Sensory polyneuropathy in human immunodeficiency virus-infected patients receiving tuberculosis treatment

2014 ◽  
Vol 18 (1) ◽  
pp. 27-33 ◽  
Author(s):  
C. M. Centner ◽  
H. Carrara ◽  
T. B. Harrison ◽  
M. Benatar ◽  
J. M. Heckmann
CNS Spectrums ◽  
2000 ◽  
Vol 5 (5) ◽  
pp. 66-72 ◽  
Author(s):  
Ashok Verma ◽  
Walter G. Bradley

AbstractPeripheral neuropathy associated with human immunodeficiency virus type 1 (HIV-1) infection is a major cause of morbidity in this patient population. Due to the associated chronic pain, its management has come within the purview of neuropsychiatrists. This paper will focus on the primary pathogenic aspects of HIV-1–associated peripheral neuropathies. The specific syndromes of greatest concern are distal sensory polyneuropathy, toxic neuropathy, inflammatory demyelinating polyradiculoneuropathy, and cytomegalovirus-related progressive polyradiculoneuropathy. The treatments available for these conditions and their efficacy are discussed.


1998 ◽  
Vol 72 (9) ◽  
pp. 7664-7668 ◽  
Author(s):  
Gianguglielmo Zehender ◽  
Luca Meroni ◽  
Stefania Varchetta ◽  
Chiara De Maddalena ◽  
Barbara Cavalli ◽  
...  

ABSTRACT We investigated the presence of human T-lymphotropic virus type 2 (HTLV-2) DNA in the peripheral blood mononuclear cell subsets obtained from 18 patients coinfected with human immunodeficiency virus type 1 and HTLV-2, 6 of whom also had predominantly sensory polyneuropathy (PSP). HTLV-2 DNA and RNA were found in CD8- and CD19-positive cells, and, for patients with PSP, in CD14-positive cells as well. Furthermore, the patients with PSP had higher proviral loads than those without PSP.


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