Enhancement of crystalline plane orientation in silsesquioxane-containing vaterite particles towards tuning of calcium ion release

2014 ◽  
Vol 2 (9) ◽  
pp. 1250 ◽  
Author(s):  
J. Nakamura ◽  
T. Kasuga
Keyword(s):  
Calcium Ion ◽  
Ion Release ◽  
Plane Orientation ◽  
Crystalline Plane

scholarly journals Correction: Rajasekharan, S., et al. Effect of Exposed Surface Area, Volume and Environmental pH on the Calcium Ion Release of Three Commercially Available Tricalcium Silicate Based Dental Cements. Materials 2018, 11, 123

Materials ◽  
2021 ◽  
Vol 14 (2) ◽  
pp. 340
Author(s):  
Sivaprakash Rajasekharan ◽  
Chris Vercruysse ◽  
Luc Martens ◽  
Ronald Verbeeck
Keyword(s):  
Surface Area ◽  
Calcium Ion ◽  
Tricalcium Silicate ◽  
Exposed Surface ◽  
Ion Release ◽  
Exposed Surface Area

The authors wish to make the following correction to the paper [...]

scholarly journals pH and calcium ion release evaluation of pure and calcium hydroxide-containing Epiphany for use in retrograde filling

2011 ◽  
Vol 19 (1) ◽  
pp. 1-5 ◽  
Author(s):  
Mário Tanomaru-Filho ◽  
Juliana Nogueira Saçaki ◽  
Frederico Bordini Chaves Faleiros ◽  
Juliane Maria Guerreiro-Tanomaru
Keyword(s):  
Calcium Hydroxide ◽  
Calcium Ion ◽  
Ion Release ◽  
Retrograde Filling

scholarly journals THE EFFECT OF LACTIC ACID AND ARTIFICIAL SALIVA SOLUTION IMMERSION TO THE RELEASE OF CALCIUM IONS ON BIOACTIVE RESIN

2021 ◽  
Vol 6 (2) ◽  
pp. 190
Author(s):  
Dewi Puspitasari ◽  
Nurah Tajjalia ◽  
Diana Wibowo ◽  
Agung Satria Wardhana
Keyword(s):  
Lactic Acid ◽  
Acid Solution ◽  
Calcium Ions ◽  
Calcium Ion ◽  
Artificial Saliva ◽  
Distilled Water ◽  
Acid Condition ◽  
Ion Release ◽  
Lactic Acid Solution ◽  
Solution Increase

Background: Bioactive resin can release calcium ions when contact with solution media, even in acid condition. In the oral cavity, pH may change into acid condition due to the metabolic results of Streptococcus mutans. The bacteria metabolize carbohydrates into organic acids, one of which is lactic acid. Purpose: Analyze the effect of lactic acid solution and artificial saliva on the number of the release of calcium ions of bioactive resin. Methods: Forty-two specimens (diameter 15 mm x thickness 1 mm; n= 7/group fabricated with Activa™ Bioactive Restorative (Pulpdent). The specimens that meet the criteria were divided into 6 groups. The specimen was immersed for 1 and 7 days in the incubator at 37oC. The number of calcium ion release is measured using titration method. Results: Two Way Anova test and Post Hoc Bonferonni test showed there were significant differences among all group for lactic acid 1 day (4.040 ± 0.360) µg, artificial saliva 1 day (0.640 ± 0.338) µg, distilled water 1 day (1.040 ± 0.504) µg, lactic acid 7 days (5.400 ± 0.312), artificial saliva 7 days (1.640 ± 0.215) µg, distilled water 7 days (3.520± 0.356 µg). Conclusion: There was an influence of lactic acid and artificial saliva on the number of calcium ion releases of bioactive resin. Immersion of bioactive resin in the lactic acid solution increase the calcium ion releases and artificial saliva decrease the calcium ion release compared to distilled water. Keywords: artificial saliva, bioactive resin, calcium ion release, lactic acid

Evaluation of pH and Calcium Ion Release of a Dual-cure Bisphenol A Ethoxylate Dimethacrylate/Mineral Trioxide Aggregate–based Root-end Filling Material

2013 ◽  
Vol 39 (12) ◽  
pp. 1603-1606 ◽  
Author(s):  
Giane da Silva Linhares ◽  
Maximiliano Sérgio Cenci ◽  
César Blaas Knabach ◽  
Camila Mizette Oliz ◽  
Mariana Antunes Vieira ◽  
...  
Keyword(s):  
Bisphenol A ◽  
Calcium Ion ◽  
Mineral Trioxide Aggregate ◽  
Filling Material ◽  
Ion Release

Association of increased pHi with calcium ion release in skeletal muscle

1978 ◽  
Vol 234 (3) ◽  
pp. C110-C114 ◽  
Author(s):  
R. J. Connett
Keyword(s):  
Skeletal Muscle ◽  
Calcium Ion ◽  
Calcium Release ◽  
Muscle Membrane ◽  
Sartorius Muscle ◽  
Ion Release ◽  
Extracellular Medium ◽  
Dantrolene Sodium ◽  
External Potassium ◽  
Frog Sartorius

The pH difference across the cell membrane of frog sartorius muscle cells was measured with the distribution of 5,5-dimethyl-2,4-oxazolidine-dione (DMO) as the marker. Depolarization of the muscles to values at or below the contraction threshold caused by elevating external potassium up to approximately 20 mM resulted in an internal alkalinization. The change was smaller with superthreshold depolarization (20--30 mM [K+]). The alkalinization was blocked by agents that block calcium release from the sarcoplasmic reticulum (procaine and dantrolene sodium). Other agents that cause calcium release (caffeine, theophylline, and quinine) were found to give alkalinization when tested at concentrations just below the contracture threshold. Increased acidification of the extracellular medium was associated with the internal alkalinization. The data were interpreted as indicating the presence of a calcium-stimulated H+ and/or OH- ion transport system in the muscle membrane.

scholarly journals Comparison of calcium ion release from MTA-Angelus® and Biodentine®

2018 ◽  
Vol 1073 ◽  
pp. 052008 ◽  
Author(s):  
I Aprillia ◽  
M Usman ◽  
D Asrianti
Keyword(s):  
Calcium Ion ◽  
Ion Release

Malignant Hyperpyrexia during General Anesthesia

1973 ◽  
Vol 82 (5) ◽  
pp. 729-733
Author(s):  
Robert H. Maisel ◽  
Donald G. Sessions ◽  
Robert N. Miller
Keyword(s):  
General Anesthesia ◽  
Calcium Ion ◽  
Autosomal Dominant ◽  
Treatment Protocol ◽  
Enzyme Activation ◽  
Malignant Hyperpyrexia ◽  
Ion Release ◽  
Anesthetic Agents ◽  
Parenteral Delivery ◽  
Support Measures

Malignant hyperpyrexia during general anesthesia requires emergency treatment. The hyperthermic trait is due to an inborn error of metabolism with variably expressed autosomal dominant inheritance, as well as a sporadic incidence. Volatile inhalant anesthetic agents are thought to produce abnormal calcium ion release in susceptible individuals. Resultant enzyme activation and oxidative phosphorylation then cause hypermetabolism and produce fever to 108° and higher. Combined morbidity and mortality of the syndrome is still in excess of 75%. A treatment protocol based on the presently understood pathophysiology of the disease is described. It includes parenteral delivery of procaine, corticosteroids and Thorazine® as well as routine support measures. Herald signs of this syndrome, such as abnormal anesthetic induction with masseter muscle spasm suggest curtailing the operation or obtaining full consultation.

Sign in / Sign up

Export Citation Format

Share Document