Volume Integral Equation Method Solution for Spheroidal Inclusion Problem

In this paper, the volume integral equation method (VIEM) is introduced for the numerical analysis of an infinite isotropic solid containing a variety of single isotropic/anisotropic spheroidal inclusions. In order to introduce the VIEM as a versatile numerical method for the three-dimensional elastostatic inclusion problem, VIEM results are first presented for a range of single isotropic/orthotropic spherical, prolate and oblate spheroidal inclusions in an infinite isotropic matrix under uniform remote tensile loading. We next considered single isotropic/orthotropic spherical, prolate and oblate spheroidal inclusions in an infinite isotropic matrix under remote shear loading. The authors hope that the results using the VIEM cited in this paper will be established as reference values for verifying the results of similar research using other analytical and numerical methods.

Equivalent inclusion approach and approximations for conductivity of isotropic matrix composites with sphere-like, platelet, and fibrous fillers

The construction starts from certain typical effective medium approximations for conductivity of idealistic isotropic matrix composites with randomly oriented inclusions of perfect spherical, platelet, and circular fiber forms, which obey Hashin–Shtrikman bounds over all the ranges of volume proportions of the component materials. Equivalent inclusion approach is then developed to account for possible diversions, such as non-idealistic geometric forms of the inhomogeneities, imperfect matrix-inclusion contacts, filler dispersions, and when the particular values of the fillers’ properties are unspecified, using available numerical or experimental reference conductivity data for particular composites. Illustrating applications involving experimental data from the literature show the usefulness of the approach.

Modeling the Deformation of the Elastin Network in the Aortic Valve

This paper is concerned with proposing a suitable structurally motivated strain energy function, denoted by Weelastin network, for modeling the deformation of the elastin network within the aortic valve (AV) tissue. The AV elastin network is the main noncollagenous load-bearing component of the valve matrix, and therefore, in the context of continuum-based modeling of the AV, the Weelastin network strain energy function would essentially serve to model the contribution of the “isotropic matrix.” To date, such a function has mainly been considered as either a generic neo-Hookean term or a general exponential function. In this paper, we take advantage of the established structural analogy between the network of elastin chains and the freely jointed molecular chain networks to customize a structurally motivated Weelastin network function on this basis. The ensuing stress–strain (force-stretch) relationships are thus derived and fitted to the experimental data points reported by (Vesely, 1998, “The Role of Elastin in Aortic Valve Mechanics,” J. Biomech., 31, pp. 115–123) for intact AV elastin network specimens under uniaxial tension. The fitting results are then compared with those of the neo-Hookean and the general exponential models, as the frequently used models in the literature, as well as the “Arruda–Boyce” model as the gold standard of the network chain models. It is shown that our proposed Weelastin network function, together with the general exponential and the Arruda–Boyce models provide excellent fits to the data, with R2 values in excess of 0.98, while the neo-Hookean function is entirely inadequate for modeling the AV elastin network. However, the general exponential function may not be amenable to rigorous interpretation, as there is no structural meaning attached to the model. It is also shown that the parameters estimated by the Arruda–Boyce model are not mathematically and structurally valid, despite providing very good fits. We thus conclude that our proposed strain energy function Weelastin network is the preferred choice for modeling the behavior of the AV elastin network and thereby the isotropic matrix. This function may therefore be superimposed onto that of the anisotropic collagen fibers family in order to develop a structurally motivated continuum-based model for the AV.