors which can affect it. Plasma and serum Cu are not equivalent but are related by the equation:Serum Cu (mg/l) = 11.7 + 0.66 plasma Cu (mg/l). The regression was obtained from the means of 24 groups of suckler cows and calves (> 10 animals/group). The equivalent relationship between serum and plasma caeruloplasmin is:Serum caeruloplasmin = 0.0018+ 0.59 plasma caeruloplasmin.Units of caeruloplasmin are absorbance units in the phenylene diamine assay.Thus, the range of normality will depend on the sample being used for the assay. The reduction in serum values is due to the loss of caeruloplasmin during blood clotting.

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