Gingival Epithelial Cell Expression of Macrophage Inflammatory Protein-1α Induced by Interleukin-1β and Lipopolysaccharide

2007 ◽  
Vol 78 (8) ◽  
pp. 1627-1634 ◽  
Author(s):  
Ok H. Ryu ◽  
Sun J. Choi ◽  
Ana Maria G. Linares ◽  
In S. Song ◽  
Young J. Kim ◽  
...  
Keyword(s):  
Epithelial Cell ◽  
Interleukin 1Β ◽  
Gingival Epithelial Cell ◽  
Inflammatory Protein ◽  
Cell Expression ◽  
Macrophage Inflammatory Protein

Fever induced by macrophage inflammatory protein-1β in the rat is independent of hypothalamic interleukin-1β or interleukin-6

2000 ◽  
Vol 25 (1-2) ◽  
pp. 5-10 ◽  
Author(s):  
Khalid Benamar ◽  
Antonio Fernández-Alonso ◽  
Eva Tavares ◽  
Francisco J López-Valpuesta ◽  
Manuel Sancibrián ◽  
...  
Keyword(s):  
Interleukin 6 ◽  
Interleukin 1Β ◽  
Inflammatory Protein ◽  
Macrophage Inflammatory Protein

Associations of interleukin-6 with interleukin-1β, interleukin-8 and macrophage inflammatory protein-1β in midlife women

Cytokine ◽  
2008 ◽  
Vol 41 (3) ◽  
pp. 302-306 ◽  
Author(s):  
Toshiyuki Yasui ◽  
Hirokazu Uemura ◽  
Masayo Yamada ◽  
Toshiya Matsuzaki ◽  
Naoko Tsuchiya ◽  
...  
Keyword(s):  
Interleukin 6 ◽  
Midlife Women ◽  
Interleukin 8 ◽  
Interleukin 1Β ◽  
Inflammatory Protein ◽  
Macrophage Inflammatory Protein

Interleukin-1β stimulates macrophage inflammatory protein-1α and -1β expression in human neuronal cells (NT2-N)

2003 ◽  
Vol 84 (5) ◽  
pp. 997-1005 ◽  
Author(s):  
Chang-Jiang Guo ◽  
Steven D. Douglas ◽  
Jian-Ping Lai ◽  
David E. Pleasure ◽  
Yuan Li ◽  
...  
Keyword(s):  
Neuronal Cells ◽  
Interleukin 1Β ◽  
Inflammatory Protein ◽  
Human Neuronal Cells ◽  
Macrophage Inflammatory Protein

Role of the type 1 TNF receptor in lung inflammation after inhalation of endotoxin orPseudomonas aeruginosa

1999 ◽  
Vol 276 (5) ◽  
pp. L715-L727 ◽  
Author(s):  
Shawn J. Skerrett ◽  
Thomas R. Martin ◽  
Emil Y. Chi ◽  
Jacques J. Peschon ◽  
Kendall M. Mohler ◽  
...  
Keyword(s):  
Lung Inflammation ◽  
Interleukin 1Β ◽  
Intercellular Adhesion Molecule ◽  
Bacterial Clearance ◽  
Tnf Receptor ◽  
Protein Levels ◽  
Inflammatory Protein ◽  
Tnf Α ◽  
Macrophage Inflammatory Protein

To determine the roles of the type 1 tumor necrosis factor (TNF) receptor (TNFR1) in lung inflammation and antibacterial defense, we exposed transgenic mice lacking TNFR1 [TNFR1(−/−)] and wild-type control mice to aerosolized lipopolysaccharide or Pseudomonas aeruginosa. After LPS, bronchoalveolar lavage fluid (BALF) from TNFR1(−/−) mice contained fewer neutrophils and less macrophage inflammatory protein-2 than BALF from control mice. TNF-α, interleukin-1β, and total protein levels in BALF as well as tissue intercellular adhesion molecule-1 expression did not differ between the two groups. In contrast, lung inflammation and bacterial clearance after infection were augmented in TNFR1(−/−) mice. BALF from infected TNFR1(−/−) mice contained more neutrophils and TNF-α and less interleukin-1β and macrophage inflammatory protein-2 than that from control mice, but protein levels were similarly elevated in both groups. Lung inflammation and bacterial clearance were also augmented in mice lacking both TNF receptors. Thus TNFR1 facilitates neutrophil recruitment after inhalation of lipopolysaccharide, in part by augmenting chemokine induction. In contrast, TNFR1 attenuates lung inflammation in response to live bacteria but does not contribute to increased lung permeability and is not required for the elimination of P. aeruginosa.

Role of interleukin-1β, interleukin-6 and macrophage inflammatory protein-1β in prostaglandin-E2-induced hyperthermia in rats

Life Sciences ◽  
1996 ◽  
Vol 59 (12) ◽  
pp. PL185-PL190 ◽  
Author(s):  
A. Fernández-Alonso ◽  
K. Benamar ◽  
M. SancibriÁn ◽  
F.J. López-Valpuesta ◽  
F.J. Mifiano
Keyword(s):  
Prostaglandin E2 ◽  
Interleukin 6 ◽  
Interleukin 1Β ◽  
Induced Hyperthermia ◽  
Inflammatory Protein ◽  
Macrophage Inflammatory Protein
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