scholarly journals Quantitative description of publications (1986-2020) related to Alzheimer disease and oxidative stress: A bibliometric study

2021 ◽  
Vol 13 (1) ◽  
pp. 971-984
Author(s):  
Entesar Yaseen Abdo QAID ◽  
Idris LONG ◽  
Khairunnuur Fairuz AZMAN ◽  
Asma Hayati AHMAD ◽  
Zahiruddin OTHMAN ◽  
...  
2012 ◽  
Vol 11 (6) ◽  
pp. 3295-3304 ◽  
Author(s):  
Daniela Hartl ◽  
Victoria Schuldt ◽  
Stephanie Forler ◽  
Claus Zabel ◽  
Joachim Klose ◽  
...  

2014 ◽  
Vol 2 (1) ◽  
pp. 85-94
Author(s):  
Shahnaz Babaei Abraki ◽  
Sara Chavoshi-Nezhad ◽  
◽  

2011 ◽  
Vol 138-139 ◽  
pp. 1179-1182
Author(s):  
Chang Jun Lin ◽  
Han Chang Huang ◽  
Wen Juan Liu ◽  
Zhao Feng Jiang

Mitochondrial dysfunction and oxidative stress are among the earliest events linked to Alzheimer Disease and might play a causative role in disease onset and progression. In this paper, a biomedical imaging and visualization model was set to investigate the mitochondrial dysfunction and oxidative stress in Aβ/Cu2+-treated cells, which contribute in a significant manner to bioenergetic failure and mitochondrial dysfunction, by measuring mitochondrial membrane potential, Ca2+, chromatin condensation and ROS. This evaluating method opens a window for analyzing the protective effect of a certain substance in improving mitochondrial function and preventing oxidative stress in AD development.


2001 ◽  
Vol 15 (1) ◽  
pp. 51-55 ◽  
Author(s):  
Federico Licastro ◽  
Steve Pedrini ◽  
Lizabeth Jane Davis ◽  
Ludovica Caputo ◽  
Jacopo Tagliabue ◽  
...  

2017 ◽  
Vol 22 (2) ◽  
pp. 110-118 ◽  
Author(s):  
Mohammad Gol ◽  
Davoud Ghorbanian ◽  
Nabiollah Soltanpour ◽  
Jamshid Faraji ◽  
Mohsen Pourghasem

Author(s):  
Annarita Cito ◽  
Brunetta Porcelli ◽  
Maria Grazia Coppola ◽  
Paola Mangiavacchi ◽  
Alessio Cortelazzo ◽  
...  

Author(s):  
George Perry ◽  
Clyde F. Phelix ◽  
Akihiko Nunomura ◽  
Luis V. Colom ◽  
Rudy J. Castellani ◽  
...  

2002 ◽  
Vol 2 (3) ◽  
pp. 120-123 ◽  
Author(s):  
George Perry ◽  
Adam D. Cash ◽  
Mark A. Smith

Research in Alzheimer disease has recently demonstrated compelling evidence on the importance of oxidative processes in its pathogenesis. Cellular changes show that oxidative stress is an event that precedes the appearance of the hallmark pathologies of the disease, neurofibrillary tangles, and senile plaques. While it is still unclear what the initial source of the oxidative stress is in Alzheimer disease, it is likely that the process is highly dependent on redox-active transition metals such as iron and copper. Further investigation into the role that oxidative stress mechanisms seem to play in the pathogenesis of Alzheimer disease may lead to novel clinical interventions.


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