scholarly journals Transcriptional Roles of CCAAT/Enhancer Binding Protein-β, Nuclear Factor-κB, and C-promoter Binding Factor 1 in Interleukin (IL)-1β-induced IL-6 Synthesis by Human Rheumatoid Fibroblast-like Synoviocytes

1998 ◽  
Vol 273 (13) ◽  
pp. 7620-7627 ◽  
Author(s):  
Keiji Miyazawa ◽  
Akio Mori ◽  
Kazuhiko Yamamoto ◽  
Hirokazu Okudaira
Keyword(s):  
Binding Protein ◽  
Nuclear Factor Κb ◽  
Nuclear Factor ◽  
Enhancer Binding Protein ◽  
Binding Factor ◽  
Ccaat Enhancer Binding Protein ◽  
Fibroblast Like Synoviocytes

CCAAT/Enhancer Binding Protein α (C/EBPα) and C/EBPα Myeloid Oncoproteins Induce Bcl-2 via Interaction of Their Basic Regions with Nuclear Factor-κB p50

2005 ◽  
Vol 3 (10) ◽  
pp. 585-596 ◽  
Author(s):  
Ido Paz-Priel ◽  
Dong Hong Cai ◽  
Dehua Wang ◽  
Jeanne Kowalski ◽  
Amanda Blackford ◽  
...  
Keyword(s):  
Binding Protein ◽  
Nuclear Factor Κb ◽  
Nuclear Factor ◽  
Enhancer Binding Protein ◽  
Ccaat Enhancer Binding Protein

scholarly journals Regulation of the Human Oxytocin Receptor by Nuclear Factor-κB and CCAAT/Enhancer-Binding Protein-β

2006 ◽  
Vol 91 (6) ◽  
pp. 2317-2326 ◽  
Author(s):  
Vasso Terzidou ◽  
Yooni Lee ◽  
Tamsin Lindström ◽  
Mark Johnson ◽  
Steven Thornton ◽  
...  
Keyword(s):  
Binding Protein ◽  
Nuclear Factor Κb ◽  
Oxytocin Receptor ◽  
Nuclear Factor ◽  
Enhancer Binding Protein ◽  
Ccaat Enhancer Binding Protein

scholarly journals CCAAT enhancer binding protein β and hepatocyte nuclear factor 3β are necessary and sufficient to mediate dexamethasone-induced up-regulation of alpha2HS-glycoprotein/fetuin-A gene expression

2006 ◽  
Vol 36 (2) ◽  
pp. 261-277 ◽  
Author(s):  
Michael Wöltje ◽  
Beate Tschöke ◽  
Verena von Bülow ◽  
Ralf Westenfeld ◽  
Bernd Denecke ◽  
...  
Keyword(s):  
Serum Protein ◽  
Binding Protein ◽  
Protein A ◽  
Nuclear Factor ◽  
Hepatocyte Nuclear Factor ◽  
Tissue Remodelling ◽  
Mobility Shift ◽  
Fetuin A ◽  
Enhancer Binding Protein ◽  
Ccaat Enhancer Binding Protein

Alpha2HS-glycoprotein/fetuin-A (Ahsg) is a serum protein preventing soft tissue calcification. In trauma and inflammation, Ahsg is down-regulated and therefore considered a negative acute phase protein. Enhancement of Ahsg expression as a protective serum protein is desirable in several diseases including tissue remodelling after trauma and infection, kidney and heart failure, and cancer. Using reporter gene assays in hepatoma cells combined with electrophoretic mobility shift assays we determined that dexamethasone up-regulates hepatic Ahsg. A steroid response unit at position −146/−119 within the mouse Ahsg promoter mediates the glucocorticoid-induced increase of Ahsg mRNA. It binds the hepatocyte nuclear factor 3β and CCAAT enhancer binding protein β (C/EBP-β). The up-regulation is mediated indirectly via glucocorticoid hormone-induced transcriptional up-regulation in C/EBP-β protein. A high degree of sequence identity in mouse, rat and human Ahsg promoters suggests that the promoter is similarly up-regulated by dexamethasone in all three species. Therefore, our findings suggest that glucocorticoids may be used to enhance the level of Ahsg protein circulating in serum.

scholarly journals CCAAT/Enhancer Binding Protein and Nuclear Factor-Y Regulate Adiponectin Gene Expression in Adipose Tissue

Diabetes ◽  
2004 ◽  
Vol 53 (11) ◽  
pp. 2757-2766 ◽  
Author(s):  
S.-k. Park ◽  
S.-Y. Oh ◽  
M.-Y. Lee ◽  
S. Yoon ◽  
K.-S. Kim ◽  
...  
Keyword(s):  
Gene Expression ◽  
Adipose Tissue ◽  
Binding Protein ◽  
Nuclear Factor ◽  
Adiponectin Gene ◽  
Nuclear Factor Y ◽  
Enhancer Binding Protein ◽  
Ccaat Enhancer Binding Protein
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