Role of the Chick's Begging Behavior in the Regulation of Parental Feeding Behavior of Larus glaucescens

The Condor ◽  
1975 ◽  
Vol 77 (4) ◽  
pp. 488 ◽  
Author(s):  
Bryan A. Henderson
Keyword(s):  
Feeding Behavior ◽  
Parental Feeding ◽  
Begging Behavior ◽  
Larus Glaucescens

Associations Between Parent–Child Communication and Connectedness, Parent Feeding Behavior, and Child Body Mass in Pre-Adolescent Children

2020 ◽  
Author(s):  
Kristina D Lowe ◽  
Mark A Lott ◽  
Chad D Jensen
Keyword(s):  
Feeding Behavior ◽  
Body Mass ◽  
Weight Status ◽  
Community Sample ◽  
Feeding Strategies ◽  
Feeding Behaviors ◽  
Parent Child Communication ◽  
Parental Feeding ◽  
Parent Feeding ◽  
Parent Child

Abstract Objective  This study evaluated associations between parent–child connectedness and communication, parent feeding behaviors (restriction, pressure to eat, and monitoring), and age- and sex-standardized child body mass index (zBMI) in a sample of pre-adolescent children aged 8–12 years. Methods  A community sample of three hundred and eight child–parent dyads completed measures of communication and connectedness. Parents completed a feeding behavior measure and children were weighed and their height was measured. We examined whether parental feeding behaviors and parent–child communication and connectedness predicted child zBMI and whether parental feeding behaviors moderated the association between parent–child communication and connectedness and child zBMI. Results  Feeding restriction was positively associated with zBMI, while both pressure to eat and food monitoring exhibited negative associations with zBMI. Child-reported communication was inversely associated with zBMI and parental pressure to eat moderated this association such that lower pressure to eat predicted a stronger association between communication and zBMI. Conclusions  These findings are consistent with previous research suggesting that parent feeding strategies and parent–child communication are important contributors to child weight status. This study also provides preliminary evidence suggesting that adaptive parent–child communication is associated with lower body mass when parents avoid pressuring their child to eat. Our study provides an important extension of this body of research into middle childhood, a relatively understudied developmental stage.

scholarly journals GLP-1 Receptor Stimulation of the Lateral Parabrachial Nucleus Reduces Food Intake: Neuroanatomical, Electrophysiological, and Behavioral Evidence

Endocrinology ◽  
2014 ◽  
Vol 155 (11) ◽  
pp. 4356-4367 ◽  
Author(s):  
Jennifer E. Richard ◽  
Imre Farkas ◽  
Fredrik Anesten ◽  
Rozita H. Anderberg ◽  
Suzanne L. Dickson ◽  
...  
Keyword(s):  
Feeding Behavior ◽  
Fluorescent Protein ◽  
Yellow Fluorescent Protein ◽  
Parabrachial Nucleus ◽  
Lateral Parabrachial Nucleus ◽  
Electrophysiological Studies ◽  
Glucagon Like Peptide 1 ◽  
Behavioral Evidence ◽  
Stimulation Of

Abstract The parabrachial nucleus (PBN) is a key nucleus for the regulation of feeding behavior. Inhibitory inputs from the hypothalamus to the PBN play a crucial role in the normal maintenance of feeding behavior, because their loss leads to starvation. Viscerosensory stimuli result in neuronal activation of the PBN. However, the origin and neurochemical identity of the excitatory neuronal input to the PBN remain largely unexplored. Here, we hypothesize that hindbrain glucagon-like peptide 1 (GLP-1) neurons provide excitatory inputs to the PBN, activation of which may lead to a reduction in feeding behavior. Our data, obtained from mice expressing the yellow fluorescent protein in GLP-1-producing neurons, revealed that hindbrain GLP-1-producing neurons project to the lateral PBN (lPBN). Stimulation of lPBN GLP-1 receptors (GLP-1Rs) reduced the intake of chow and palatable food and decreased body weight in rats. It also activated lPBN neurons, reflected by an increase in the number of c-Fos-positive cells in this region. Further support for an excitatory role of GLP-1 in the PBN is provided by electrophysiological studies showing a remarkable increase in firing of lPBN neurons after Exendin-4 application. We show that within the PBN, GLP-1R activation increased gene expression of 2 energy balance regulating peptides, calcitonin gene-related peptide (CGRP) and IL-6. Moreover, nearly 70% of the lPBN GLP-1 fibers innervated lPBN CGRP neurons. Direct intra-lPBN CGRP application resulted in anorexia. Collectively, our molecular, anatomical, electrophysiological, pharmacological, and behavioral data provide evidence for a functional role of the GLP-1R for feeding control in the PBN.

