scholarly journals Transient renal Fanconi syndrome in a Chihuahua exposed to Chinese chicken jerky treats

2015 ◽  
Vol 43 (03) ◽  
pp. 188-192 ◽  
Author(s):  
E. Furman ◽  
J. Leidinger ◽  
D. Brandstetter ◽  
C. Hochleithner ◽  
A. C. Sewell ◽  
...  
Keyword(s):  
North America ◽  
Fanconi Syndrome ◽  
Clinical Signs ◽  
Metabolic Abnormalities ◽  
Renal Fanconi Syndrome ◽  
First Report ◽  
History Of ◽  
Renal Tubular

SummaryTransient Fanconi syndrome without azotemia was diagnosed in a dog and was associated with ingestion of Chinese chicken jerky treats. Fanconi syndrome is a proximal renal tubular defect and a diagnosis was made based upon severe glucosuria with normoglycemia, and severe generalized aminoaciduria. The clinical signs of polyuria and polydipsia as well as the massive urinary metabolic abnormalities resolved after jerky treat withdrawal. While frequently seen in North America and Australia, this is the first report of jerky treat induced Fanconi syndrome in continental Europe. Clinicians should be aware of this potential intoxication and be vigilant for a history of jerky treat consumption in a dog with glucosuria.

Balloon dilation of a urethral stricture caused by a congenital obstructive proximal urethral membrane (COPUM) in a dog

2020 ◽  
Vol 8 (2) ◽  
pp. e001084
Author(s):  
Darren Kelly ◽  
Ingrid Isaac ◽  
Judith Cruzado-Perez ◽  
Florence Juvet
Keyword(s):  
Urethral Stricture ◽  
Balloon Dilation ◽  
Clinical Signs ◽  
Human Beings ◽  
Membranous Structure ◽  
First Report ◽  
Urethral Strictures ◽  
The Future ◽  
History Of ◽  
Proximal Urethra

Congenital urethral strictures are well recognised in human beings and have recently been described in two cats but have not been previously reported in dogs. A 10-month-old female English Bull Terrier presented with a life-long history of being unable to pass a normal stream of urine. Urethrocystoscopy confirmed the presence of a stricture lesion in the proximal urethra. This thin, membranous structure was effaced under endoscopic visualisation using a 10 mm diameter balloon-dilation catheter. Complete and sustained resolution of clinical signs occurred after a single dilation procedure. To the best of our knowledge, this is the first report of a congenital urethral stricture in a dog and the term congenital obstructive proximal urethral membrane may be useful for describing these lesions in the future.

A Skipper, Thymelicus lineola (Ochs.) (Lepidoptera: Hesperiidae) and its Parasites in Ontario

1962 ◽  
Vol 94 (10) ◽  
pp. 1082-1089 ◽  
Author(s):  
A. P. Arthur
Keyword(s):  
North America ◽  
Present Author ◽  
The West ◽  
West Side ◽  
Southern Ontario ◽  
First Report ◽  
History Of ◽  
Lake Simcoe ◽  
First Time ◽  
Extensive Damage

The European, or Essex skipper, Thymelicus (= Adopaea) lineola (Ochs.), was accidentally introduced into North America at London, Ontario, sometime before 1910 (Saunders, 1916). The history of its subsequent spread through southern Ontario and adjoining parts of Michigan and Ohio was reviewed by Pengelly (1961), who received the first report of extensive damage to hay and pasture crops by this insect in Ontario from the Markdale area of Grey County in 1956. A survey in 1958 (Pengelly, 1961) showed that the skipper “appeared to be present throughout the southern part of the province except for the Bruce peninsula and possibly the Windsor area. The northeasterly boundary appeared to he along a line from Midland, south around the west side of Lake Simcoe, east to Lindsay and south to Whitby.” The present author collected T. lineola larvae from the Belleville area for the first time in 1959.

