Permanent neurological deficit caused by misdiagnosis of a giant intrapelvic sciatic schwannoma: A case report

2021 ◽  
Author(s):  
Sijie Yi ◽  
Xia Jin ◽  
Yuanhuan Xiong ◽  
Bing Fu
Keyword(s):  
Case Report ◽  
Neurological Deficit ◽  
Permanent Neurological Deficit

scholarly journals Insulinoma presenting as refractory seizure disorder

F1000Research ◽  
2012 ◽  
Vol 1 ◽  
pp. 15 ◽  
Author(s):  
Pamela Correia ◽  
Roopal Panchani ◽  
Rajeev Ranjan ◽  
Chandrashekhar Agrawal
Keyword(s):  
Differential Diagnosis ◽  
Case Report ◽  
Neurological Deficit ◽  
Seizure Disorder ◽  
Transient Ischemia ◽  
Intractable Seizures ◽  
Chronic Disorders ◽  
Permanent Neurological Deficit ◽  
Treatable Condition ◽  
Delay In Treatment

Hypoglycaemia can lead to acute disorders of cognition, consciousness, epilepsy, transient ischemia, psychosis and chronic disorders of dementia and neuropathy. Misdiagnosis and delay in treatment are common and prolonged hypoglycemia can lead to permanent neurological deficit or fatal coma. Hypoglycemia caused by an insulinoma is a readily treatable condition that should be considered in the differential diagnosis of intractable seizures. The following case report highlights the need for careful reassessment of all seizures that are atypical and refractory to medication.

scholarly journals Neurological morbidity in vaccine-associated paralytic poliomyelitis in Brazil from 1989 up to 1995

2004 ◽  
Vol 62 (2b) ◽  
pp. 414-420 ◽  
Author(s):  
Elza Dias-Tosta ◽  
Carlos Santos Kückelhaus
Keyword(s):  
Virus Infection ◽  
Neurological Deficit ◽  
Strong Relationship ◽  
Vaccine Virus ◽  
Paralytic Poliomyelitis ◽  
Ministry Of Health ◽  
Minimum Risk ◽  
Clinical Pattern ◽  
Neurological Morbidity ◽  
Permanent Neurological Deficit

We collected 30 cases of vaccine associated paralytic poliomyelitis (VAPP) from 4081 cases of acute flaccid palsies cases notified from 1989 to 1995 to the Brazilian Ministry of Health. There were 30 VAPP cases with 56% of children younger than 1 year old, 56.7% of female. 46% of cases were reported in the Northeast. Ten P2 vaccine virus, 8 P3 and 2 P1 and associations amongst them were isolated. The clinical pattern in 60 days was: monoplegia (16), paraplegia (6), tetraplegia (5), hemiplegia (2) and triplegia (1). There was no strong relationship between fever, before or after the prodrome period, or the use of intramuscular medication to morbidity. CONCLUSION: if the anti-poliomyelitis strategy adopted in Brazil has lead to the eradication of the poliomyelitis with wild virus infection, the existence of a minimum risk of vaccine-associated poliomyelitis is a matter of concern because there will be a permanent neurological deficit.

Neurological Deficit after Carotid Infusion of Cisplatin and 1,3-Bis(2-chloroethyl)-1-nitrosourea (BCNU) for Malignant Glioma: An Analysis of Risk Factors

Neurosurgery ◽  
1986 ◽  
Vol 19 (5) ◽  
pp. 779-783 ◽  
Author(s):  
P. Kapp John ◽  
A. Sanford Robert
Keyword(s):  
Risk Factors ◽  
Neurological Deficit ◽  
Infusion Pump ◽  
Malignant Gliomas ◽  
Internal Capsule ◽  
Neurological Deficits ◽  
Infusion Therapy ◽  
Infusion Line ◽  
Permanent Neurological Deficit ◽  
Infusion Technique

Abstract The records of 24 patients with malignant gliomas treated with carotid infusion of cisplatin and 1,3-bis(2-chloroethyl)-1-nitrosourea (BCNU) are reviewed for risk factors that might suggest the development of a permanent neurological deficit after infusion. Permanent neurological deficits were seen with doses of cisplatin as low as 69 mg/m2, although doses as high as 100 mg/m2 were tolerated by other patients. All 3 patients who developed permanent neurological deficits received fixed doses of cisplatin of 150 mg and supplied only 2 major intracranial branches from the infused carotid artery. In none of these patients was a filter used in the infusion line. Other risk factors identified in 2 of the 3 patients were diffuse neoplasm involving the region of the internal capsule and the use of an infusion pump rather than a pulsatile bolus infusion technique. The development of a permanent neurological deficit appeared unrelated to the dose of BCNU within the range utilized, and preinfusion administration of corticosteroids did not prevent neurological deficit. These possible risk factors should be considered in the future development of protocols for arterial infusion therapy of malignant gliomas.

scholarly journals Brain abscess due to Aggregatibacter aphrophilus and Bacteroides uniformis

2015 ◽  
Vol 44 (2) ◽  
pp. 181
Author(s):  
Maja Bogdan ◽  
Vlasta Zujić Atalić ◽  
Ivan Hećimović ◽  
Dubravka Vuković
Keyword(s):  
Magnetic Resonance Imaging ◽  
Case Report ◽  
Surgical Treatment ◽  
Brain Abscess ◽  
Neurological Deficit ◽  
Resonance Imaging ◽  
Aggregatibacter Aphrophilus ◽  
Threatening Condition ◽  
Life Threatening ◽  
The Brain

