Sunlight-activated long persistent luminescence in the ultraviolet-B from Bi3+-doped garnet phosphors for covert optical tagging

Author(s):  
Jingwei Liu ◽  
Yanjie Liang ◽  
Shao Yan ◽  
Dongxun Chen ◽  
Shihai Miao ◽  
...  

Ultraviolet (UV) luminescent materials hold great promise for applications across a wide number of fields, whether environmental, biological or medical. However, luminescent materials with the capability to continue emitting UV...

2021 ◽  
Author(s):  
Jingwei Liu ◽  
Yanjie Liang ◽  
Shao Yan ◽  
Dongxun Chen ◽  
Shihai Miao ◽  
...  

Luminescent materials that emit in the narrowband ultraviolet-B (NB-UVB; 310–313 nm) spectral region have attracted considerable attention due to their unique spectral features, which endow them with great potential applications...


Author(s):  
Yaling Lin ◽  
Jie Hu ◽  
Luyan Wu ◽  
Qilin Zou ◽  
Dejian Chen ◽  
...  

Persistent luminescence nanoparticles (PLNPs) emitting in the NIR window (700 - 1700 nm) have shown great promise in the field of fluorescence imaging due to their unique properties including the...


2020 ◽  
Author(s):  
Sheikh Ahmad Umar ◽  
Sheikh Abdullah Tasduq

AbstractUltraviolet-B (UV-B) exposure to skin causes photo-damage and acts as the primary etiological agent in photo-carcinogenesis. UV-B exposure induces photodamage in epidermal cells and is the major factor that challenges skin homeostasis. Autophagy allows fundamental adaptation of cells to metabolic needs and stresses. Cellular dysfunction is observed in aged tissues and in toxic insults to cells that undergo through stress. Conversely, promising anti-aging strategies aimed at inhibiting the mTOR pathway has been found to significantly improve the aging related disorders. Recently, autophagy has been found to positively regulate skin homeostasis by enhancing DNA damage recognition. Here we investigated the Geno-protective roles of autophagy in UV-B exposed primary HDFs. We found that improving autophagy levels in HDFs regulates UV-B mediated cellular stress by decreasing the formation of DNA photo adducts, alleviates oxidative and ER stress response and by regulating the expression levels of cell cycle regulatory proteins P21 and P27. Autophagy also prevents HDFs from UV-B -induced nuclear damage as is evident from Tunnel assay and Acridine Orange/Ethidium Bromide co-staining. Salubrinal, (an eIf2α inhibitor) significantly decreases the DNA damage response in HDFs. P62 silenced HDFs show enhanced DNA damage response and disturbs the tumor suppressor axis PTEN/pAKT towards damage whereas ATG7 silenced HDFs reveal an unexpected consequence by decreasing the UV-B -induced DNA damage compared to UV-B treated HDFs. Together, our results suggest that autophagy is essential in protecting skin cells from UV-B radiation -induced photo-damage and holds great promise in devising it as a suitable therapeutic strategy against skin photo-damage.HighlightsAutophagy is an immediate molecular event induced following exposure of primary HDFs to UV-B –irradiationAutophagy offers pro-survival capacity to HDFs under UV-B induced genotoxic stressAutophagy regulates DNA Damage Response via regulation of oxidative and ER stress in UV-B exposed HDFsRelieving ER stress response offers significant protection to primary HDFs from UV-B by decreasing the DNA damageAutophagy deprivation to HDFs via P62 silencing potentiates UV-B -induced DNA damage responseATG7 silencing in UV-B exposed HDFs unexpectedly alleviates the DNA Damage Response in primary HDFs


Author(s):  
Chiara Chiatti ◽  
Claudia Fabiani ◽  
Anna Laura Pisello

In recent decades, research on persistent luminescence has led to new phosphors and promising performance. Efforts to improve the quality of phosphors’ afterglow have paved the way toward innovative solutions for many disciplines. However, there are few examples of the implementation of luminescent materials. In addition to providing a general background on persistent luminescence, the techniques used for its analysis, and its multidisciplinary potential in energy and environmental science, this article aims to explain the existing gap between the physical-chemical approach and the effective implementation of luminescent materials in larger-scale applications. It investigates engineering solutions in terms of the possible benefits of luminescence in lighting energy savings and passive cooling of urban surfaces. Finally, this article aims to reduce the abovementioned gap by suggesting what is most needed for the successful application of luminescent materials in the built environment. Expected final online publication date for the Annual Review of Materials Science, Volume 51 is July 2021. Please see http://www.annualreviews.org/page/journal/pubdates for revised estimates.


