Oxidative stress and low level radiation

1998 ◽  
Vol 25 ◽  
pp. S83
Author(s):  
Ingrid Emerit ◽  
Urij Antipkine ◽  
Natascha Zybina ◽  
Amina Alaoui ◽  
Paulo Filipe ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Low Level

scholarly journals Photobiomodulation with Red and Near-Infrared Light Improves Viability and Modulates Expression of Mesenchymal and Apoptotic-Related Markers in Human Gingival Fibroblasts

Materials ◽  
2021 ◽  
Vol 14 (12) ◽  
pp. 3427
Author(s):  
Ievgeniia Kocherova ◽  
Artur Bryja ◽  
Katarzyna Błochowiak ◽  
Mariusz Kaczmarek ◽  
Katarzyna Stefańska ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Energy Density ◽  
Near Infrared ◽  
Infrared Light ◽  
Specific Gene ◽  
Gingival Fibroblasts ◽  
Inflammation Markers ◽  
Low Level ◽  
Low Level Laser ◽  
Level Laser

Photobiomodulation (PBM), also called low-level laser treatment (LLLT), has been considered a promising tool in periodontal treatment due to its anti-inflammatory and wound healing properties. However, photobiomodulation’s effectiveness depends on a combination of parameters, such as energy density, the duration and frequency of the irradiation sessions, and wavelength, which has been shown to play a key role in laser-tissue interaction. The objective of the study was to compare the in vitro effects of two different wavelengths—635 nm and 808 nm—on the human primary gingival fibroblasts in terms of viability, oxidative stress, inflammation markers, and specific gene expression during the four treatment sessions at power and energy density widely used in dental practice (100 mW, 4 J/cm2). PBM with both 635 and 808 nm at 4 J/cm2 increased the cell number, modulated extracellular oxidative stress and inflammation markers and decreased the susceptibility of human primary gingival fibroblasts to apoptosis through the downregulation of apoptotic-related genes (P53, CASP9, BAX). Moreover, modulation of mesenchymal markers expression (CD90, CD105) can reflect the possible changes in the differentiation status of irradiated fibroblasts. The most pronounced results were observed following the third irradiation session. They should be considered for the possible optimization of existing low-level laser irradiation protocols used in periodontal therapies.

Low level laser therapy reduces oxidative stress in cortical neurons in vitro

2012 ◽  
Author(s):  
Ying-Ying Huang ◽  
Clark E. Tedford ◽  
Thomas McCarthy ◽  
Michael R. Hamblin
Keyword(s):  
Oxidative Stress ◽  
Laser Therapy ◽  
Cortical Neurons ◽  
Low Level Laser Therapy ◽  
Low Level ◽  
Low Level Laser ◽  
Level Laser

scholarly journals Oxidative Stress, hogg1 Expression and NF‐κB Activity in Cells Exposed to Low Level Chromium

2003 ◽  
Vol 45 (5) ◽  
pp. 271-277 ◽  
Author(s):  
Yong‐Dae Kim ◽  
Sung‐Chul An ◽  
Tsunehiro Oyama ◽  
Toshihiro Kawamoto ◽  
Heon Kim
Keyword(s):  
Oxidative Stress ◽  
Low Level ◽  
In Cells

Sites and Mechanisms of Low-Level Oxidative Stress in Cultured Cells

1995 ◽  
Vol 206 (1) ◽  
pp. 421-428 ◽  
Author(s):  
D. Gelvan ◽  
V. Moreno ◽  
D.A. Clopton ◽  
Q. Chen ◽  
P. Saltman
Keyword(s):  
Oxidative Stress ◽  
Cultured Cells ◽  
Low Level

Effects of low Level Laser Therapy on Oxidative Stress in Rats

2006 ◽  
Vol 36 (3) ◽  
pp. 667-676
Author(s):  
Hoda Hamdy ◽  
Mamdouh El-Yamany ◽  
Mohamed Shetewy ◽  
Afaf El-Faras ◽  
Aziza Ibrahim
Keyword(s):  
Oxidative Stress ◽  
Laser Therapy ◽  
Low Level Laser Therapy ◽  
Low Level ◽  
Low Level Laser ◽  
Level Laser

