scholarly journals The transition state regulator Hpr of Bacillus subtilis is a DNA-binding protein

1991 ◽  
Vol 266 (20) ◽  
pp. 13411-13417
Author(s):  
P.T. Kallio ◽  
J.E. Fagelson ◽  
J.A. Hoch ◽  
M.A. Strauch
Keyword(s):  
Bacillus Subtilis ◽  
Dna Binding ◽  
Transition State ◽  
Binding Protein ◽  
Dna Binding Protein ◽  
State Regulator ◽  
Transition State Regulator

scholarly journals The transition state transcription regulator abrB of Bacillus subtilis is a DNA binding protein.

1989 ◽  
Vol 8 (5) ◽  
pp. 1615-1621 ◽  
Author(s):  
M. A. Strauch ◽  
G. B. Spiegelman ◽  
M. Perego ◽  
W. C. Johnson ◽  
D. Burbulys ◽  
...  
Keyword(s):  
Bacillus Subtilis ◽  
Dna Binding ◽  
Transition State ◽  
Binding Protein ◽  
Dna Binding Protein ◽  
Transcription Regulator

Evaluation of the DNA Binding Tendencies of the Transition State Regulator AbrB†

Biochemistry ◽  
2004 ◽  
Vol 43 (51) ◽  
pp. 16106-16118 ◽  
Author(s):  
Benjamin G. Bobay ◽  
Linda Benson ◽  
Stephen Naylor ◽  
Brett Feeney ◽  
A. Clay Clark ◽  
...  
Keyword(s):  
Dna Binding ◽  
Transition State ◽  
State Regulator ◽  
Transition State Regulator

A dual mode of regulation of flgM by ScoC in Bacillus subtilis

2009 ◽  
Vol 55 (8) ◽  
pp. 983-989 ◽  
Author(s):  
Prashant Kodgire ◽  
K. Krishnamurthy Rao
Keyword(s):  
Bacillus Subtilis ◽  
Transition State ◽  
Promoter Region ◽  
Dual Mode ◽  
State Regulator ◽  
Transition State Regulator ◽  
Dependent Pathway ◽  
Mode Of Regulation

In Bacillus subtilis , the transition state regulator ScoC indirectly, negatively regulates the anti-σD factor FlgM in a SinR-dependent pathway leading to an increased availability of σD. In addition to the SinR-dependent pathway, ScoC negatively regulates FlgM via directly repressing flgM transcription by binding to two sites in the promoter region of the flgM operon. Our studies also show that the regulation of FlgM by SinR is not at the transcriptional or translational levels. Thus, ScoC shows a dual mode of downregulation of FlgM, via both SinR-dependent and -independent pathways, which eventually results in the increased σD activity.

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