Nicotine dependence and major depression among young adults and adults in Japan

2003 ◽  
Vol 55 (2) ◽  
pp. 168-169
Author(s):  
K Muramatsu ◽  
Y Muramatsu ◽  
H Miyaoka ◽  
K Kamijima ◽  
F Yoshimine ◽  
...  
1993 ◽  
Vol 33 (2) ◽  
pp. 129-137 ◽  
Author(s):  
Naomi Breslau ◽  
Nancy Fenn ◽  
Edward L. Peterson

2008 ◽  
Vol 10 (1) ◽  
pp. 97-108 ◽  
Author(s):  
Michael Lyons ◽  
Brian Hitsman ◽  
Hong Xian ◽  
Matthew Panizzon ◽  
Beth Jerskey ◽  
...  

2019 ◽  
Vol 22 (11) ◽  
pp. 2006-2013
Author(s):  
Jungeun Olivia Lee ◽  
L John Horwood ◽  
Woo Jung Lee ◽  
Daniel A Hackman ◽  
Geraldine Fh McLeod ◽  
...  

Abstract Introduction Unemployment has been related to smoking, yet the causal nature of the association is subject to continued debate. Social causation argues that unemployment triggers changes in smoking, whereas the social selection hypothesis proposes that pre-existing smoking behavior lowers the probability of maintaining employment. The present study tested these competing explanations while accounting for another alternative explanation—common liability. Methods Data were from the Christchurch Health and Development Study, a longitudinal cohort followed from birth to age 35. Odds were generated for having nicotine dependence in models for social causation and being unemployed in models for social selection. These models were extended to include possible common liability factors during childhood (eg, novelty seeking) and young adulthood (eg, major depression). Results In the model testing social causation, coefficients representing the impacts of unemployment on nicotine dependence remained statistically significant and robust (odds ratio [OR] = 1.55; 95% confidence interval [CI] = 1.20, 2.00), even after accounting for common determinant measures. In contrast, a reverse social selection model revealed that coefficients representing the impacts of nicotine dependence on unemployment substantially attenuated and became statistically nonsignificant as childhood factors were added (OR = 1.14; 95% CI = 0.90, 1.45). Conclusions Unemployment may serve as inroads to nicotine addiction among young adults, not the other way, even in the context of nicotine dependence, a more impaired form of smoking that may arguably hold higher potential to generate social selection processes. This social causation process cannot be completely attributable to common determinant factors. Implications It is critical to clarify whether unemployment triggers changes in smoking behaviors (ie, social causation) or vice versa (ie, social selection)—the answers to the question will lead to public health strategies with very different intervention targets to break the linkage. The current study findings favor social causation over social selection, regardless of gender, and support a needed shift in service profiles for unemployed young adults—from a narrow focus on job skills training to a more holistic approach that incorporates knowledge from addiction science in which unemployed young adults can find needed services to cope with job loss.


2007 ◽  
Vol 10 (3) ◽  
pp. 470-478 ◽  
Author(s):  
Qiang Fu ◽  
Andrew C. Heath ◽  
Kathleen K. Bucholz ◽  
Michael J. Lyons ◽  
Ming T. Tsuang ◽  
...  

AbstractMany studies that found associations between depression and nicotine dependence have ignored possible shared genetic influences associated with antisocial traits. The present study examined the contribution of genetic and environmental effects associated with conduct disorder (CD) and antisocial personality disorder (ASPD) to the comorbidity of major depression (MD) and nicotine dependence (ND). A telephone diagnostic interview, the Diagnostic Interview Schedule-III-R, was administered to eligible twins from the Vietnam Era Twin (VET) Registry in 1992. Multivariate genetic models were fitted to 3360 middle-aged and predominantly white twin pairs (1868 monozygotic, 1492 dizygotic pairs) of which both members completed the pertinent diagnostic interview sections. Genetic influences on CD accounted for 100%, 68%, and 50% of the total genetic variance in risk for ASPD, MD and ND, respectively. After controlling for genetic influences on CD, the partial genetic correlation between MD and ND was no longer statistically significant. Nonshared environmental contributions to the comorbidity among these disorders were not significant. This study not only demonstrates that the comorbidity between ND and MD is influenced by common genetic risk factors, but also further suggests that the common genetic risk factors overlapped with those for antisocial traits such as CD and ASPD in men.


2008 ◽  
Vol 161 (2) ◽  
pp. 170-176 ◽  
Author(s):  
David Gourion ◽  
Louise Arseneault ◽  
Frank Vitaro ◽  
Jelena Brezo ◽  
Gustavo Turecki ◽  
...  

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