Rectal ganglioneuroma in a dog

1999 ◽  
Vol 35 (2) ◽  
pp. 107-110 ◽  
Author(s):  
ME Reimer ◽  
MS Leib ◽  
MS Reimer ◽  
GK Saunders ◽  
SA Johnston
Keyword(s):  
Gastrointestinal Tract ◽  
Surgical Resection ◽  
Large Bowel ◽  
Clinical Signs ◽  
Successful Outcome ◽  
First Report ◽  
History Of ◽  
The Masses

An 18-month-old, spayed female Australian terrier cross was presented with a 10-month history of chronic large bowel diarrhea. Ulceration and two proliferative masses in the rectum were seen on colonoscopy. Surgical resection was performed to remove the masses, and the dog recovered without complications related to surgery. Histopathology was consistent with the diagnosis of ganglioneuroma. The dog had no clinical signs of disease within three months of surgery and was completely normal 2.5 years after diagnosis. This is the first report providing follow-up and successful outcome of a ganglioneuroma in the gastrointestinal tract of a dog.

scholarly journals Tracheal stenosis in a Holstein cow

2020 ◽  
Vol 50 (8) ◽  
Author(s):  
Franciéli Adriane Molossi ◽  
Regina Tose Kemper ◽  
Bianca Santana de Cecco ◽  
Rafael Biondo Rosa ◽  
Luciana Sonne ◽  
...  
Keyword(s):  
Tracheal Stenosis ◽  
Plasma Cells ◽  
Clinical Signs ◽  
Tracheal Mucosa ◽  
First Report ◽  
Holstein Cow ◽  
Adjacent Mucosa ◽  
Squamous Cell Metaplasia ◽  
History Of ◽  
Well Differentiated

ABSTRACT: Tracheal stenosis, also known as “Honker syndrome”, is characterized by tracheal edema and hemorrhage, leading to partial obstruction of the lumen; therefore, death. Its etiology is not yet well elucidated. A 3-year-old Holstein cow, with a history of dyspnea, and a large amount of reddish foam flowing from the mouth and nose, had died after 10 minutes of clinical signs. Macroscopic examination revealed focally extensive hemorrhage and clot organization in tracheal mucosa extending to the submucosa, surrounded by well-differentiated fibrous connective tissue. In adjacent mucosa was observed moderate multifocal inflammatory infiltrate composed by lymphocytes and plasma cells, as well as moderate squamous cell metaplasia. The bacterial culture showed growth of contaminant and environmental bacteria and the RT-PCR to detect Herpesvirus 1 (BoHV-1) and 5 (BoHV-5) was negative. To the author’s knowledge, this is the first report of tracheal stenosis in South America, as well as the first report of this condition described in a Holstein cow.

A new fossil wood of Koelreuteria (Sapindaceae) from the Pliocene of China and remarks on the phytogeographic history of Koelreuteria

IAWA Journal ◽  
2012 ◽  
Vol 33 (3) ◽  
pp. 301-307
Author(s):  
Ye-Ming Cheng ◽  
Ya-Fang Yin ◽  
R.C. Mehrotra ◽  
Cheng-Sen Li
Keyword(s):  
North America ◽  
East Asia ◽  
Fossil Wood ◽  
First Report ◽  
History Of

Koelreuteria yuanmouensis sp. nov. (Sapindaceae) is described from the Pliocene fluvio-lacustrine rocks of Hutiaotan Earth Forest, Yuanmou Basin, Yunnan, China. This is the first report of fossil Koelreuteria wood from Asia. The history of the genus is reviewed. Fruits and leaves of the genus have been reported from the Paleocene onwards in Asia, North America, and Europe, with the genus becoming restricted to East Asia during the Neogene.

scholarly journals Clinical Heterogeneity and Phenotypic Expansion of NaPi-IIa–Associated Disease

2017 ◽  
Vol 102 (12) ◽  
pp. 4604-4614 ◽  
Author(s):  
Korcan Demir ◽  
Melek Yıldız ◽  
Hilla Bahat ◽  
Michael Goldman ◽  
Nisreen Hassan ◽  
...  
Keyword(s):  
Fanconi Syndrome ◽  
Loss Of Function ◽  
Renal Fanconi Syndrome ◽  
Whole Exome ◽  
Proximal Tubular ◽  
History Of ◽  
Proximal Tubulopathy ◽  
Idiopathic Infantile Hypercalcemia ◽  
Close Relatives

