Rapamycin-Induced Impaired Wound Healing Is Associated with Compromised Tissue Lactate Accumulation and Extracellular Matrix Remodeling

2011 ◽  
Vol 47 (1) ◽  
pp. 39-44 ◽  
Author(s):  
J. Weinreich ◽  
S. Löb ◽  
M. Löffler ◽  
I. Königsrainer ◽  
D. Zieker ◽  
...  
2019 ◽  
Vol 47 (6) ◽  
pp. 1679-1687
Author(s):  
Mavis A.A. Tenkorang ◽  
Upendra Chalise ◽  
Michael J. Daseke, II ◽  
Shelby R. Konfrst ◽  
Merry L. Lindsey

Myocardial Infarction (MI) initiates a series of wound healing events that begins with up-regulation of an inflammatory response and culminates in scar formation. The extracellular matrix (ECM) is intricately involved in all stages from initial break down of existing ECM to synthesis of new ECM to form the scar. This review will summarize our current knowledge on the processes involved in ECM remodeling after MI and identify the gaps that still need to be filled.


2015 ◽  
Vol 31 (7) ◽  
pp. 1186-1195 ◽  
Author(s):  
Elizabeth H. Stephens ◽  
Tom C. Nguyen ◽  
Jack G. Blazejewski ◽  
Dragoslava P. Vekilov ◽  
Jennifer P. Connell ◽  
...  

2015 ◽  
Vol 79 (1) ◽  
pp. 50-56 ◽  
Author(s):  
Bruno Cogliati ◽  
Mathieu Vinken ◽  
Tereza C. Silva ◽  
Cintia M.M. Araújo ◽  
Thiago P.A. Aloia ◽  
...  

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