scholarly journals Analisis Kestabilan Model Penyebaran Penyakit Toxoplasmosis pada Populasi Manusia dan Kucing

2019 ◽  
Vol 1 (3) ◽  
pp. 37-46
Author(s):  
Febrina Tedjo Utami ◽  
Retno Wahyu Dewanti ◽  
Subchan Subchan

This final project studies the contruction and analyzes the stability of Toxoplasmosis epidemic model. Using a compartment model, Toxoplasmosis epidemic model classified into human population and cat population. The human population is divided into three subpopulations such as susceptible human, infected human, and recovered human. The cat population is divided into two subpopulations such as susceptible cat and infected cat. Stability analysis performed on two equilibrium models of disease free equilibrium and endemic equilibrium point. Stability analysis result showed that cat infected spread, natural birthd rate of cat population, natural death rate of cat population, and the probability of cat natality in the form of basic reproductive number (R0) is value that affect human population and cat population changed spread. When R0<1 the population will be free of disease and when R0>1 the disease will be spread. Model of the simulation is numerically analyzed by Runge Kutta 4th Order methods.

Author(s):  
Laid Chahrazed

In this work, we consider a nonlinear epidemic model with temporary immunity and saturated incidence rate. Size N(t) at time t, is divided into three sub classes, with N(t)=S(t)+I(t)+Q(t); where S(t), I(t) and Q(t) denote the sizes of the population susceptible to disease, infectious and quarantine members with the possibility of infection through temporary immunity, respectively. We have made the following contributions: The local stabilities of the infection-free equilibrium and endemic equilibrium are; analyzed, respectively. The stability of a disease-free equilibrium and the existence of other nontrivial equilibria can be determine by the ratio called the basic reproductive number, This paper study the reduce model with replace S with N, which does not have non-trivial periodic orbits with conditions. The endemic -disease point is globally asymptotically stable if R0 ˃1; and study some proprieties of equilibrium with theorems under some conditions. Finally the stochastic stabilities with the proof of some theorems. In this work, we have used the different references cited in different studies and especially the writing of the non-linear epidemic mathematical model with [1-7]. We have used the other references for the study the different stability and other sections with [8-26]; and sometimes the previous references.


2017 ◽  
Vol 82 (5) ◽  
pp. 945-970 ◽  
Author(s):  
Jinliang Wang ◽  
Min Guo ◽  
Shengqiang Liu

Abstract An SVIR epidemic model with continuous age structure in the susceptibility, vaccination effects and relapse is proposed. The asymptotic smoothness, existence of a global attractor, the stability of equilibria and persistence are addressed. It is shown that if the basic reproductive number $\Re_0&lt;1$, then the disease-free equilibrium is globally asymptotically stable. If $\Re_0&gt;1$, the disease is uniformly persistent, and a Lyapunov functional is used to show that the unique endemic equilibrium is globally asymptotically stable. Combined effects of susceptibility age, vaccination age and relapse age on the basic reproductive number are discussed.


2013 ◽  
Vol 21 (03) ◽  
pp. 1350023 ◽  
Author(s):  
B. DUBEY ◽  
ATASI PATRA ◽  
P. K. SRIVASTAVA ◽  
UMA S. DUBEY

In this study, an SEIR epidemic model is proposed for treatment of infectives considering the development of acquired immunity in recovered individuals. We employed two different types of treatment functions. Stability analysis for disease-free as well as endemic equilibria is performed. It is observed that the existence of unique endemic equilibrium depends on the basic reproductive number R0as well as on treatment rate. Numerical simulations are performed on the proposed models to support and analyze theoretical findings.


Author(s):  
Laid Chahrazed

In this work, we consider a nonlinear epidemic model with a saturated incidence rate. we consider a population of size N(t) at time t, this population is divided into six subclasses, with N(t)=S(t)+I(t)+I₁(t)+I₂(t)+I₃(t)+Q(t). Where S(t), I(t), I₁(t), I₂(t), I₃(t), and Q(t) denote the sizes of the population susceptible to disease, infectious members, and quarantine members, respectively. We have made the following contributions: 1. The local stabilities of the infection-free equilibrium and endemic equilibrium are; analyzed, respectively. The stability of a disease-free equilibrium and the existence of other nontrivial equilibria can be determined by the ratio called the basic reproductive number. 2. We find the analytical solution of the nonlinear epidemic model by Homotopy perturbation method. 3. Finally the stochastic stabilities. The study of its sections are justified with theorems and demonstrations under certain conditions. In this work, we have used the different references cited in different studies in the three sections already mentioned.


Author(s):  
Ruksana Shaikh ◽  
Pradeep Porwal ◽  
V. K. Gupta

The study indicates that we should improve the model by introducing the immigration rate in the model to control the spread of disease. An SEIRS epidemic model with Immigration and Vertical Transmission and analyzed the steady state and stability of the equilibrium points. The model equations were solved analytically. The stability of the both equilibrium are proved by Routh-Hurwitz criteria. We see that if the basic reproductive number R0<1 then the disease free equilibrium is locally asymptotically stable and if R0<1 the endemic equilibrium will be locally asymptotically stable.


