ULTRASOUND CORRELATION OF PLACENTA PREVIA IN ANC FEMALES WITH PREVIOUS LSCS PREGNANCIES

The placenta is a highly specialized tissue that serves a number of crucial functions for survival of the foetus. It allows the foetus to receive oxygen and nutrition before it can breathe or nourish itself. The placenta releases multiple hormones that initiate and maintain the essential changes of pregnancy in both the mother and the foetus. (1, 2)The normal human placenta averages 22 cm (9 inch) in length and 2–2.5 cm (0.8–1 inch) in thickness, with the center being the thickest, and the edges being the thinnest.

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Expression of ZO-1 and occludin in normal human placenta and in hydatidiform moles

MHR Basic science of reproductive medicine ◽

10.1093/molehr/7.3.279 ◽

2001 ◽

Vol 7 (3) ◽

pp. 279-285 ◽

Cited By ~ 28

Author(s):

D. Marzioni

Keyword(s):

Human Placenta ◽

Normal Human ◽

Hydatidiform Moles

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Time-dependent expression pattern of cytochrome P450 epoxygenases and soluble epoxide hydrolase in normal human placenta

Acta Histochemica ◽

10.1016/j.acthis.2018.06.002 ◽

2018 ◽

Vol 120 (6) ◽

pp. 513-519 ◽

Cited By ~ 6

Author(s):

K. Cizkova ◽

Z. Tauber

Keyword(s):

Cytochrome P450 ◽

Expression Pattern ◽

Human Placenta ◽

Epoxide Hydrolase ◽

Soluble Epoxide Hydrolase ◽

Time Dependent ◽

Normal Human

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CELLULAR GROWTH IN HUMAN PLACENTA

PEDIATRICS ◽

10.1542/peds.39.2.248 ◽

1967 ◽

Vol 39 (2) ◽

pp. 248-251

Author(s):

Myron Winick ◽

Anthony Coscia ◽

Adele Noble

Keyword(s):

Cell Division ◽

Protein Content ◽

Human Placenta ◽

Normal Values ◽

Cellular Growth ◽

Base Line ◽

Rate Of Increase ◽

Normal Human ◽

Total Dna ◽

Human Placentae

Fifty normal human placentae from various gestations were analyzed for total DNA, RNA, and protein content. The data indicate that, although weight, RNA, and protein continue to increase linearly until term, the rate of increase in DNA rapidly declines when the placenta reaches about 300 gm or the fetus 2,300 gm. When these data are interpreted in terms of number and size of cells they suggest that cell division stops in human placenta about 1 month prior to term. The latter portion of placental growth is by enlargement of already existing cells. These data establish normal values for human placenta from 26 through 42 weeks and provide a base line to compare placentae of abnormal pregnancies.

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Ex ‐ Vivo MRI of the Normal Human Placenta: Structural–Functional Interplay and the Association With Birth Weight

Journal of Magnetic Resonance Imaging ◽

10.1002/jmri.28002 ◽

2021 ◽

Author(s):

Daphna Link‐Sourani ◽

Netanell Avisdris ◽

Shaul Harel ◽

Liat Ben‐Sira ◽

Tuvia Ganot ◽

...

Keyword(s):

Birth Weight ◽

Human Placenta ◽

Ex Vivo ◽

Normal Human

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Human placental vitamin B6 (pyridoxal) transport: normal characteristics and effects of ethanol

AJP Regulatory Integrative and Comparative Physiology ◽

10.1152/ajpregu.1992.262.6.r966 ◽

1992 ◽

Vol 262 (6) ◽

pp. R966-R974 ◽

Cited By ~ 4

Author(s):

S. Schenker ◽

R. F. Johnson ◽

J. D. Mahuren ◽

G. I. Henderson ◽

S. P. Coburn

Keyword(s):

Human Placenta ◽

Binding Sites ◽

Vitamin B6 ◽

Passive Transport ◽

Short Term ◽

Placental Transport ◽

Normal Human ◽

Important Form ◽

Composite Data ◽

In Pregnancy

The aims of this study were to define normal human placental transport of pyridoxal, an important form of vitamin B6 in pregnancy, and to determine the effect of short-term alcohol on this process. Our studies used the isolated single cotyledon from the term placenta. Pyridoxal crossed the human placenta readily in both directions, but the transfer was a little less than half that of antipyrine and was significantly greater in the direction of the fetus. Pyridoxine appeared to have a similar clearance from the maternal compartment as pyridoxal, but transport of intact pyridoxal 5'-phosphate was much smaller. There was no saturable transfer of pyridoxal, and it was not transferred from the maternal to fetal compartments against a concentration gradient. Placental concentration of pyridoxal exceeded both maternal and fetal perfusate pyridoxal concentrations, but this concentration was equal for both perfusion directions. These composite data are most suggestive of passive transport of pyridoxal across the placenta, binding of the vitamin in the placenta as an explanation for its concentration there, and greater phosphorylation of pyridoxal in the placenta when the compound is transferred in the fetal direction, possibly displacing pyridoxal from its binding sites and permitting its greater release into the fetal compartment. Alcohol, 400-250 mg/dl over 2.5 h, inhibited the transport of pyridoxal from the maternal to fetal compartments by approximately 42% (P = 0.03) and resulted in a lower transfer of pyridoxal 5'-phosphate into the fetal perfusate (P = 0.02).

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