scholarly journals A biopsychosocial perspective on parenting and developmental psychopathology

2013 ◽  
Vol 25 (4pt2) ◽  
pp. 1399-1414 ◽  
Author(s):  
Susan D. Calkins ◽  
Cathi Propper ◽  
W. Roger Mills-Koonce

AbstractAlthough considerable research has examined the relations between parental behavior and a range of child developmental outcomes, much of this work has been conducted at a very broad level of behavioral analysis. A developmental psychopathology framework and recent research conducted within this framework point to the need for models of parenting and child psychopathology that offer greater specificity regarding processes that may be implicated in the effects of these relationships. In addition, recent animal work and some human work has focused more on theproximalbiological and social mechanisms through which parenting affects child outcomes. Our conceptualization of parenting effects acknowledges that family and child factors are embedded in a dynamic biological and social context that is key to understanding developmental trajectories of child adjustment. In this paper, we review two areas of research that are illuminating the biological processes underlying links between parenting and child psychopathology: molecular genetics and psychophysiology. We adopt a biopsychosocial perspective on developmental psychopathology that implies that a set of hierarchically organized, but reciprocally interacting, processes, from the genetic to the environmental, provide the essential elements of both normative and nonnormative development (Gottlieb, 2007). New directions stimulated by this general approach are discussed, with an emphasis on the contextual and developmental issues and applications implied by such a perspective.

2020 ◽  
pp. 1-14
Author(s):  
Florian Scharpf ◽  
Getrude Mkinga ◽  
Frank Neuner ◽  
Maregesi Machumu ◽  
Tobias Hecker

Abstract Maltreatment by parents can be conceptualized as pathogenic escalations of a disturbed parent–child relationship that have devastating consequences for children's development and mental health. Although parental psychopathology has been shown to be a risk factor both for maltreatment and insecure attachment representations, these factors` joint contribution to child psychopathology has not been investigated. In a sample of Burundian refugee families living in refugee camps in Western Tanzania, the associations between attachment representations, maltreatment, and psychopathology were examined by conducting structured interviews with 226 children aged 7 to 15 and both their parents. Structural equation modeling revealed that children's insecure attachment representations and maltreatment by mothers fully mediated the relation between maternal and child psychopathology [model fit: comparative fit index (CFI) = 0.96; root mean square error of approximation (RMSEA) = 0.05]. A direct association between paternal and child psychopathology was observed (model fit: CFI = 0.96; RMSEA = 0.05). The findings suggest a vicious cycle, wherein an insecure attachment to a mother suffering from psychopathology may be linked to children's risk to be maltreated, which may reinforce insecure representations and perpetuate the pathogenic relational experience. Interventions targeting the attachment relationship and parental mental health may prevent negative child outcomes.


Author(s):  
Benjamin L. Hankin

Information on depression during childhood and adolescence is reviewed from a developmental psychopathology perspective. The chapter covers descriptive and diagnostic features, as well as considering key concerns in the assessment of depression from childhood through adolescence. Issues of developmental epidemiology, including prevalence rates, developmental trajectories, sex differences, and developmental patterns of sequential comorbidity, are reviewed. Multiple risks and vulnerabilities, with particular attention to the developmental relevance of these factors and processes in children and adolescence, are considered. Finally, future directions are emphasized, including continued investigation of transdiagnostic versus specific etiologies, employing multiple levels of analysis, and translating risk knowledge into personalization of evidence-based, developmentally sensitive assessment and intervention approaches.


2014 ◽  
Vol 33 (1) ◽  
pp. 77-86 ◽  
Author(s):  
Karen B. MacLeod ◽  
E. B. Brownlie

This paper provides a review of mental health and developmental issues specific to the phases of adolescence and emerging adulthood. Prevalence estimates highlight the frequency and severity of mental health difficulties in this age cohort, and point to the pressing need to integrate a developmental perspective in planning support, prevention, and intervention services. A brief overview of common mental health issues is provided, as well as a consideration of relevant developmental trajectories and variations. Using a health equity lens, we discuss diversity issues in relation to transition-aged youth. Conclusions include recommendations to enhance the system of care for transition-aged youth.


