Gender differences in dependency, separation, object relations and pathological eating behavior and attitudes

2004 ◽  
Vol 36 (4) ◽  
pp. 801-811 ◽  
Author(s):  
Steven K Huprich ◽  
Stephanie D Stepp ◽  
Ariel Graham ◽  
Lacey Johnson
1991 ◽  
Vol 10 (2) ◽  
pp. 133-142 ◽  
Author(s):  
Barbara J. Rolls ◽  
Ingrid C. Fedoroff ◽  
Joanne F. Guthrie

Appetite ◽  
2017 ◽  
Vol 117 ◽  
pp. 135-142 ◽  
Author(s):  
Ellen Hart ◽  
Chong Man Chow ◽  
Cin Cin Tan

Author(s):  
Anika Hoffmann ◽  
Frank P. Postma ◽  
Anthe S. Sterkenburg ◽  
Ursel Gebhardt ◽  
Hermann L. Müller

AbstractAs a result of hypothalamic involvement and/or treatment-related hypothalamic damage, up to 75% of childhood craniopharyngioma patients develop hypothalamic obesity.Eating behavior was analyzed in 101 survivors of childhood craniopharyngioma, recruited from 1980 to 2001 in the HIT-Endo multicenter study, and in 85 body mass index (BMI)-matched healthy controls using the Inventory for Eating Behavior and Weight Problems (IEG) and the Inventory for Eating Disorders (ESI).Severely obese patients (BMI>8 SD; n=9) presented with pathological eating behavior, more weight problems, and eating disorders, as compared to obese (BMI 3–8 SD; n=44) and normal or overweight patients (BMI<3 SD; n=48). Craniopharyngioma patients with different degrees of obesity showed similar or even less pathological findings as compared to BMI-matched normal controls.Severe obesity is associated with pathological eating behavior/disorders in craniopharyngioma patients. As these disorders are not disease-specific, risk factors for hypothalamic obesity should be the focus of further craniopharyngioma research.


PLoS ONE ◽  
2017 ◽  
Vol 12 (1) ◽  
pp. e0170413 ◽  
Author(s):  
Aude Lagarrigue ◽  
Soufiane Ajana ◽  
Lucile Capuron ◽  
Catherine Féart ◽  
Marie-Pierre Moisan

Appetite ◽  
2017 ◽  
Vol 110 ◽  
pp. 103-107 ◽  
Author(s):  
Mareile Opwis ◽  
Jennifer Schmidt ◽  
Alexandra Martin ◽  
Christel Salewski

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