2020 ◽  
Vol 8 (3) ◽  
pp. 1143-1150 ◽  
Author(s):  
Zhizhen Wang ◽  
Zhen Song ◽  
Lixin Ning ◽  
Quanlin Liu

A novel warm-color persistent luminescent materials Nb-doped Sr3SiO5:Eu2+, with peak wavelength at ∼580 nm and persistent time more than 7 hours at the 0.32 mcd m−2 threshold value after sunlight radiation has been reported.


2021 ◽  
Author(s):  
Rui Gao ◽  
Mohamad S. Kodaimati ◽  
Dongpeng Yan

In this review, we summarize recent advances in establishing persistently luminescent materials from the view of examining experimental and theoretical approaches to room-temperature phosphorescence and thermally-activated delayed fluorescence.


2021 ◽  
Vol 11 ◽  
Author(s):  
Sheikh Ahmad Umar ◽  
Naikoo Hussain Shahid ◽  
Lone Ahmad Nazir ◽  
Malik Ahmad Tanveer ◽  
Gupta Divya ◽  
...  

Ultraviolet (UV) exposure to the skin causes photo-damage and acts as the primary etiological agent in photo-carcinogenesis. UV-B exposure induces cellular damage and is the major factor challenging skin homeostasis. Autophagy allows the fundamental adaptation of cells to metabolic and oxidative stress. Cellular dysfunction has been observed in aged tissues and in toxic insults to cells undergoing stress. Conversely, promising anti-aging strategies aimed at inhibiting the mTOR pathway have been found to significantly improve the aging-related disorders. Recently, autophagy has been found to positively regulate skin homeostasis by enhancing DNA damage recognition. Here, we investigated the geno-protective roles of autophagy in UV-B-exposed primary human dermal fibroblasts (HDFs). We found that UV-B irradiation to HDFs impairs the autophagy response in a time- and intensity-independent manner. However, improving autophagy levels in HDFs with pharmacological activators regulates the UV-B-induced cellular stress by decreasing the induction of DNA photo-adducts, promoting the DNA repair process, alleviating oxidative and ER stress responses, and regulating the expression levels of key cell cycle regulatory proteins. Autophagy also prevents HDFs from UV-B-induced nuclear damage as is evident in TUNEL assay and Acridine Orange/Ethidium Bromide co-staining. Salubrinal (an eIF2α phosphatase inhibitor) relieves ER stress response in cells and also significantly alleviates DNA damage and promotes the repair process in UV-B-exposed HDFs. P62-silenced HDFs show enhanced DNA damage response and also disturb the tumor suppressor PTEN/pAKT signaling axis in UV-B-exposed HDFs whereas Atg7-silenced HDFs reveal an unexpected consequence by decreasing the UV-B-induced DNA damage. Taken together, these results suggest that interventional autophagy offers significant protection against UV-B radiation-induced photo-damage and holds great promise in devising it as a suitable therapeutic strategy against skin pathological disorders.


2020 ◽  
Vol 10 (5) ◽  
pp. 1296 ◽  
Author(s):  
Hongxu Sun ◽  
Qingqing Gao ◽  
Aiying Wang ◽  
Yichun Liu ◽  
Xiao-jun Wang ◽  
...  

2020 ◽  
Vol 217 ◽  
pp. 116785 ◽  
Author(s):  
Ou Hai ◽  
Enlong Yang ◽  
Dongming Li ◽  
Wenni Bai ◽  
Qiang Ren ◽  
...  

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