The involvement of protein TNFSF18 in promoting p‐STAT1 phosphorylation to induce coronary microcirculation disturbance in atherosclerotic mouse model

2020 ◽  
Author(s):  
Jing Gao ◽  
Sai Wang ◽  
Shilei Liu

1996 ◽  
Vol 19 (5) ◽  
pp. 379-383 ◽  
Author(s):  
Hideo Kakoi ◽  
Teruo Inoue ◽  
Shigenori Morooka ◽  
Terumi Hayashi ◽  
Yutaka Takabatake


Author(s):  
H. D. Geissinge ◽  
L.D. Rhodes

A recently discovered mouse model (‘mdx’) for muscular dystrophy in man may be of considerable interest, since the disease in ‘mdx’ mice is inherited by the same mode of inheritance (X-linked) as the human Duchenne (DMD) muscular dystrophy. Unlike DMD, which results in a situation in which the continual muscle destruction cannot keep up with abortive regenerative attempts of the musculature, and the sufferers of the disease die early, the disease in ‘mdx’ mice appears to be transient, and the mice do not die as a result of it. In fact, it has been reported that the severely damaged Tibialis anterior (TA) muscles of ‘mdx’ mice seem to display exceptionally good regenerative powers at 4-6 weeks, so much so, that these muscles are able to regenerate spontaneously up to their previous levels of physiological activity.



1998 ◽  
Vol 13 (11-s4) ◽  
pp. S178-S184 ◽  
Author(s):  
PETER KONTUREK ◽  
TOMASZ BRZOZOWSKI ◽  
STANISLAW KONTUREK ◽  
ELZBIETA KARCZEWSKA ◽  
ROBERT PAJDO ◽  
...  




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