Prenatal Stress Exposure Generates Higher Early Survival and Smaller Size without Impacting Developmental Rate in a Pacific Salmon

2016 ◽  
Vol 325 (10) ◽  
pp. 641-650 ◽  
Author(s):  
Pauline M. Capelle ◽  
Christina A. D. Semeniuk ◽  
Natalie M. Sopinka ◽  
John W. Heath ◽  
Oliver P. Love
Keyword(s):  
Prenatal Stress ◽  
Pacific Salmon ◽  
Developmental Rate ◽  
Stress Exposure ◽  
Early Survival

scholarly journals Prenatal stress exposure is associated with increased dyspnoea perception in adulthood

2017 ◽  
Vol 50 (2) ◽  
pp. 1700642 ◽  
Author(s):  
Andreas von Leupoldt ◽  
Eline Mangelschots ◽  
Nils Georg Niederstrasser ◽  
Marijke Braeken ◽  
Thibo Billiet ◽  
...  
Keyword(s):  
Prenatal Stress ◽  
Stress Exposure

scholarly journals Hidden pandemic: COVID-19-related stress, SLC6A4 methylation, and infants’ temperament at 3 months

2021 ◽  
Vol 11 (1) ◽  
Author(s):  
Livio Provenzi ◽  
Fabiana Mambretti ◽  
Marco Villa ◽  
Serena Grumi ◽  
Andrea Citterio ◽  
...  
Keyword(s):  
Prenatal Stress ◽  
Collective Trauma ◽  
Transporter Gene ◽  
Stress Exposure ◽  
Buccal Cells ◽  
Stress Effects ◽  
Cpg Sites ◽  
Stress Related Genes ◽  
Related Stress ◽  
Epigenetic Signatures

AbstractThe COVID-19 pandemic represents a collective trauma that may have enduring stress effects during sensitive periods, such as pregnancy. Prenatal stress may result in epigenetic signatures of stress-related genes (e.g., the serotonin transporter gene, SLC6A4) that may in turn influence infants’ behavioral development. In April 2020, we launched a longitudinal cohort study to assess the behavioral and epigenetic vestiges of COVID-19-related prenatal stress exposure in mothers and infants. COVID-19-related prenatal stress was retrospectively assessed at birth. SLC6A4 methylation was assessed in thirteen CpG sites in mothers and infants’ buccal cells. Infants’ temperament was assessed at 3-month-age. Complete data were available from 108 mother-infant dyads. Greater COVID-19-related prenatal stress was significantly associated with higher infants’ SLC6A4 methylation in seven CpG sites. SLC6A4 methylation at these sites predicted infants’ temperament at 3 months.

Prenatal stress exposure and early childhood BMI: Exploring associations in a New Zealand context

2018 ◽  
Vol 30 (4) ◽  
pp. e23116 ◽  
Author(s):  
Charlotte V. Farewell ◽  
Zaneta M. Thayer ◽  
David P. Tracer ◽  
Susan Morton
Keyword(s):  
Early Childhood ◽  
New Zealand ◽  
Prenatal Stress ◽  
Stress Exposure

The weight gain response to stress during adulthood is conditioned by both sex and prenatal stress exposure

2010 ◽  
Vol 35 (3) ◽  
pp. 403-413 ◽  
Author(s):  
Cristina García-Cáceres ◽  
Yolanda Diz-Chaves ◽  
Natalia Lagunas ◽  
Isabel Calmarza-Font ◽  
Iñigo Azcoitia ◽  
...  
Keyword(s):  
Weight Gain ◽  
Prenatal Stress ◽  
Stress Exposure ◽  
Response To Stress

Effects of transgenic sterilization constructs and their repressor compounds on hatch, developmental rate and early survival of electroporated channel catfish embryos and fry

2014 ◽  
Vol 24 (2) ◽  
pp. 333-352 ◽  
Author(s):  
Baofeng Su ◽  
Mei Shang ◽  
Chao Li ◽  
Dayan A. Perera ◽  
Carl A. Pinkert ◽  
...  
Keyword(s):  
Channel Catfish ◽  
Developmental Rate ◽  
Early Survival

A Nationwide Study on the Risk of Autism After Prenatal Stress Exposure to Maternal Bereavement

PEDIATRICS ◽  
2009 ◽  
Vol 123 (4) ◽  
pp. 1102-1107 ◽  
Author(s):  
J. Li ◽  
M. Vestergaard ◽  
C. Obel ◽  
J. Christensen ◽  
D. H. Precht ◽  
...  
Keyword(s):  
Prenatal Stress ◽  
Stress Exposure ◽  
Nationwide Study

scholarly journals Hidden Pandemic: COVID-19-Related Stress, SLC6A4 Methylation and Infants’ Temperament at 3 Months

2021 ◽  
Author(s):  
Livio Provenzi ◽  
Fabiana Mambretti ◽  
Marco Villa ◽  
Serena Grumi ◽  
Andrea Citterio ◽  
...  
Keyword(s):  
Prenatal Stress ◽  
Well Being ◽  
Stress Exposure ◽  
Buccal Cells ◽  
Stress Effects ◽  
Cpg Sites ◽  
Preventive Actions ◽  
Stress Related Genes ◽  
Related Stress ◽  
Epigenetic Signatures

Abstract The COVID-19 pandemic represents a collective trauma that may have enduring stress effects during sensitive periods, such as pregnancy. Prenatal stress may result in epigenetic signatures of stress-related genes (e.g., the serotonin transporter gene, SLC6A4) that may in turn influence infants’ behavioral development. In April 2020, we launched a longitudinal cohort study to assess the behavioral and epigenetic vestiges of COVID-19-related prenatal stress exposure in mothers and infants. COVID-19-related prenatal stress was retrospectively assessed at birth. SLC6A4 methylation was assessed in thirteen CpG sites in mothers and infants’ buccal cells. Infants’ temperament was assessed at 3-month-age. Complete data were available from 108 mother-infant dyads. Greater COVID-19-related prenatal stress was significantly associated with higher infants’ SLC6A4 methylation in seven CpG sites. SLC6A4 methylation at these sites predicted infants’ temperament at 3 months. Clinicians and policymakers should promote timely preventive actions to promote infant well-being during and after the present pandemic.

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