The role of the cell-mediated immune response to Actinobacillus actinomycetemcomitans and Porphyromonas gingivalis in periodontitis

1999 ◽  
Vol 20 (1) ◽  
pp. 239-288 ◽  
Author(s):  
Homayoun H. Zadeh ◽  
Frank C. Nichols ◽  
Kenneth T. Miyasaki
Keyword(s):  
Immune Response ◽  
Porphyromonas Gingivalis ◽  
Actinobacillus Actinomycetemcomitans ◽  
Cell Mediated Immune Response

Regulatory role of miR-2909 in cell-mediated immune response

2012 ◽  
Vol 30 (6) ◽  
pp. 500-504 ◽  
Author(s):  
Deepak Kaul ◽  
M. Sasikala ◽  
A. Raina
Keyword(s):  
Immune Response ◽  
Regulatory Role ◽  
Cell Mediated Immune Response

The rôle of cortisol and β-endorphin in the response of the immune system to weaning in lambs

1998 ◽  
Vol 66 (2) ◽  
pp. 397-402 ◽  
Author(s):  
S. M. Rhind ◽  
H. W. Reid ◽  
S. R. McMillen ◽  
G. Palmarini
Keyword(s):  
Immune Response ◽  
Immune Responses ◽  
Stress Hormone ◽  
Keyhole Limpet Haemocyanin ◽  
Weaning Stress ◽  
Cell Mediated Immune Response ◽  
Acth Treatment ◽  
Induced Changes ◽  
The Relationship

AbstractThe relationship between weaning stress-induced changes in stress hormone profiles and immune function was investigated in groups of 10 lambs immunized against adrenocorticotrophic hormone (ACTH; treatment A) or fi-endorphin (treatment B) to reduce the circulating concentrations of cortisol and fi-endorphin respectively. Control animals (treatment C) were immunized against a porcine thyroglobulin carrier protein. Application of weaning stress was associated with significantly elevated plasma cortisol concentrations but no significant increase in fi-endorphin concentrations in C lambs. Immunization against ACTH suppressed the post-weaning increase in cortisol concentration. This was associated with a transient reduction in the lymphocyte stimulation response to keyhole limpet haemocyanin (KLH) antigen in the A animals but there was no effect on the antibody response or interferon-y production by antigen stimulated lymphocytes. There were no significant effects of immunization against fi-endorphin on the capacity to mount antibody or cell-mediated immune responses. It is concluded that weaning stress-induced increases in cortisol did not inhibit the immune response. Since cortisol concentrations and the cell mediated immune response at 8 days after immunization were positively associated it is concluded that these indices are not independent measures of stress.

Role of the cell-mediated immune response in the pathogenesis of hepatitis B virus infection

1995 ◽  
Vol 121 (S1) ◽  
pp. S15-S15
Author(s):  
C. Ferrari ◽  
A. Penna ◽  
A. Bertoletti ◽  
A. Cavalli ◽  
G. Missale ◽  
...  
Keyword(s):  
Immune Response ◽  
Hepatitis B Virus ◽  
Virus Infection ◽  
Hepatitis B ◽  
Hepatitis B Virus Infection ◽  
Cell Mediated Immune Response ◽  
B Virus

Role of prostaglandin in the depressed cell-mediated immune response in rheumatoid arthritis

1980 ◽  
Vol 17 (1) ◽  
pp. 31-37 ◽  
Author(s):  
Steven I. Wolinsky ◽  
James S. Goodwin ◽  
Ronald P. Messner ◽  
Ralph C. Williams
Keyword(s):  
Rheumatoid Arthritis ◽  
Immune Response ◽  
Cell Mediated Immune Response

scholarly journals PERIODONTOPHATOGENIC BACTERIA OF THE MAIN FACTORS OF EMERGENCE AND DEVELOPMENT OF PERIODONTITIS

2017 ◽  
pp. 101-112 ◽  
Author(s):  
V. N. Tsarev ◽  
E. N. Nikolaeva ◽  
E. V. Ippolitov
Keyword(s):  
Porphyromonas Gingivalis ◽  
Quantitative Evaluation ◽  
Inflammatory Diseases ◽  
Aggregatibacter Actinomycetemcomitans ◽  
Actinobacillus Actinomycetemcomitans ◽  
Tannerella Forsythia ◽  
Main Factors ◽  
Level Of Importance ◽  
Development Risk

Presents a modern understanding of the role of periodontophatogenic bacteria types: Aggregatibacter actinomycetemcomitans (Actinobacillus actinomycetemcomitans), Tannerella forsythia (Bacteroides forsythus) and Porphyromonas gingivalis as indicators of periodontitis emergence and development risk. Therminological definitions are given of factors, indicators and markers of risk of periodontium inflammatory diseases development that show the level of importance of their association with the disease as well as the quantitative evaluation of these associations.

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