scholarly journals Physiological Response of the Physical Capabilities of Adolescents with Sensorineural Hearing Loss to Regular Adaptive Handball

2021 ◽  
Vol 14 (1) ◽  
pp. 99-103
Author(s):  
Vladimir Yu. Karpov ◽  
Svetlana Yu. Zavalishina ◽  
Alexander V. Dorontsev ◽  
Anastasia A. Svetlichkina ◽  
Roman V. Kozjakov ◽  
...  

Training within the framework of adaptive sports and especially handball helps to significantly increase the level of adaptation to social conditions in people with hearing system pathology. Regular practice of this sport increases the accuracy of motor acts and stimulates general adaptive capabilities. The study involved 31 male teenagers aged 13-14 with sensorineural hearing loss of I-II degree. Of these, two groups were formed: the observation group (14 adolescents), who began to engage in adaptive handball, and the comparison group (17 adolescents), who continued to engage in physical education at school. The results of the annual medical examination, the results of functional tests and control standards of physical fitness were used. The results were processed using the Student's t test and correlation analysis.Regular physical trainings within the framework of handball allowed to increase the coordination-motor potential and stabilize the vestibular-dynamic stability of adolescents. They increased their physical readiness and the accuracy of their motor actions fulfillment in the conditions of play and in everyday life.Regular training in adaptive handball in adolescents with sensorineural hearing loss increases coordination abilities, speed capabilities, strength qualities and general endurance.

2020 ◽  
Vol 24 (01) ◽  
pp. e5-e10 ◽  
Author(s):  
Francesco Ciodaro ◽  
Francesco Freni ◽  
Giuseppe Alberti ◽  
Marco Forelli ◽  
Francesco Gazia ◽  
...  

Abstract Introduction The cochlea and the vestibular receptors are closely related in terms of anatomy and phylogeny. Patients with moderate to profound sensorineural hearing loss (MPSHL) should have their vestibular organ functions tested. Objective To evaluate the incidence of vestibular abnormalities in patients with MPSHL and to study the correlation between the etiology of hearing loss (HL) and a possible damage to the labyrinth. Methods A case-control retrospective study was performed. In the case group, 20 adults with MPSHL of known etiology were included. The control group was composed of 15 adults with normal hearing. The case group was divided into 4 subgroups based on the etiology (bacterial meningitis, virus, vascular disease, congenital). Cervical vestibular-evoked myogenic potentials (cVEMPs) were used to rate the saccular function and lower vestibular nerve. Results The study was performed in 70 ears, and it highlighted the presence of early biphasic P1-N1 complex in 29 (71.5%) out of 40 ears in the study group, and in all of the 30 ears in the control group (p = 0.001). Regarding the presence or absence of cVEMPs among the four subgroups of patients with MPSHL, the data were statistically significant (p < 0.001). The comparison between the latencies and amplitude of P1-N1 in case and control groups from other studies and in the four subgroups of cases in the present study did not detect statistically significant differences. Conclusion The present study demonstrates that patients with MPSHL have a high incidence of damage to the labyrinthine organs, and it increases the current knowledge about the etiopathogenesis of sensorineural HL, which is often of unknown nature.


2020 ◽  
Vol 12 (4) ◽  
pp. 114-117
Author(s):  
Alireza Bina

There are some studies which confirmed that dysfunction in Central Nervous System(CNS) may cause a malfunction in the Peripheral Auditory system (Cochlea_ Auditory Nerve, Auditory Neuropathy), but the question is could Brain Disorder without any lesion in the Cochlea and/or Auditory nerve cause Sensorineural Hearing Loss? It means that the Audiogram shows that the patient suffers from sensorineural hearing loss but the site of the lesion is neither Sensory nor Neural while Brain may be involved in charge of this. And if the answer is yes then could we hear with our Brain and without Cochlea and /or Auditory nerve? We deal with this subject in this paper by: Otosclerosis and Meniere’s disease and The Brain Involvement. Hearing Loss following dysfunction in the Central Auditory and/or central non auditory system. Auditory Brainstem Implant in Patients who suffer from Neurofibromatosis Type two compare to Non Tumor cases, Mondini Syndrome, Michel aplasia. Possible role of Utricle and Saccule in Auditory (Hearing) System We propose a new Hypothesis that the External Ear Canal is not the only input of Auditory Signals, Sounds could transfer by our eyes and skin to the Cerebral Cortex and approach to the Cochlea (Backward Auditory input pathway of Sounds).


