Arsenic transport between water and sediments

1992 ◽  
pp. 533-544
Author(s):  
Jack Cornett ◽  
Lorna Chant ◽  
Bert Risto
Keyword(s):  
Arsenic Transport

Systematic engineering of phytochelatin synthesis and arsenic transport for enhanced arsenic accumulation inE. coli

2009 ◽  
pp. n/a-n/a ◽  
Author(s):  
Shailendra Singh ◽  
Seung Hyun Kang ◽  
Wonkyu Lee ◽  
Ashok Mulchandani ◽  
Wilfred Chen
Keyword(s):  
Arsenic Accumulation ◽  
Arsenic Transport

Mathematical modelling of arsenic transport, distribution and detoxification processes in yeast

2014 ◽  
Vol 92 (6) ◽  
pp. 1343-1356 ◽  
Author(s):  
Soheil Rastgou Talemi ◽  
Therese Jacobson ◽  
Vijay Garla ◽  
Clara Navarrete ◽  
Annemarie Wagner ◽  
...  
Keyword(s):  
Mathematical Modelling ◽  
Arsenic Transport

scholarly journals Arsenic Transport by the Human Multidrug Resistance Protein 1 (MRP1/ABCC1)

2004 ◽  
Vol 279 (31) ◽  
pp. 32700-32708 ◽  
Author(s):  
Elaine M. Leslie ◽  
Anass Haimeur ◽  
Michael P. Waalkes
Keyword(s):  
Multidrug Resistance ◽  
Multidrug Resistance Protein ◽  
Multidrug Resistance Protein 1 ◽  
Resistance Protein ◽  
Arsenic Transport

Unexpected Beneficial Effects of Arsenic on Corn Roots Grown in Culture

2005 ◽  
Vol 2 (3) ◽  
pp. 167 ◽  
Author(s):  
Grant Evans ◽  
Julyette Evans ◽  
Andrea Redman ◽  
Nancy Johnson ◽  
Richard D. Foust
Keyword(s):  
Root Development ◽  
Contaminated Soils ◽  
Branch Length ◽  
Beneficial Effects ◽  
Chemically Modified ◽  
Transport Studies ◽  
Soil Arsenic ◽  
Low Levels ◽  
Arsenic Transport ◽  
First Time

Environmental Context. Phytoremediation, the process of using plants to remove metals from contaminated soils, shows promise as a low-technology method for economically removing arsenic, and other toxic metals, from soil. Arsenic transport studies in vascular plants have examined how arsenic is taken up, chemically modified, and transported from roots to other parts of the plant. No studies, to our knowledge, have examined the effect of low-level doses of arsenic on the roots themselves. This paper shows, for the first time, that arsenic at low levels may beneficially affect root development. Abstract. Corn (Zea mays) roots were grown in culture on modified Strullu–Roman medium in two separate experiments. Roots were exposed to one of four treatments combining arsenic (100 µg L−1 or 0.0 µg L−1) and phosphorous (4.8 mg L−1 or 0.0 mg L−1). The cultures were allowed to grow for 18 days or 21 days before they were used for quantitative measurement of root mass, root length, number of branches, and branch length. Results indicate roots grown in medium lacking phosphate but containing arsenic were longer and had greater mass than roots grown in medium with only phosphate. The data presented here suggest that arsenic at low levels might be beneficial for root development.

Cd(II)-binding transcriptional regulator interacts with isoniazid and regulates drug susceptibility in mycobacteria

2020 ◽  
Author(s):  
Min Yang ◽  
Shi-Hua Jia ◽  
Hui-Ling Tao ◽  
Chen Zhu ◽  
Wan-Zhong Jia ◽  
...  
Keyword(s):  
Drug Resistance ◽  
Bacterial Resistance ◽  
Critical Role ◽  
Integral Membrane Protein ◽  
Drug Susceptibility ◽  
Binding Activity ◽  
Dependent Manner ◽  
Minimal Inhibitory Concentrations ◽  
Dna Binding Activity ◽  
Arsenic Transport

Abstract It is urgent to understand the regulatory mechanism of drug resistance in widespread bacterial pathogens. In Mycobacterium tuberculosis, several transcriptional regulators have been found to play essential roles in regulating its drug resistance. In this study, we found that an ArsR family transcription regulator encoded by Rv2642 (CdiR) responds to isoniazid (INH), a widely used anti-tuberculosis (TB) drug. CdiR negatively regulates self and adjacent genes, including arsC (arsenic-transport integral membrane protein ArsC). CdiR directly interacts with INH and Cd(II). The binding of INH and Cd(II) both reduce its DNA-binding activity. Disrupting cdiR increased the drug susceptibility to INH, whereas overexpressing cdiR decreased the susceptibility. Strikingly, overexpressing arsC increased the drug susceptibility as well as cdiR. Additionally, both changes in cdiR and arsC expression caused sensitivity to other drugs such as rifamycin and ethambutol, where the minimal inhibitory concentrations in the cdiR deletion strain were equal to those of the arsC-overexpressing strain, suggesting that the function of CdiR in regulating drug resistance primarily depends on arsC. Furthermore, we found that Cd(II) enhances bacterial resistance to INH in a CdiR-dependent manner. As a conclusion, CdiR has a critical role in directing the interplay between Cd(II) metal ions and drug susceptibility in mycobacteria.

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