Low-Level Laser Therapy Restores the Oxidative Stress Balance in Acute Lung Injury Induced by Gut Ischemia and Reperfusion

2012 ◽  
Vol 89 (1) ◽  
pp. 179-188 ◽  
Author(s):  
Flávia Mafra de Lima ◽  
Regiane Albertini ◽  
Yvana Dantas ◽  
Antonio Luis Maia-Filho ◽  
Cristiano de Loura Santana ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Acute Lung Injury ◽  
Lung Injury ◽  
Laser Therapy ◽  
Low Level Laser Therapy ◽  
Ischemia And Reperfusion ◽  
Low Level ◽  
Low Level Laser ◽  
Level Laser

Oxidative stress in the rat heart, studies on low-level chemiluminescence

1989 ◽  
Vol 4 (1) ◽  
pp. 241-244 ◽  
Author(s):  
Fulvio Ursini ◽  
Renata Barsacchi ◽  
Gualtiero Pelosi ◽  
Antonio Benassi
Keyword(s):  
Oxidative Stress ◽  
Rat Heart ◽  
Low Level

scholarly journals Antioxidant activity of vitamin B6 delays homocysteine-induced atherosclerosis in rats

2006 ◽  
Vol 95 (6) ◽  
pp. 1088-1093 ◽  
Author(s):  
Naoko Endo ◽  
Kazuo Nishiyama ◽  
Akira Otsuka ◽  
Hiroaki Kanouchi ◽  
Masaki Taga ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Serum Lipid ◽  
Lipid Peroxide ◽  
Rat Aorta ◽  
Histochemical Staining ◽  
Vitamin B ◽  
Vascular Integrity ◽  
Homocysteine Thiolactone ◽  
Low Level ◽  
Biochemical Profiles

Elevated plasma homocysteine is a risk factor for atherosclerotic disease. In the present study, we have examined whether the oxidative stress due to a low level of vitamin B6 accelerates the development of homocysteine-induced atherosclerosis in rats. First, the effect of homocysteine thiolactone intake (50mg/kgperd) on vascular integrity, lipid peroxide concentration, endothelial NO synthase (eNOS) expression and biochemical profiles was examined at day 1, day 21 and day 42 (five rats per group). The histochemical staining ofthe rat aorta showed no change at day 1 and day 21, but the subendothelial space was observed to be enlarged in rat aorta at day 42 with exposure to homocysteine thiolactone. Expression of eNOS was observed in rat aorta at day 42, but not at day 1 and day 21. Serum lipid peroxide concentration and biochemical profiles including glucose cholesterol and triacylglycerol showed no change at any day. Second, the effect of homocysteine thiolactone intake in the presence and absence of vitamin B6 on vascular integrity was examined at day 1 and day 14 (five rats per group). Aortic lesions were observed in vitamin B6-deficient rat aorta at day 14 but not in vitamin B6-supplemented rats. The expression of eNOS was also observedin vitamin B6-deficient rat aorta at day 14. Serum lipid concentrations of the vitamin B6-deficient group significantly increased compared with concentrations of the vitamin B6-supplemented group, though serum concentration of homocysteine did not change between both groups. These results suggest that the oxidative stress caused by a low level of vitamin B6 accelerates the development of homocysteine-induced atherosclerosis in rats.

scholarly journals Endonucleolytic cleavage in the expansion segment 7 of 25S rRNA is an early marker of low-level oxidative stress in yeast

2017 ◽  
Vol 292 (45) ◽  
pp. 18469-18485 ◽  
Author(s):  
Daniel Shedlovskiy ◽  
Jessica A. Zinskie ◽  
Ethan Gardner ◽  
Dimitri G. Pestov ◽  
Natalia Shcherbik
Keyword(s):  
Oxidative Stress ◽  
Low Level ◽  
Early Marker ◽  
Endonucleolytic Cleavage ◽  
Expansion Segment
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