scholarly journals Internal jugular vein ultrasound in patients with chronic congestive heart failure

2021 ◽  
Vol 42 (Supplement_1) ◽  
Author(s):  
P Thacker ◽  
D Amaratunga ◽  
K Shah ◽  
R Watson ◽  
A Singh ◽  
...  

Abstract Background Bedside assessment of intravascular volume in patients with chronic congestive heart failure (CHF) is often difficult. Under- and over-diuresis are common causes of morbidity and readmissions in these patients. Purpose We hypothesized that ultrasound assessment of the internal jugular vein would be easier and more reproducible than clinically assessing jugular venous pressure (JVP). Our goal was to create a bedside test that would be simpler to learn than inferior vena cava (IVC) assessment and easier to perform in obese patients. Methods Adults with HF (n=53, 52% men, mean age 65 years, mean BMI 29.6 kg/m2, mean LVEF 44%) scheduled for right heart catheterization (RHC) had an ultrasound of their right internal jugular (RIJ) vein performed immediately prior. Cross-sectional area of RIJ was measured during normal breathing with patients at 90 and 45 degrees recumbency and was indexed by height (RIJI). JVP was also assessed clinically. Results were compared to right atrial pressure (RAP) measured by RHC. Operators were blinded to RHC results and vice versa. Results JVP was correctly assessed clinically in only 43%. RIJI at 90 and 45 degrees were significantly larger in patients with elevated RAP compared to euvolemic patients (Table). At 90 degrees, RIJI of >15 predicted a RAP of >10 mmHg with 68% sensitivity and 72% specificity. At 45 degrees, RIJI of >10 predicted a RAP of >10 mmHg with 94% sensitivity and a negative predictive value of 80% (Table). Simply being able to see the RIJ at 90 degrees (n=34) had an 82.4% positive predictive value for elevated RAP. IVC data could not be obtained on 23% of patients due to body habitus or inability to lay flat. Conclusion Ultrasonographic RIJI is more accurate than clinical assessment in patients with CHF and can be accurately performed even in obese patients. It requires only a basic linear ultrasound probe and was easily performed by clinicians at various stages of training with reproducible results. With the increased availability of bedside ultrasound in clinical practice, it is a feasible method of evaluating chronic CHF patients. FUNDunding Acknowledgement Type of funding sources: None.

1992 ◽  
Vol 73 (4) ◽  
pp. 1637-1643 ◽  
Author(s):  
R. J. Cody ◽  
S. H. Kubo ◽  
J. H. Laragh ◽  
S. A. Atlas

We have previously reported a fivefold increase of plasma atrial natriuretic factor (ANF) in patients with congestive heart failure (CHF) compared with normal subjects. However, given the marked increase of ANF under basal conditions, the extent to which ANF secretion can further increase under physiological stress is not been clarified in CHF. We therefore evaluated ANF secretion during ergometric exercise in 11 patients with CHF, with peripheral venous ANF samples obtained at rest and peak exercise. In seven patients, simultaneous peripheral venous and right ventricular ANF samples were obtained to estimate myocardial ANF secretion. Hemodynamic characteristics of exercise included a significant increase of heart rate, mean arterial pressure, and cardiac output (all P < 0.01); reduction of systemic vascular resistance (P < 0.001); and increase of right atrial and pulmonary wedge pressures (P < 0.001). ANF was abnormally elevated at baseline (108 +/- 58 fmol/ml) yet increased further to 183 +/- 86 fmol/ml with exercise (P < 0.003). A step-up of right ventricular ANF, particularly during exercise, was consistent with active myocardial secretion, despite elevated baseline ANF levels.


1960 ◽  
Vol 199 (4) ◽  
pp. 603-608 ◽  
Author(s):  
Nicholas A. Yankopoulos ◽  
James O. Davis ◽  
Ernest Cotlove ◽  
Mary Trapasso

The mechanism of myocardial edema was studied by measuring the changes in the water and electrolyte content of the myocardium of dogs with chronic congestive heart failure and of dogs with other experimental lesions producing chronic ascites. Adrenocortical tissue was also studied for comparison with the changes observed in heart muscle. A marked increase in the water content and in the Na and Cl per gram of fat free tissue solids of the myocardium occurred in congestive heart failure; the K content was unaltered. Assuming that the increment in Cl was distributed extracellularly, this increase in water and electrolytes was extracellular. Chronic passive congestion of the myocardium secondary to elevated right atrial pressure seems to be a major pathogenic factor in myocardial edema formation although hyperaldosteronemia and hypoproteinemia may also play a role.


1950 ◽  
Vol 29 (3) ◽  
pp. 342-348 ◽  
Author(s):  
Morton H. Maxwell ◽  
Ernest S. Breed ◽  
Irving L. Schwartz

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