Tertiary Teachers Strike (TTS) and e-Learning Deficit amidst Covid-19 Crisis in Nigeria

Amid the raging Covid-19 pandemic across the world and the debilitating tertiary teachers strike in Nigeria, this study’s objective seeks to examine the prevailing un-lived experiences of Nigerian tertiary students in e-learning. The study argues that Covid-19 has widened the digital divide between Nigerian universities and other universities in other parts of the world on the one hand and between public and private tertiary institutions in Nigeria on the other. This e-learning deficit is worsened by university teachers’ strikes, constituting a twin inhibition into which higher education is consigned in Nigeria. The study identifies poor funding of education as a major constraint to virtual learning and instruction faced by public tertiary students especially in the era of the pandemic. Data collection for the study will be carried out through oral interviews basically focus group discussion (FGD) from a sample population of 50 university students (male and female) in three universities across the southeast region of Nigeria, newspaper reports, and participant-observer methods of research analysis.

A pair of dopamine neurons mediate chronic stress signals to induce learning deficit in Drosophila melanogaster

Chronic stress could induce severe cognitive impairments. Despite extensive investigations in mammalian models, the underlying mechanisms remain obscure. Here, we show that chronic stress could induce dramatic learning and memory deficits in Drosophila melanogaster. The chronic stress–induced learning deficit (CSLD) is long lasting and associated with other depression-like behaviors. We demonstrated that excessive dopaminergic activity provokes susceptibility to CSLD. Remarkably, a pair of PPL1-γ1pedc dopaminergic neurons that project to the mushroom body (MB) γ1pedc compartment play a key role in regulating susceptibility to CSLD so that stress-induced PPL1-γ1pedc hyperactivity facilitates the development of CSLD. Consistently, the mushroom body output neurons (MBON) of the γ1pedc compartment, MBON-γ1pedc>α/β neurons, are important for modulating susceptibility to CSLD. Imaging studies showed that dopaminergic activity is necessary to provoke the development of chronic stress–induced maladaptations in the MB network. Together, our data support that PPL1-γ1pedc mediates chronic stress signals to drive allostatic maladaptations in the MB network that lead to CSLD.

Education during and after the pandemics

Education in the pandemic time often received an improvised and non-realistic treatment. This is a major reason for planning now the post-pandemic period. According to the literature and document analysis, social, international and intra-national fractures, detected by current theories, were largely exposed. Besides Covid-19, other crises developed, such as impoverishment and violence. In general, the population has had losses; however, the least privileged have been subject to a greater burden. Countries cannot repeat past mistakes or try to restore Education as it was before, since the circumstances are different. Furthermore, in addition to the learning deficit, it is urgent to recover Education in its multiple ends and goals. Therefore, it is important to support educators, students and families, with a specific focus on the underprivileged, counting on the participation of the social forces of community and society.

Not All Procedural Learning Tasks Are Difficult for Adults With Developmental Language Disorder

Purpose The experiment reported here compared two hypotheses for the poor statistical and artificial grammar learning often seen in children and adults with developmental language disorder (DLD; also known as specific language impairment). The procedural learning deficit hypothesis states that implicit learning of rule-based input is impaired, whereas the sequential pattern learning deficit hypothesis states that poor performance is only seen when learners must implicitly compute sequential dependencies. The current experiment tested learning of an artificial grammar that could be learned via feature activation, as observed in an associatively organized lexicon, without computing sequential dependencies and should therefore be learnable on the sequential pattern learning deficit hypothesis, but not on the procedural learning deficit hypothesis. Method Adults with DLD and adults with typical language development (TD) listened to consonant–vowel–consonant–vowel familiarization words from one of two artificial phonological grammars: Family Resemblance (two out of three features) and a control (exclusive OR, in which both consonants are voiced OR both consonants are voiceless) grammar in which no learning was predicted for either group. At test, all participants rated 32 test words as to whether or not they conformed to the pattern in the familiarization words. Results Adults with DLD and adults with TD showed equal and robust learning of the Family Resemblance grammar, accepting significantly more conforming than nonconforming test items. Both groups who were familiarized with the Family Resemblance grammar also outperformed those who were familiarized with the OR grammar, which, as predicted, was learned by neither group. Conclusion Although adults and children with DLD often underperform, compared to their peers with TD, on statistical and artificial grammar learning tasks, poor performance appears to be tied to the implicit computation of sequential dependencies, as predicted by the sequential pattern learning deficit hypothesis.

Autism-linked gene FoxP1 selectively regulates the cultural transmission of learned vocalizations

Autism spectrum disorders (ASDs) are characterized by impaired learning of social skills and language. Memories of how parents and other social models behave are used to guide behavioral learning. How ASD-linked genes affect the intertwined aspects of observational learning and behavioral imitation is not known. Here, we examine how disrupted expression of the ASD gene FOXP1, which causes severe impairments in speech and language learning, affects the cultural transmission of birdsong between adult and juvenile zebra finches. FoxP1 is widely expressed in striatal-projecting forebrain mirror neurons. Knockdown of FoxP1 in this circuit prevents juvenile birds from forming memories of an adult song model but does not interrupt learning how to vocally imitate a previously memorized song. This selective learning deficit is associated with potent disruptions to experience-dependent structural and synaptic plasticity in mirror neurons. Thus, FoxP1 regulates the ability to form memories essential to the cultural transmission of behavior.