Role of gut-brain axis in persistent abnormal feeding behavior in mice following eradication of Helicobacter pylori infection

2009 ◽  
Vol 296 (3) ◽  
pp. R587-R594 ◽  
Author(s):  
Premysl Bercik ◽  
Elena F. Verdú ◽  
Jane A. Foster ◽  
Jun Lu ◽  
Angela Scharringa ◽  
...  
Keyword(s):  
Gastric Emptying ◽  
Feeding Behavior ◽  
Computer Assisted ◽  
Bacterial Eradication ◽  
Cell Counts ◽  
Tnf Α ◽  
Gastric Sensitivity ◽  
H Pylori ◽  
The Brain

Bacterial infection can trigger the development of functional GI disease. Here, we investigate the role of the gut-brain axis in gastric dysfunction during and after chronic H. pylori infection. Control and chronically H. pylori-infected Balb/c mice were studied before and 2 mo after bacterial eradication. Gastric motility and emptying were investigated using videofluoroscopy image analysis. Gastric mechanical viscerosensitivity was assessed by cardioautonomic responses to distension. Feeding patterns were recorded by a computer-assisted system. Plasma leptin, ghrelin, and CCK levels were measured using ELISA. IL-1β, TNF-α, proopiomelanocortin (POMC), and neuropeptide Y mRNAs were assessed by in situ hybridizations on frozen brain sections. Gastric inflammation was assessed by histology and immunohistochemistry. As shown previously, H. pylori-infected mice ate more frequently than controls but consumed less food per bout, maintaining normal body weight. Abnormal feeding behavior was accompanied by elevated plasma ghrelin and postprandial CCK, higher TNF-α (median eminence), and lower POMC (arcuate nucleus) mRNA. Infected mice displayed delayed gastric emptying and visceral hypersensitivity. Eradication therapy normalized gastric emptying and improved gastric sensitivity but had no effect on eating behavior. This was accompanied by persistently increased TNF-α in the brain and gastric CD3+ T-cell counts. In conclusion, chronic H. pylori infection in mice alters gastric emptying and mechanosensitivity, which improve after bacterial eradication. A feeding pattern reminiscent of early satiety persists after H. pylori eradication and is accompanied by increased TNF-α in the brain. The results support a role for altered gut-brain pathways in the maintenance of postinfective gut dysfunction.

scholarly journals THE ROLE OF THE OBESOGENIC ENVIRONMENT AND PARENTAL LIFESTYLES IN INFANT FEEDING BEHAVIOR

2019 ◽  
Vol 37 (3) ◽  
pp. 363-371
Author(s):  
Rafaela Ramos Dantas ◽  
Giselia Alves Pontes da Silva
Keyword(s):  
Feeding Behavior ◽  
Infant Feeding ◽  
Eating Behavior ◽  
Family Environment ◽  
Correct Choice ◽  
Obesogenic Environment ◽  
Medical Subject Headings ◽  
The Family ◽  
Infant Feeding Behavior

ABSTRACT Objective: To identify the role of the obesogenic environment and parental lifestyles in infant feeding behavior. Data sources: The searches were performed in PubMed, Medline, Cochrane, Lilacs and Scielo databases, in Portuguese, English and Spanish. The descriptors used were found in the Medical Subject Headings and in the Descriptors in Health Sciences being these: Comportamento alimentar/Feeding Behavior/ Conducta Alimentaria; Crianças/Child/ Niño; Relações familiares/Family Relations/Relaciones Familiares; e Ecologia/ Ecology/ Ecología. These were combined by the Boolean operator AND. Data synthesis: Researchers consider that parents (orprimary caregivers) are responsible, in part, for the unhealthy eating behavior presented by children, and for them to change it is necessary to change the behavior of the family, ensuring the correct choice of food and the practice of physical activity. The family environment has a significant impact on the development of eating behavior, so adults should provide a good model of this behavior for children. Conclusions: It was verified through this review that, in order to maintain and develop a healthy eating behavior, it is necessary to reach different spheres of life of the individual - physical, social, psychological, family, cultural and mediatic environment.

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