scholarly journals Proteomic differences in amyloid plaques in rapidly progressive and sporadic Alzheimer’s disease

2017 ◽  
Vol 133 (6) ◽  
pp. 933-954 ◽  
Author(s):  
Eleanor Drummond ◽  
Shruti Nayak ◽  
Arline Faustin ◽  
Geoffrey Pires ◽  
Richard A. Hickman ◽  
...  
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Amyloid Plaques ◽  
Sporadic Alzheimer’S Disease

IC-P-126: Divergent pattern of changes in astrocytosis and fibrillar amyloid plaques as measured by PET in autosomal-dominant and sporadic Alzheimer's disease

2015 ◽  
Vol 11 (7S_Part_2) ◽  
pp. P86-P86
Author(s):  
Elena Rodriguez-Vieitez ◽  
Stephen F. Carter ◽  
Laure Saint-Aubert ◽  
Ove Almkvist ◽  
Karim Farid ◽  
...  
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Autosomal Dominant ◽  
Amyloid Plaques ◽  
Sporadic Alzheimer’S Disease

O1-02-03: Divergent pattern of changes in astrocytosis and fibrillar amyloid plaques as measured by PET in autosomal-dominant and sporadic Alzheimer's disease

2015 ◽  
Vol 11 (7S_Part_3) ◽  
pp. P127-P127
Author(s):  
Elena Rodriguez-Vieitez ◽  
Stephen F. Carter ◽  
Laure Saint-Aubert ◽  
Ove Almkvist ◽  
Karim Farid ◽  
...  
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Autosomal Dominant ◽  
Amyloid Plaques ◽  
Sporadic Alzheimer’S Disease

scholarly journals Study on the cause and pathogenesis of Alzheimer's disease

2019 ◽  
Author(s):  
zuodong sun
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Neurodegenerative Diseases ◽  
Cell Activation ◽  
Amyloid Plaques ◽  
Brain Cell ◽  
Research Field ◽  
Brain Cells ◽  
Sporadic Alzheimer’S Disease ◽  
Long Time

An enlightening viewpoint based on the theory of brain cell activation is to explore the pathogenesis and mechanism of sporadic Alzheimer's disease (AD) at molecular level by applying the principles of cell physics and biology. Its purpose is to coordinate and unify the existing theories of AD etiology and to solve the problems that have puzzled the research field of neurodegenerative diseases for a long time. Basic Contents: Excessive cations are transferred from extracellular to intracellular, and compete with potassium ions on the inner surface of the cell membrane. As a result, the action potential produced can not activate calcium channels normally, leading to abnormal apoptosis of brain cells. Amyloid plaques are the remains of abnormal apoptotic brain cells. Amyloid plaques are aggregated by van der Waals force and electrostatic attraction between amyloid plaques. The interstitium is amyloid protein. Brain cells consist of neurons, microglia and astrocytes in turn. Most of the spotted nuclei in the remains are cations. Applicable but not limited to neurodegenerative diseases such as Alzheimer's disease, physical means should be preferred to solve such diseases.

The Weak Combined Magnetic Fields Reduce the Brain β-Amyloid in an Animal Model of Sporadic Alzheimer's Disease

PIERS Online ◽  
2009 ◽  
Vol 5 (4) ◽  
pp. 311-315 ◽  
Author(s):  
Natalia V. Bobkova ◽  
Vadim V. Novikov ◽  
Natalia I. Medvinskaya ◽  
Irina Yu. Aleksandrova ◽  
Eugenii E. Fesenko
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Animal Model ◽  
Magnetic Fields ◽  
Sporadic Alzheimer’S Disease ◽  
Combined Magnetic Fields ◽  
The Brain
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