Abstract Context NaPi-IIa, encoded by SLC34A1, is a key phosphate transporter in the mammalian proximal tubule and plays a cardinal role in renal phosphate handling. NaPi-IIa impairment has been linked to various overlapping clinical syndromes, including hypophosphatemic nephrolithiasis with osteoporosis, renal Fanconi syndrome with chronic kidney disease, and, most recently, idiopathic infantile hypercalcemia and nephrocalcinosis. Objectives We studied the molecular basis of idiopathic infantile hypercalcemia with partial proximal tubulopathy in two apparently unrelated patients of Israeli and Turkish descent. Design Genetic analysis in two affected children and their close relatives was performed using whole-exome sequencing, followed by in vitro localization and trafficking analysis of mutant NaPi-IIa. Results Mutation and haplotype analyses in both patients revealed a previously described homozygous loss-of-function inserted duplication (p.I154_V160dup) in NaPi-IIa, which is inherited identical-by-descent from a common ancestor. The shared mutation was originally reported by our team in two adult siblings with renal Fanconi syndrome, hypophosphatemic bone disease, and progressive renal failure who are family members of one of the infants reported herein. In vitro localization assays and biochemical analysis of p.I154_V160dup and of additional NaPi-IIa mutants harboring a trafficking defect indicate aberrant retention at the endoplasmic reticulum in an immature and underglycosylated state, leading to premature proteasomal degradation. Conclusions Our findings expand the phenotypic spectrum of NaPi-IIa disruption, reinforce its link with proximal tubular impairment, enable longitudinal study of the natural history of the disease, and shed light on cellular pathways associated with loss of function and impaired trafficking of NaPi-IIa mutants.

scholarly journals Primary adenocarcinoma of the gland of the nictitating membrane in a cat

1997 ◽  
Vol 33 (4) ◽  
pp. 333-336 ◽  
Author(s):  
AM Komaromy ◽  
DT Ramsey ◽  
JA Render ◽  
P Clark
Keyword(s):  
Clinical Signs ◽  
Biological Behavior ◽  
Primary Adenocarcinoma ◽  
Nictitating Membrane ◽  
Posterior Aspect ◽  
First Report ◽  
Natural Progression ◽  
History Of ◽  
The Right

An 11-year-old, neutered, male domestic shorthair was presented with a five-month history of recurrent, unilateral, seromucoid discharge from the right eye. A verrucous mass extended from the posterior aspect of the nictitating membrane. Adenocarcinoma of the gland of the nictitating membrane (GNM) was diagnosed upon biopsy. The cat subsequently developed metastases to the lungs, pleura, mediastinum, liver, and kidneys and died six months after clinical signs first were observed. Little is known about the biological behavior of adenocarcinoma of the GNM in cats. This is the first report that describes the natural progression of this disease.

Megaesophagus in a 6-month-old cat secondary to a nasopharyngeal polyp

2010 ◽  
Vol 12 (4) ◽  
pp. 322-324 ◽  
Author(s):  
Julie K. Byron ◽  
Steven R. Shadwick ◽  
Avery R. Bennett
Keyword(s):  
Clinical Signs ◽  
Cervical Esophagus ◽  
First Report ◽  
History Of ◽  
Upper Respiratory

A 6-month-old male domestic shorthair cat was presented for a 3-month history of dysphagia and upper respiratory signs. The cat was diagnosed with a generalized megaesophagus secondary to a large nasopharyngeal polyp that extended into the cervical esophagus. The polyp was removed by traction and a left ventral bulla osteotomy was performed to remove the polyp base. The cat's clinical signs resolved and follow-up radiographs 14 days after surgery revealed resolution of the megaesophagus. To the authors' knowledge, this is the first report of resolution of megaesophagus after removal of a nasopharyngeal polyp in a cat.

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