Inheritance of the Resistant Trait in Goldfish against Herpesviral Hematopoietic Necrosis

2018 ◽  
Vol 53 (4) ◽  
pp. 117-123
Author(s):  
Mikio Tanaka ◽  
Keitaro Dairiki ◽  
Mayuri Nakajima ◽  
Goshi Kato ◽  
Takashi Sakamoto ◽  
...  
Keyword(s):  

Weed Science ◽  
1995 ◽  
Vol 43 (1) ◽  
pp. 95-102 ◽  
Author(s):  
George P. Stallings ◽  
Donald C. Thill ◽  
Carol A. Mallory-Smith ◽  
Bahman Shafii

The movement of sulfonylurea herbicide-resistant (R) kochia pollen was investigated in a spring barley field near Moscow, ID, using a Nelder plot design in 1991 and 1992. Each 61 m diameter plot had 16 rays spaced 22.5° apart and contained 211 kochia plants. There were 12 susceptible (S) plants and one R plant along each ray. The R and S plants were 1.5 m and 3.0 to 30.5 m from the center of the plot, respectively. Wind direction and speed in the 16 vectors, air and soil temperature, and rainfall were monitored continuously. Mature kochia seed was collected from individual plants, planted in the greenhouse, and sprayed with chlorsulfuron to test for resistant F1progeny. Results from the 2-yr study showed outcrossing of R pollen onto S plants at rates up to 13.1% per plant 1.5 m from the R plants and declining to 1.4% per plant or less 29 m from the R plants. At least 35% of the total R x S crosses occurred in the direction of prevailing southeastward winds. Predicted percentages of R x S crosses per plant ranged from 0.16 to 1.29 at 1.5 m, and 0.00 to 0.06% at 29 m. Thus, resistant kochia pollen can spread the sulfonylurea-resistant trait at least 30 m during each growing season.



PLoS ONE ◽  
2020 ◽  
Vol 15 (5) ◽  
pp. e0233503
Author(s):  
Huirong Dong ◽  
Delin Wang ◽  
Zhijing Bai ◽  
Yuge Yuan ◽  
Wei Yang ◽  
...  


2019 ◽  
Vol 10 ◽  
Author(s):  
Daryn Sutton ◽  
Paul G. Livingstone ◽  
Eleanor Furness ◽  
Martin T. Swain ◽  
David E. Whitworth


HortScience ◽  
2019 ◽  
Vol 54 (5) ◽  
pp. 783-786
Author(s):  
Jesús Enrique Retes-Manjarrez ◽  
Sergio Hernández-Verdugo ◽  
Carlos Alfonso López-Orona ◽  
Raymundo Medina-López ◽  
José Antonio Garzón-Tiznado ◽  
...  

Pepper huasteco yellow vein virus (PHYVV) is a major disease in pepper (Capsicum annuum) that causes quantitative and qualitative losses to the crop in Central America and part of North America. To date, no resistant cultivars are available, and breeding is hampered by the lack of knowledge of the inheritance of this trait. Sources of resistance to PHYVV have been identified in the wild peppers of Mexico. The objectives of this study were to determine the grade of dominance, to analyze the maternal influence, and to estimate the number of genes involved in this resistant trait to PHYVV in the resistant wild pepper accession UAS12. Three susceptible parent lines—‘Anaheim’ (Ana), ‘Ancho Gigante’ (AG), and ‘Yolo Wonder’ (YW)—were crossed with resistant UAS12 accession to develop F1 (reciprocal), F2, and BC1 progenies in three families. Plants from this study were inoculated with PHYVV through Bemisia tabaci, evaluated phenotypically, and the segregation of disease scores was studied. A single recessive gene was found to control resistance to PHYVV in the resistant UAS12 accession, although segregation patterns suggested that other minor genes could participate in the expression of this resistant trait. No proof was found for maternal inheritance of PHYVV resistance. The gene symbol phv is proposed for PHYVV resistance in UAS12 accession in pepper. These results provide useful information for the design of pepper breeding programs in the introgression of this trait into commercial pepper backgrounds.