2018 ◽  
Vol 61 (11) ◽  
pp. 2615-2628 ◽  
Author(s):  
Amy M. Wetherby ◽  
Juliann Woods ◽  
Whitney Guthrie ◽  
Abigail Delehanty ◽  
Jennifer A. Brown ◽  
...  

PurposeThe need for community-viable, evidence-based intervention strategies for toddlers with autism spectrum disorder (ASD) is a national priority. The purpose of this research forum article is to identify gaps in intervention research and needs in community practice for toddlers with ASD, incorporate published findings from a randomized controlled trial (RCT) of the Early Social Interaction (ESI) model (Wetherby et al., 2014) to illustrate community-based intervention, report new findings on child active engagement from the ESI RCT, and offer solutions to bridge the research-to-community practice gap.MethodResearch findings were reviewed to identify gaps in the evidence base for toddlers with ASD. Published and new findings from the multisite ESI RCT compared the effects of two different ESI conditions for 82 toddlers with ASD to teach parents how to support active engagement in natural environments.ResultsThe RCT of the ESI model was the only parent-implemented intervention that reported differential treatment effects on standardized measures of child outcomes, including social communication, developmental level, and adaptive behavior. A new measure of active engagement in the natural environment was found to be sensitive to change in 3 months for young toddlers with ASD and to predict outcomes on the standardized measures of child outcomes. Strategies for utilizing the Autism Navigator collection of web-based courses and tools using extensive video footage for families and professional development are offered for scaling up in community settings to change developmental trajectories of toddlers with ASD.ConclusionsCurrent health care and education systems are challenged to provide intervention of adequate intensity for toddlers with ASD. The use of innovative technology can increase acceleration of access to evidence-based early intervention for toddlers with ASD that addresses health disparities, enables immediate response as soon as ASD is suspected, and rapidly bridges the research-to-practice gap.Presentation Videohttps://doi.org/10.23641/asha.7297817


2018 ◽  
Vol 30 (5) ◽  
pp. 1729-1747 ◽  
Author(s):  
Kathryn Lemery-Chalfant ◽  
Sierra Clifford ◽  
Thomas J. Dishion ◽  
Daniel S. Shaw ◽  
Melvin N. Wilson

AbstractDevelopment involves synergistic interplay among genotypes and the physical and cultural environments, and integrating genetics into experimental designs that manipulate the environment can improve understanding of developmental psychopathology and intervention efficacy. Consistent with differential susceptibility theory, individuals can vary in their sensitivity to environmental conditions including intervention for reasons including their genotype. As a consequence, understanding genetic influences on intervention response is critical. Empirically, we tested an interaction between a genetic index representing sensitivity to the environment and the Family Check-Up intervention. Participants were drawn from the Early Steps Multisite randomized prevention trial that included a low-income and racially/ethnically diverse sample of children and their families followed longitudinally (n = 515). As hypothesized, polygenic sensitivity to the environment moderated the effects of the intervention on 10-year-old children's symptoms of internalizing psychopathology, such that children who were genetically sensitive and were randomly assigned to the intervention had fewer symptoms of child psychopathology than genetically sensitive children assigned to the control condition. A significant difference in internalizing symptoms assessed with a clinical interview emerged between the intervention and control groups for those 0.493 SD above the mean on polygenic sensitivity, or 25% of the sample. Similar to personalized medicine, it is time to understand individual and sociocultural differences in treatment response and individualize psychosocial interventions to reduce the burden of child psychopathology and maximize well-being for children growing up in a wide range of physical environments and cultures.