<p><strong>Objective</strong>. The aim of this report was to describe the occurrence of a bacterial brain abscess in a healthy individual, without any predisposing condition. <strong>Case report</strong>. A thirteen-year old boy was admitted to the Department of Neurosurgery after the onset of vomiting, headache and dizziness. A neurological deficit was detected during the physical examination so urgent magnetic resonance imaging of the brain was performed, revealing an intrahemispheric, right positioned solitary expansive mass with ring enhancement. Purulent material was obtained during osteoplastic craniotomy with total extirpation of the brain abscess. Aggregatibacter aphrophilus and Bacteroides uniformis were isolated. The patient’s general condition improved and the neurological deficit subsided as a result of the prompt recognition and treatment of this life threatening condition. <strong>Conclusion</strong>. To achieve a favourable clinical outcome, prompt recognition and surgical treatment of a brain abscess are of primary importance,followed by administration of appropriate antimicrobial therapy. To our best knowledge, this is the first report of this combination of microorganisms as the cause of a brain abscess.</p>

Focal Neurological Deficit with Sudden Onset as the First Manifestation of Sarcoidosis: A Case Report with MRI Follow-Up

1991 ◽  
Vol 31 (6) ◽  
pp. 376-379 ◽  
Author(s):  
A. Michotte ◽  
P. Dequenne ◽  
D. Jacobovitz ◽  
J. Hildebrand
Keyword(s):  
Case Report ◽  
Neurological Deficit ◽  
Sudden Onset ◽  
Focal Neurological Deficit

scholarly journals Penetrating knife injury to the frontal lobe – a case report

2010 ◽  
Vol 92 (6) ◽  
pp. e41-e42 ◽  
Author(s):  
Aakshay Gulati ◽  
Badri Srinivasan ◽  
Roger Hunter ◽  
Timothy R Flood
Keyword(s):  
Case Report ◽  
Frontal Lobe ◽  
Neurological Deficit ◽  
Cerebral Circulation ◽  
Penetrating Injuries ◽  
Stab Injury ◽  
Close Proximity ◽  
Cranial Fossa ◽  
Knife Injury ◽  
Orbital Region

Penetrating injuries of the craniofacial region are increasing and have the potential to cause severe vascular and neurological deficit. We present our management of a case with a knife stab injury to the infra-orbital region, traversing the orbit and penetrating into the anterior cranial fossa, the tip lying in close proximity to the anterior cerebral circulation.

Surgery for Unruptured Spetzler-Martin Grade 3 Brain Arteriovenous Malformations

Neurosurgery ◽  
2015 ◽  
Vol 77 (3) ◽  
pp. 362-370 ◽  
Author(s):  
Michael Kerin Morgan ◽  
Nazih Assaad ◽  
Miikka Korja
Keyword(s):  
Neurological Deficit ◽  
Arteriovenous Malformations ◽  
Late Recurrence ◽  
Adverse Outcomes ◽  
Brain Arteriovenous Malformations ◽  
Complete Obliteration ◽  
Martin Grade ◽  
Grade 3 ◽  
Permanent Neurological Deficit

Abstract BACKGROUND: There is uncertainty regarding the management of unruptured Spetzler-Martin grade 3 brain arteriovenous malformations (SMG3 ubAVM). OBJECTIVE: To analyze our series of patients treated by surgery. METHODS: A single-surgeon database of consecutively enrolled bAVMs (between 1989 and 2014) was analyzed. Adverse outcomes due to surgery were assigned within the first 6 weeks following surgery and outcome was prospectively recorded and assigned at the last follow-up visit by using modified Rankin Scale (mRS) score. RESULTS: Of the 137 reviewed patients, 112 (82%) were treated by surgery, 15 (11%) were treated elsewhere or by radiosurgery, and 10 (7%) were recommended for conservative management. Surgery for SMG3 ubAVM was associated with adverse outcomes with a new permanent neurological deficit of mRS &gt;1 in 23 of 112 (21%) patients. Permanent neurological deficit leading to a mRS &gt;2 from surgery was 3.6% (95% confidence interval, 1.1%-9.1%). Late recurrence of a bAVM occurred in 3 of 103 (2.9%) patients who had complete obliteration of bAVM confirmed immediately after surgery and who were subsequently later followed with radiological studies during the mean follow-up period of 3.0 years (range, 6 days to 18.8 years). CONCLUSION: When discussing surgical options for SMG3 ubAVM, a thorough understanding of the significance and incidence of adverse events and outcomes is required to fully inform patients. For our series, the additional subclassification of SMG ubAVM (based on variables contributing to the SMG or age) would not have been of use.

scholarly journals Clip reconstruction of a large right MCA bifurcation aneurysm. Case report

2014 ◽  
Vol 21 (2) ◽  
pp. 163-167
Author(s):  
A. Giovani ◽  
Angela Neacsu ◽  
Ana Gheorghiu ◽  
R.M. Gorgan
Keyword(s):  
Case Report ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Neurological Deficit ◽  
Sylvian Fissure ◽  
Pterional Craniotomy ◽  
Bifurcation Aneurysm ◽  
Temporary Clip

Abstract We report a case of complex large middle cerebral artery (MCA) bifurcation aneurysm that ruptured during dissection from the very adherent MCA branches but was successfully clipped and the MCA bifurcation reconstructed using 4 Yasargill clips. Through a right pterional craniotomy the sylvian fissure was largely opened as to allow enough workspace for clipping the aneurysm and placing a temporary clip on M1. The pacient recovered very well after surgery and was discharged after 1 week with no neurological deficit. Complex MCA bifurcation aneurysms can be safely reconstructed using regular clips, without the need of using fenestrated clips or complex by-pass procedures.

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