Author(s):  
Jeevan Jyoti ◽  
Ihsan Ali ◽  
Syed Waseem Abbas

<p class="Normal1"><strong>Background: </strong>The association of sensorineural hearing loss in diabetes mellitus patients is known since decades, yet there is no clear consensus among previous studies, with respect to the prevalence of SNHL in type 2 diabetes patients and the effect of duration and control of diabetes on hearing acuity. Hence the objectives of this study are to find the prevalence of SNHL in type 2 diabetes patients and to find the effect of duration and control of diabetes on hearing loss.</p><p class="Normal1"><strong>Methods: </strong>The present study was conducted on 100 type 2 diabetes patients and age and gender matched 100 non- diabetic controls in the age group of &lt;50 years, selected based on inclusion and exclusion criteria. After detailed history taking and clinical examination, all subjects underwent FBS, PPBS estimation and HbA1c evaluation was done for diabetic patients. All underwent pure tone audiometry, DPOAE and BERA and the findings were recorded and analyzed.</p><p class="Normal1"><strong>Results: </strong>Diabetes patients had insidious onset, gradually progressive, bilaterally symmetrical SNHL. SNHL is prevalent in 73% of type 2 diabetes patients compared to 16% of controls. It is aggravated with the increasing age and duration of diabetes. Poor control of diabetes showed increased prevalence of SNHL compared to good control of diabetes.</p><p class="Normal1"><strong>Conclusions: </strong>There is increased prevalence of SNHL in type 2 diabetes patients and it is more evident in patients with long duration of diabetes and more pronounced in patients with poor diabetic control.</p>


2021 ◽  
Author(s):  
Boeun Lee ◽  
Yun Jung Bae ◽  
Byung Yoon Choi ◽  
Hyojin Kim ◽  
Byung Se Choi ◽  
...  

Abstract Purpose Autoimmune and inner ear diseases (AIED/AID) are characterized by the symptom of sensorineural hearing loss (SNHL). To date, standardized diagnostic tools for AIED/AID are lacking, and clinically differentiating AIED/AID from chronic otomastoiditis (COM) with SNHL is challenging. This retrospective study aimed to construct an MRI-based decision tree using classification and regression tree (CART) analysis to distinguish AIED/AID from COM.Methods In total, 67 patients were enrolled between January 2004 and October 2019, comprising AIED/AID (n = 18), COM (n = 24), and control groups (n = 25). All patients underwent 3T temporal bone MRI, including post-contrast T1-weighted images (postT1WI) and post-contrast FLAIR images (postFLAIR). Two radiologists evaluated the presence of otomastoid effusion and inner ear contrast enhancement on MRI. A CART decision tree model was constructed using MRI features to differentiate AIED/AID from COM and control groups, and diagnostic performance was analyzed.Results High-intensity bilateral effusion (61.1%) and inner ear enhancement (postFLAIR, 93.8%; postT1WI, 61.1%) were the most common in the AIED/AID group. We constructed two CART decision tree models; the first used effusion amount as the first partitioning node and postT1WI-inner ear enhancement as the second node, whereas the second comprised two partitioning nodes with the degree of postFLAIR-enhancement of the inner ear. The first and second models enabled distinction of AIED/AID from COM with high specificity (100% and 94.3%, respectively).Conclusion The amount of effusion and degree of inner ear enhancement on MRI may facilitate the distinction between AIED/AID and COM with SNHL using decision tree models, thereby contributing to early diagnosis and intervention.


Author(s):  
Jyothi Anand Chavadaki ◽  
Mohammed Nadeem Afroze Malli

<p class="abstract"><strong>Background:</strong> The association of sensorineural hearing loss (SNHL) in diabetes mellitus patients is known since decades, yet there is no clear consensus among previous studies, with respect to the prevalence of SNHL in type 2 diabetes patients and the effect of duration and control of diabetes on hearing acuity. Hence the objectives of this study are to find the prevalence of SNHL in type 2 diabetes patients and to find the effect of duration and control of diabetes on hearing loss.</p><p class="abstract"><strong>Methods:</strong> The present study was conducted on 86 type 2 diabetes patients attending Navodaya Medical College Hospital, Raichur and age and gender matched 86 non- diabetic controls in the age group of 30-65 years, selected based on inclusion and exclusion criteria. After detailed history taking and clinical examination, all subjects underwent FBS, PPBS estimation and HbA1c evaluation was done for diabetic patients. All underwent pure tone audiometry and the findings were recorded and analyzed.  </p><p class="abstract"><strong>Results:</strong> Diabetes patients had insidious onset, gradually progressive, bilaterally symmetrical SNHL. SNHL is prevalent in 67.44% of type 2 diabetes patients compared to 23.26% of controls. It is aggravated with the increasing age and duration of diabetes. Poor control of diabetes showed increased prevalence of SNHL compared to good control of diabetes.</p><p align="left"><strong>Conclusions:</strong> There is increased prevalence of SNHL in type 2 diabetes patients and it is more evident in patients with long duration of diabetes and more pronounced in patients with poor diabetic control.</p>


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