2018 ◽  
Vol 96 (1) ◽  
pp. 52 ◽  
Author(s):  
Jesús Enrique Retes-Manjarrez ◽  
Sergio Hernández-Verdugo ◽  
Benedicte Pariaud ◽  
Luis Alberto Hernández-Espinal ◽  
Saúl Parra-Terraza ◽  
...  

<p><strong>Background: </strong><em>Pepper huastecto yellow vein virus</em> (PHYVV) is one of the main viruses affecting pepper (<em>Capsicum</em> spp.) plants in Mexico.</p><p><strong>Question: </strong>Why there are no pepper resistant cultivars to PHYVV currently? Could it be due for the lack of new pepper resistant sources and knowledge about the heritability of the resistant trait?</p><p><strong>Study species: </strong><em>Capsicum annuum, Pepper huasteco yellow vein virus </em>and<em> Bemisia tabaci</em>.</p><p><strong>Study site: </strong>Culiacan<strong> </strong>Sinaloa, Mexico; January 2013 to August 2014.</p><p><strong>Methods: </strong>Two assays were performed in 2013 and 2014 with three resistant wild lines of <em>Capsicum annuum</em> in the S2 and S3 generation under greenhouse conditions to analyze the resistance to the <em>Pepper huasteco yellow vein virus</em> (PHYVV) and its heritability. Plants were inoculated with PHYVV through <em>Bemisia tabaci</em> G. and by grafting.</p><p><strong>Results: </strong>Line UAS12 showed a significantly higher proportion of resistant plants, longer incubation time, and less amount of viral DNA, followed by lines UAS13, UAS10 and the Maverick cultivar under both inoculation methods in both assays. Distribution of symptoms revealed a bimodal tendency in both assays. The novel gene "<em>CchGLP</em>" which confer resistance to PHYVV in pepper plants, was identified in the three lines evaluated on this study. Heritability of line UAS12 was of 0.35 and 0.26 in the insects and grafting inoculations, and of 0.58 and 0.10 in the first and second assays, respectively. Lines UAS13 and UAS10 showed close to zero heritability in the first and second assays with both inoculation methods.</p><strong>Conclusions: </strong>Line UAS12 is the most promising genetic resource for its high resistance and for showing heritability for the resistance trait. The intermediate resistance of lines UAS13 and UAS10 could be also useful for breeding programs. At least two genes are involved in the resistance trait to PHYVV. Part of the resistance shown in these lines may be due to the presence of the "<em>CchGLP</em>" gene. Line UAS12 count with variability for the resistant trait and can, therefore, be used to improve resistance and the other two lines possibly are stable as they did not show heritability.



1999 ◽  
Vol 13 (1) ◽  
pp. 100-103 ◽  
Author(s):  
G. Anthony Ohmes ◽  
J. Andy Kendig

A greenhouse experiment was conducted from 1995 to 1996 to determine the inheritance of ALS-cross-resistant common cocklebur. Two known biotypes (ALS-susceptible and ALS-resistant) were used in the experiment. Susceptible biotypes were crossed with pollen from resistant biotypes. F1 plants were self-pollinated. F2 plants were sprayed postemergence (POST) with a tank mixture of imazaquin at 280 g ai/ha and chlorimuron at 18 g ai/ha. Three distinct responses were observed: (1) no apparent effect (resistant), (2) yellowing of leaves with live terminals (intermediate), and (3) yellow leaves with dead terminals (typical symptoms of an ALS-susceptible common cocklebur). Chi-square tests indicated that an incomplete dominance model did not fit the data. Responses one and two were, therefore, combined and chi-square tests indicated a complete dominance model fit the data. This indicates that the cross-resistant trait is a dominant to semidominant trait.





PLoS ONE ◽  
2017 ◽  
Vol 12 (5) ◽  
pp. e0177470 ◽  
Author(s):  
Zongxiang Zhan ◽  
Chinedu Charles Nwafor ◽  
Zhaoke Hou ◽  
Jianfang Gong ◽  
Bin Zhu ◽  
...  


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