scholarly journals Four A rabidopsis   AREB / ABF transcription factors function predominantly in gene expression downstream of SnRK2 kinases in abscisic acid signalling in response to osmotic stress

2014 ◽  
Vol 38 (1) ◽  
pp. 35-49 ◽  
Author(s):  
TAKUYA YOSHIDA ◽  
YASUNARI FUJITA ◽  
KYONOSHIN MARUYAMA ◽  
JUNRO MOGAMI ◽  
DAISUKE TODAKA ◽  
...  
Keyword(s):  
Gene Expression ◽  
Transcription Factors ◽  
Abscisic Acid ◽  
Osmotic Stress

scholarly journals The Transcription Factors HvABI5 and HvVP1 Are Required for the Abscisic Acid Induction of Gene Expression in Barley Aleurone Cells

2002 ◽  
Vol 15 (1) ◽  
pp. 271-284 ◽  
Author(s):  
Jose Casaretto ◽  
Tuan-hua David Ho
Keyword(s):  
Gene Expression ◽  
Transcription Factors ◽  
Abscisic Acid ◽  
Aleurone Cells ◽  
Barley Aleurone

Gene Expression in Response to Abscisic Acid and Osmotic Stress

1990 ◽  
Vol 2 (6) ◽  
pp. 503 ◽  
Author(s):  
Karen Skriver ◽  
John Mundy
Keyword(s):  
Gene Expression ◽  
Abscisic Acid ◽  
Osmotic Stress

scholarly journals Interaction of Osmotic Stress, Temperature, and Abscisic Acid in the Regulation of Gene Expression in Arabidopsis

1999 ◽  
Vol 119 (1) ◽  
pp. 205-212 ◽  
Author(s):  
Liming Xiong ◽  
Manabu Ishitani ◽  
Jian-Kang Zhu
Keyword(s):  
Gene Expression ◽  
Abscisic Acid ◽  
Osmotic Stress ◽  
Regulation Of Gene Expression

scholarly journals Gene expression in response to abscisic acid and osmotic stress.

1990 ◽  
Vol 2 (6) ◽  
pp. 503-512 ◽  
Author(s):  
K Skriver ◽  
J Mundy
Keyword(s):  
Gene Expression ◽  
Abscisic Acid ◽  
Osmotic Stress

Global Genome Expression Analysis of Transcription Factors under PEG Osmotic Stress in Rice Root System

2009 ◽  
Vol 35 (6) ◽  
pp. 1030-1037 ◽  
Author(s):  
Ting-Chen MA ◽  
Rong-Jun CHEN ◽  
Rong-Rong YU ◽  
Han-Lai ZENG ◽  
Duan-Pin ZHANG
Keyword(s):  
Transcription Factors ◽  
Osmotic Stress ◽  
Root System ◽  
Expression Analysis ◽  
Rice Root ◽  
Genome Expression

scholarly journals Transcriptome Analysis of Hypertrophic Heart Tissues from Murine Transverse Aortic Constriction and Human Aortic Stenosis Reveals Key Genes and Transcription Factors Involved in Cardiac Remodeling Induced by Mechanical Stress

2019 ◽  
Vol 2019 ◽  
pp. 1-10
Author(s):  
Peng Yu ◽  
Baoli Zhang ◽  
Ming Liu ◽  
Ying Yu ◽  
Ji Zhao ◽  
...  
Keyword(s):  
Gene Expression ◽  
Transcription Factors ◽  
Aortic Stenosis ◽  
Mechanical Stress ◽  
Cardiac Remodeling ◽  
Interaction Network ◽  
Cytokine Signaling ◽  
Transverse Aortic Constriction ◽  
Aortic Constriction ◽  
Genomic Changes

Background. Mechanical stress-induced cardiac remodeling that results in heart failure is characterized by transcriptional reprogramming of gene expression. However, a systematic study of genomic changes involved in this process has not been performed to date. To investigate the genomic changes and underlying mechanism of cardiac remodeling, we collected and analyzed DNA microarray data for murine transverse aortic constriction (TAC) and human aortic stenosis (AS) from the Gene Expression Omnibus database and the European Bioinformatics Institute. Methods and Results. The differential expression genes (DEGs) across the datasets were merged. The Venn diagrams showed that the number of intersections for early and late cardiac remodeling was 74 and 16, respectively. Gene ontology and protein–protein interaction network analysis showed that metabolic changes, cell differentiation and growth, cell cycling, and collagen fibril organization accounted for a great portion of the DEGs in the TAC model, while in AS patients’ immune system signaling and cytokine signaling displayed the most significant changes. The intersections between the TAC model and AS patients were few. Nevertheless, the DEGs of the two species shared some common regulatory transcription factors (TFs), including SP1, CEBPB, PPARG, and NFKB1, when the heart was challenged by applied mechanical stress. Conclusions. This study unravels the complex transcriptome profiles of the heart tissues and highlighting the candidate genes involved in cardiac remodeling induced by mechanical stress may usher in a new era of precision diagnostics and treatment in patients with cardiac remodeling.

scholarly journals Arabidopsis heat shock transcription factor HSFA7b positively mediates salt stress tolerance by binding to an E-box-like motif to regulate gene expression

2019 ◽  
Vol 70 (19) ◽  
pp. 5355-5374 ◽  
Author(s):  
Dandan Zang ◽  
Jingxin Wang ◽  
Xin Zhang ◽  
Zhujun Liu ◽  
Yucheng Wang
Keyword(s):  
Gene Expression ◽  
Transcription Factors ◽  
Heat Shock ◽  
Salt Tolerance ◽  
Stress Responses ◽  
Ion Homeostasis ◽  
Cellulose Synthase ◽  
Regulate Gene Expression ◽  
E Box ◽  
Regulate Gene

Abstract Plant heat shock transcription factors (HSFs) are involved in heat and other abiotic stress responses. However, their functions in salt tolerance are little known. In this study, we characterized the function of a HSF from Arabidopsis, AtHSFA7b, in salt tolerance. AtHSFA7b is a nuclear protein with transactivation activity. ChIP-seq combined with an RNA-seq assay indicated that AtHSFA7b preferentially binds to a novel cis-acting element, termed the E-box-like motif, to regulate gene expression; it also binds to the heat shock element motif. Under salt conditions, AtHSFA7b regulates its target genes to mediate serial physiological changes, including maintaining cellular ion homeostasis, reducing water loss rate, decreasing reactive oxygen species accumulation, and adjusting osmotic potential, which ultimately leads to improved salt tolerance. Additionally, most cellulose synthase-like (CSL) and cellulose synthase (CESA) family genes were inhibited by AtHSFA7b; some of them were randomly selected for salt tolerance characterization, and they were mainly found to negatively modulate salt tolerance. By contrast, some transcription factors (TFs) were induced by AtHSFA7b; among them, we randomly identified six TFs that positively regulate salt tolerance. Thus, AtHSFA7b serves as a transactivator that positively mediates salinity tolerance mainly through binding to the E-box-like motif to regulate gene expression.

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