Author(s):  
F. Daniel Armstrong

Cancer, sickle cell disease (SCD), hemophilia, and other blood-related and immunologic disorders represent some of the most complex medical conditions of childhood. All involve a diagnosis that is directly associated with a genetic risk that is interpreted within a complex family system. All have complex biology involving multiple organ systems, and all are potentially fatal. All involve treatment that is demanding, both biologically and behaviorally. All inevitably alter the normal course of development, often during critical periods in the lives of children and families. All have potential significant economic and social consequences that include costs of treatment, indirect costs associated with disease management, and potential long-term costs associated with disability. All have potential long-term effects of treatment that may involve additional new diseases or disabilities. Surprisingly, however, hematologic and oncologic diseases of childhood have one other commonality; despite the complexity and high potential for devastating biologic, psychosocial, family, and economic consequences, all have affected individuals and families who do not experience these devastating consequences and in fact demonstrate a biologic and psychologic resiliency that defies conventional wisdom. Understanding the complex interactions among genetic risk; biology of disease; effectiveness and outcome of treatment; child and family coping, adjustment, and resilience; developmental trajectories; and community support is the challenge for investigators and clinicians during this century, particularly as basic advances in diagnosis and treatment result in anticipation of probable survival for the vast majority of children with these conditions. It is for this reason that these diseases of childhood are frequently considered from a biopsychosocial perspective (Engel, 1980), although we argue that this term must be expanded to incorporate developmental complexity, particularly when applied to children. Since Engel (1980) first proposed a biopsychosocial model of illness as a conceptual model for understanding and treating functional gastrointestinal disorders, our understanding of chronic illness has increasingly incorporated this perspective. The biopsychosocial model recognizes that illness, and one’s experience of illness, occurs through a dynamic interaction among biologic, psychologic, social, and environmental factors, all of which overlap as potential causes and maintenance factors of symptoms associated with the illness.


1990 ◽  
Vol 2 (4) ◽  
pp. 349-366 ◽  
Author(s):  
Marian Radke-Yarrow ◽  
Carolyn Zahn-Waxler

AbstractResearch in developmental psychopathology is used to examine and propose questions, concepts, and methods in the investigation of child development in the contexts of dysfunctional and well families. The adequacy of the data base for investigating the course of development, representing the socialization process, and identifying individualities and universals in development is discussed. A number of research issues that have been studied primarily in developmental psychopathology are recommended as relevant to normal child development. Multidomain and multisource longitudinal data are proposed as the means for better delineating development and for testing alternative models of developmental processes. Examples of data and experience are drawn from longitudinal studies of affectively ill parents and their children. Differences in the perspectives and approaches of normal child development research and developmental child psychopathology are discussed. The usefulness of thinking of two disciplines, normal child development and child development psychopathology, is questioned.


1993 ◽  
Vol 5 (1-2) ◽  
pp. 79-89 ◽  
Author(s):  
Carolyn Zahn-Waxler

AbstractAntisocial behaviors in females may differ from more prototypically “male” patterns of aggression, violence, and criminality that dominate criteria for conduct problems in diagnostic systems. This raises questions of how to define and investigate conduct problems in females as well as how to identify differential childhood antecedents. A developmental psychopathology perspective is advanced as one useful approach to understanding adaptive and maladaptive social patterns in males and females that may lead to different developmental trajectories and influence the forms of psychopathology that develop. The utility of de-emphasizing serious physical aggression as one important criteria for conduct problems is questioned. Recommendations that particular forms of deviance in females be considered as markers of their antisocial patterns (e.g., somatic complaints, friendlessness, underachievement) are also called into question. Rather than adopt sex-specific criteria to assess conduct problems, it is necessary to expand and broaden the diagnostic categories to include serious externalizing problems regardless of whether they occur in males or females.


2021 ◽  
pp. 089020702110129
Author(s):  
Lavinia E Damian ◽  
Oana Negru-Subtirica ◽  
Eleonora I Pop ◽  
Joachim Stoeber

Perfectionism is a pervasive and prevalent personality disposition with high implications for psychological maladjustment. Adolescence represents a particularly relevant period for the development of perfectionism, and perceived parental behaviors have been shown to play an important part. Yet, so far only few longitudinal studies have investigated the role of risk and protective parental behaviors in the development of perfectionism in adolescents. Examining a sample of 744 adolescents ( Mage = 15.2 years), the present study investigated developmental trajectories of self-oriented, socially prescribed, and other-oriented perfectionism over four waves spaced five to six months apart. Results of growth mixture modeling showed that self-oriented perfectionism followed three developmental trajectories (low and decreasing; medium and decreasing; high and stable) as did socially prescribed perfectionism (low and stable; medium and increasing; high and stable). Other-oriented perfectionism showed four developmental trajectories (low and decreasing; low and stable; high and stable; high and increasing). Significant differences were observed between groups regarding all investigated perceived parental behaviors (psychological control, behavioral control, responsiveness, and autonomy support). Similarities and differences between the development of each form of perfectionism and the role of each parental behavior as well as implications of these findings for the understanding of the development of perfectionism in adolescence are discussed.


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