Use of Clinical Improvisation to Improve Attention and Memory Deficits in Stroke Patients

2021 ◽  
Author(s):  
Molly Elizabeth Brown
Author(s):  
Alejandro A. Arca ◽  
Kaitlin M. Stanford ◽  
Mustapha Mouloua

The current study was designed to empirically examine the effects of individual differences in attention and memory deficits on driver distraction. Forty-eight participants consisting of 37 non-ADHD and 11 ADHD drivers were tested in a medium fidelity GE-ISIM driving simulator. All participants took part in a series of simulated driving scenarios involving both high and low traffic conditions in conjunction with completing a 20-Questions task either by text- message or phone-call. Measures of UFOV, simulated driving, heart rate variability, and subjective (NASA TLX) workload performance were recorded for each of the experimental tasks. It was hypothesized that ADHD diagnosis, type of cellular distraction, and traffic density would affect driving performance as measured by driving performance, workload assessment, and physiological measures. Preliminary results indicated that ADHD diagnosis, type of cellular distraction, and traffic density affected the performance of the secondary task. These results provide further evidence for the deleterious effects of cellphone use on driver distraction, especially for drivers who are diagnosed with attention-deficit and memory capacity deficits. Theoretical and practical implications are discussed, and directions for future research are also presented.


Author(s):  
C. A. Mateer ◽  
M. M. Sohlberg ◽  
P. K. Youngman

2017 ◽  
Vol 45 (10) ◽  
pp. 1742-1750 ◽  
Author(s):  
Raisa M. Schiller ◽  
Hanneke IJsselstijn ◽  
Marlous J. Madderom ◽  
André B. Rietman ◽  
Marion Smits ◽  
...  

2014 ◽  
Vol 46 (5) ◽  
pp. 274-284 ◽  
Author(s):  
Jennifer Sandson Frank ◽  
David E. Vance ◽  
Angela Jukkala ◽  
Karen M. Meneses

2017 ◽  
Vol 81 ◽  
pp. 73-78 ◽  
Author(s):  
Priscila P. Almeida ◽  
Gerardo M. de Araujo Filho ◽  
Stella M. Malta ◽  
Ronaldo R. Laranjeira ◽  
Ana Cecilia R.P. Marques ◽  
...  

Author(s):  
Selma Lugtmeijer ◽  
Nikki A. Lammers ◽  
Edward H. F. de Haan ◽  
Frank-Erik de Leeuw ◽  
Roy P. C. Kessels

AbstractThis review investigates the severity and nature of post-stroke working memory deficits with reference to the multi-component model of working memory. We conducted a systematic search in PubMed up to March 2019 with search terms for stroke and memory. Studies on adult stroke patients, that included a control group, and assessed working memory function, were selected. Effect sizes (Hedges’ g) were extracted from 50 studies (in total 3,084 stroke patients) based on the sample size, mean and standard deviation of patients and controls. Performance of stroke patients was compared to healthy controls on low-load (i.e. capacity) and high-load (executively demanding) working memory tasks, grouped by modality (verbal, non-verbal). A separate analysis compared patients in the sub-acute and the chronic stage. Longitudinal studies and effects of lesion location were systematically reviewed. Stroke patients demonstrated significant deficits in working memory with a moderate effect size for both low-load (Hedges’ g = -.58 [-.82 to -.43]) and high-load (Hedges’ g = -.59 [-.73 to -.45]) tasks. The effect sizes were comparable for verbal and non-verbal material. Systematically reviewing the literature showed that working memory deficits remain prominent in the chronic stage of stroke. Lesions in a widespread fronto-parietal network are associated with working memory deficits. Stroke patients show decrements of moderate magnitude in all subsystems of working memory. This review clearly demonstrates the global nature of the impairment in working memory post-stroke.


2001 ◽  
Vol 13 (3) ◽  
pp. 76-78
Author(s):  
N. van de Weg ◽  
F.R.J. Verhey ◽  
P J.M. Raedts ◽  
F W. Vreeling

SUMMARYWe describe the clinical history of a 49-year old woman, who demonstrated progressive personality changes more than twenty years after radiation of a pituitary adenoma (prolactinoma), with apathy, loss of initiative, memory deficits, postural instability, dysarthria and faecal incontinence. Neuropsychological assessment showed impulsivity, loss of overview, desinhibition, fluctuating deficits of attention, and memory disturbances. MRI-scanning of the brain revealed a cystic lesion along the right ventricle. The clinical picture and the findings of the other investigations are typical for dementia due to radiation encephalopathy. Such a long period between radiation and cognitive deterioration is rare, although it has been described before.


2010 ◽  
Vol 24 (2) ◽  
pp. 246-264 ◽  
Author(s):  
Nicholas S. Thaler ◽  
Daniel N. Allen ◽  
Janice C. McMurray ◽  
Joan Mayfield

2020 ◽  
Vol 4 (4) ◽  
pp. 88-91
Author(s):  
Bakou Niangoran Francois ◽  
Atayi Eugene ◽  
BA Abdoulaye

BACKGROUND: The search for disruption of memory in patients with stroke has been performed, in order to better understand the behavior of these patients and optimize their neuropsychological accompaniment. AIM: To evaluate memory disturbances following stroke in patients; in order to better understand the attitudes of these patients.MATERIALS AND METHOD: Fifty-five (55) patients with stroke participated in a spatial memory test. This test consists in studying the arrangement, the designation and the evocation of images during learning a spatial arrangement. RESULTS: Of the 55 patients, 54.54% of patients passed the test but with later learning. And 45.45% failed at different events, they present real memory problems.CONCLUSION: Our study, confirms that stroke patients face difficulties which often include cognitive impairments, such as memory deficits. Our results indicate that these memory disorders manifest themselves to varying degrees. Indeed, the working memory disorders would be more marked in group II patients than in group I patients or they can be considered transient


2013 ◽  
Vol 368 (1628) ◽  
pp. 20130062 ◽  
Author(s):  
Kathrin Finke ◽  
Nicholas Myers ◽  
Peter Bublak ◽  
Christian Sorg

The common view of Alzheimer's disease (AD) is that of an age-related memory disorder, i.e. declarative memory deficits are the first signs of the disease and associated with progressive brain changes in the medial temporal lobes and the default mode network. However, two findings challenge this view. First, new model-based tools of attention research have revealed that impaired selective attention accompanies memory deficits from early pre-dementia AD stages on. Second, very early distributed lesions of lateral parietal networks may cause these attention deficits by disrupting brain mechanisms underlying attentional biased competition. We suggest that memory and attention impairments might indicate disturbances of a common underlying neurocognitive mechanism. We propose a unifying account of impaired neural interactions within and across brain networks involved in attention and memory inspired by the biased competition principle. We specify this account at two levels of analysis: at the computational level, the selective competition of representations during both perception and memory is biased by AD-induced lesions; at the large-scale brain level, integration within and across intrinsic brain networks, which overlap in parietal and temporal lobes, is disrupted. This account integrates a large amount of previously unrelated findings of changed behaviour and brain networks and favours a brain mechanism-centred view on AD.


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