scholarly journals Thrust Force Generated by Heaving Motion of a Plate: The Role of Vortex-Induced Force

2021 ◽  
Author(s):  
Kazuo Matsuuchi

To understand the force acting on birds, insects, and fish, we take heaving motion as a simple example. This motion might deviate from the real one. However, since the mechanism of force generation is the vortex shedding due to the motion of an object, the heaving motion is important for understanding the force generated by unsteady motion. The vortices released from the object are closely related to the motion characteristics. To understand the force acting on an object, information about momentum change is necessary. However, in vortex systems, it is impossible to estimate the usual momentum. Instead of the momentum, the “virtual momentum,” or the impulse, is needed to generate the force. For calculating the virtual momentum, we traced all vortices over a whole period, which was carried out by using the vortex-element method. The force was then calculated based on the information on the vortices. We derived the thrust coefficient as a function of the ratio of the heaving to travelling velocity.

Author(s):  
V. S. Teslenko ◽  
◽  
A. P. Drozhzhin ◽  
R. N. Medvedev ◽  
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...  
Keyword(s):  

The results of experiments on measurements of pulses of thrust force and bubble pulsations generated on a flat thrust wall due to combustion of a stoichiometric propane-oxygen mixture are presented.


2001 ◽  
Vol 281 (6) ◽  
pp. L1402-L1412 ◽  
Author(s):  
Leo M. A. Heunks ◽  
Herwin A. Machiels ◽  
Ronney de Abreu ◽  
Xiao Ping Zhu ◽  
Henricus F. M. van der Heijden ◽  
...  

Recent evidence indicates that hypoxia enhances the generation of oxidants. Little is known about the role of free radicals in contractility of the rat diaphragm during hypoxia. We hypothesized that antioxidants improve contractility of the hypoxic rat diaphragm and that xanthine oxidase (XO) is an important source of free radicals in the hypoxic diaphragm. The effects of N-acetylcysteine (NAC; 18 mM), Tiron (10 mM), and the XO inhibitor allopurinol (250 μM) were studied on isometric and isotonic force generation during hypoxia (Po 2 ∼7 kPa). NAC and Tiron decreased maximal force generation, slowed the shortening velocity, and decreased the power output. Fatigue rate was decreased in the presence of either NAC or Tiron. Allopurinol did not alter the contractility or fatigability of the diaphragm. During hyperoxia (Po 2 ∼85 kPa), neither NAC nor allopurinol affected the contractility or fatigability of the diaphragm. Thus free radicals play a significant role in diaphragm contractility during hypoxia. Whether antioxidants exert a beneficial or harmful effect on muscle performance depends on the contraction pattern of the muscle. Free radicals generated by XO do not play a role in diaphragm contractility during either hypoxia or hyperoxia.


2021 ◽  
Vol 153 (3) ◽  
Author(s):  
Masataka Kawai ◽  
Robert Stehle ◽  
Gabriele Pfitzer ◽  
Bogdan Iorga

In this study, we aimed to study the role of inorganic phosphate (Pi) in the production of oscillatory work and cross-bridge (CB) kinetics of striated muscle. We applied small-amplitude sinusoidal length oscillations to rabbit psoas single myofibrils and muscle fibers, and the resulting force responses were analyzed during maximal Ca2+ activation (pCa 4.65) at 15°C. Three exponential processes, A, B, and C, were identified from the tension transients, which were studied as functions of Pi concentration ([Pi]). In myofibrils, we found that process C, corresponding to phase 2 of step analysis during isometric contraction, is almost a perfect single exponential function compared with skinned fibers, which exhibit distributed rate constants, as described previously. The [Pi] dependence of the apparent rate constants 2πb and 2πc, and that of isometric tension, was studied to characterize the force generation and Pi release steps in the CB cycle, as well as the inhibitory effect of Pi. In contrast to skinned fibers, Pi does not accumulate in the core of myofibrils, allowing sinusoidal analysis to be performed nearly at [Pi] = 0. Process B disappeared as [Pi] approached 0 mM in myofibrils, indicating the significance of the role of Pi rebinding to CBs in the production of oscillatory work (process B). Our results also suggest that Pi competitively inhibits ATP binding to CBs, with an inhibitory dissociation constant of ∼2.6 mM. Finally, we found that the sinusoidal waveform of tension is mostly distorted by second harmonics and that this distortion is closely correlated with production of oscillatory work, indicating that the mechanism of generating force is intrinsically nonlinear. A nonlinear force generation mechanism suggests that the length-dependent intrinsic rate constant is asymmetric upon stretch and release and that there may be a ratchet mechanism involved in the CB cycle.


Author(s):  
S. K. Phillips ◽  
J. L. Rowbury ◽  
S. A. Bruce ◽  
R. C. Woledge

1965 ◽  
Vol 87 (4) ◽  
pp. 1006-1010 ◽  
Author(s):  
D. P. Wang ◽  
T. Yao-Tsu Wu

The problem of a two-dimensional cavity flow of an ideal fluid with small unsteady disturbances in a gravity free field is considered. By regarding the unsteady motion as a small-perturbation of an established steady cavity flow, a fundamental formulation of the problem is presented. It is shown that the unsteady disturbance generates a surface wave propagating downstream along the free cavity boundary, much in the same way as the classical gravity waves in water, only with the centrifugal acceleration owing to the curvature of the streamlines in the basic flow playing the role of an equivalent gravity effect. As a particularly simple example, the surface waves in a hollow potential vortex flow is calculated using the present theory.


2010 ◽  
Vol 56 (3) ◽  
pp. 319-328
Author(s):  
A.G. Hasanov ◽  
T.V. Bershova ◽  
E.N. Basargina ◽  
M.I. Bakanov

The review highlighted problems of reorganization of myocardical contractile and cytoskeletal proteins in cardiomyopathy (CM). The role of the genetic factors coding contractile proteins, proteins of thin and thick filaments, and also extracellular matrix proteins in processes of formation and development of hypertrophic (HCM) and dilated (DCM) cardiomyopathy are analyzed. The mechanisms responsible for the changes in cardiac proteins on regulation involved into force generation, its transfer, recycling ATP, impairments in transmembranal signals, that finally lead to cardiac cell dysfunction determining various manifestations of CM are considered.


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