Author(s):  
Iqbal M. Batiha ◽  
Shaher Momani ◽  
Adel Ouannas ◽  
Zaid Momani ◽  
Samir B. Hadid

Today, the entire world is witnessing an enormous upsurge in coronavirus pandemic (COVID-19 pandemic). Confronting such acute infectious disease, which has taken multiple victims around the world, requires all specialists in all fields to devote their efforts to seek effective treatment or even control its disseminate. In the light of this aspect, this work proposes two new fractional-order versions for one of the recently extended forms of the SEIR model. These two versions, which are established in view of two fractional-order differential operators, namely, the Caputo and the Caputo–Fabrizio operators, are numerically solved based on the Generalized Euler Method (GEM) that considers Caputo sense, and the Adams–Bashforth Method (ABM) that considers Caputo–Fabrizio sense. Several numerical results reveal the impact of the fractional-order values on the two established disease models, and the continuation of the COVID-19 pandemic outbreak to this moment. In the meantime, some novel results related to the stability analysis and the basic reproductive number are addressed for the proposed fractional-order Caputo COVID-19 model. For declining the total of individuals infected by such pandemic, a new compartment is added to the proposed model, namely the disease prevention compartment that includes the use of face masks, gloves and sterilizers. In view of such modification, it is turned out that the performed addition to the fractional-order Caputo COVID-19 model yields a significant improvement in reducing the risk of COVID-19 spreading.


2020 ◽  
Vol 8 (2) ◽  
pp. 61-68
Author(s):  
Victor Akinsola ◽  
ADEYEMI BINUYO

In this paper, a mathematical model of the transmission dynamics of corruption among populace is analyzed. The corruption free equilibrium state, characteristic equation and Eigen values of the corruption model were obtained. The basic reproductive number of the corruption model was also determined using the next generation operator technique at the corruption free equilibrium points. The condition for the stability of the corruption free equilibrium state was determined. The local stability analysis of the mathematical model of corruption was done and the results were presented and discussed accordingly. Recommendations were made from the results on measures to reduce the rate of corrupt practices among the populace.   


2017 ◽  
Vol 2017 ◽  
pp. 1-7 ◽  
Author(s):  
Olusegun Michael Otunuga

We derive and analyze the dynamic of a stochastic SEI epidemic model for disease spread. Fluctuations in the transmission rate of the disease bring about stochasticity in model. We discuss the asymptotic stability of the infection-free equilibrium by first deriving the closed form deterministic (R0) and stochastic (R0) basic reproductive number. Contrary to some author’s remark that different diffusion rates have no effect on the stability of the disease-free equilibrium, we showed that even if no epidemic invasion occurs with respect to the deterministic version of the SEI model (i.e., R0<1), epidemic can still grow initially (if R0>1) because of the presence of noise in the stochastic version of the model. That is, diffusion rates can have effect on the stability by causing a transient epidemic advance. A threshold criterion for epidemic invasion was derived in the presence of external noise.


2020 ◽  
Vol 38 (2) ◽  
pp. 133-163
Author(s):  
Cristhian Montoya ◽  
Jhoana P. Romero Leiton

In this work, two mathematical models for malaria under resistance are presented. More precisely, the first model shows the interaction between humans and mosquitoes inside a patch under infection of malaria when the human population is resistant to antimalarial drug and mosquitoes population is resistant to insecticides. For the second model, human–mosquitoes population movements in two patches is analyzed under the same malaria transmission dynamic established in a patch. For a single patch, existence and stability conditions for the equilibrium solutions in terms of the local basic reproductive number are developed. These results reveal the existence of a forward bifurcation and the global stability of disease–free equilibrium. In the case of two patches, a theoretical and numerical framework on sensitivity analysis of parameters is presented. After that, the use of antimalarial drugs and insecticides are incorporated as control strategies and an optimal control problem is formulated. Numerical experiments are carried out in both models to show the feasibility of our theoretical results.


2019 ◽  
Vol 2019 ◽  
pp. 1-17 ◽  
Author(s):  
Zhixing Hu ◽  
Shanshan Yin ◽  
Hui Wang

This paper established a delayed vector-borne disease model with saturated infection rate and cure rate. First of all, according to the basic reproductive number R0, we determined the disease-free equilibrium E0 and the endemic equilibrium E1. Through the analysis of the characteristic equation, we consider the stability of two equilibriums. Furthermore, the effect on the stability of the endemic equilibrium E1 by delay was studied, the existence of Hopf bifurcations of this system in E1 was analyzed, and the length of delay to preserve stability was estimated. The direction and stability of the Hopf bifurcation were also been determined. Finally, we performed some numerical simulation to illustrate our main results.


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