Estimate levels of immunoglobulin G, M and AST, ALT in people infected with COVID-19, and the relation of infection with a type of blood group in Al-Najaf Governorate, Iraq

2021 ◽  
Vol 15 (11) ◽  
Keyword(s):  
Transfusion ◽  
2005 ◽  
Vol 45 (1) ◽  
pp. 56-62 ◽  
Author(s):  
Shinya Kimura ◽  
Kimiko Yurugi ◽  
Hidekazu Segawa ◽  
Junya Kuroda ◽  
Kiyoshi Sato ◽  
...  

1970 ◽  
Vol 24 (03/04) ◽  
pp. 419-431 ◽  
Author(s):  
G de Gaetano ◽  
J Vermylen ◽  
M Verstraete

SummaryA patient with severe chronic thrombocytopenic purpura is described, whose serum, after repeated transfusions of platelet-rich plasma, of her ABO blood group, was found to contain immunoglobulin G, which produced clumping in platelet-rich plasma from 15 volunteers out of 20. Direct and indirect evidence was obtained that clumping was due to release of endogenous platelet-ADP. In addition, a direct effect of the immunoglobulin preparation on the platelet membrane could be demonstrated.


Author(s):  
Parul Chopra ◽  
Sunanda Bhardwaj ◽  
Ajay Samkaria ◽  
Asha Amoli ◽  
Anil Arora

Abstract Background A variation in the measurement of ABO antibody titer has been seen among different laboratories due to lack of standardization. In our study, we aim to evaluate automated ABO isoagglutinin titer measurements by erythrocytes magnetized technology (EMT) and compare with conventional tube technique (TT). Methods We performed ABO isoagglutinin titration on samples received in a reference laboratory during a period of 2 months. A total of 134 tests for immunoglobulin G (IgG) titer and 116 for immunoglobulin M (IgM) for anti-A or anti-B were included in the study. Samples were processed for ABO isoagglutination titers by both TT and EMT by QWALYS-3 (Diagast, France). Microsoft Excel was used to compile data, for all calculations, and to draw graphs and plots. The number and percentage of cases within ±1, ±2, or ±3 titer difference (TT-EMT) were calculated. Results Median titers and their ranges obtained by EMT were higher or equal to those by TT for all IgM and IgG ABO-antibodies in all blood group (BGs), except anti-A IgM in (BG) O that was lower by EMT (32 [4:128]) than TT (48 [8:256]). One twenty one (121/134, 90.3%) cases of IgG titer showed an agreement by both methods (within ± one titer difference). One hundred seven cases (107/116, 92.2%) for IgM titer were within one titer difference by both the methods. Conclusion Results of titration by EMT-based automated instrument QWALYS-3 and conventional TT may vary by one titer dilution in the majority of cases. Use of consistent method for patient management is, therefore, advised.


Author(s):  
Hannah R. Brown ◽  
Anthony F. Nostro ◽  
Halldor Thormar

Subacute sclerosing panencephalitis (SSPE) is a slowly progressing disease of the CNS in children which is caused by measles virus. Ferrets immunized with measles virus prior to inoculation with the cell associated, syncytiogenic D.R. strain of SSPE virus exhibit characteristics very similar to the human disease. Measles virus nucleocapsids are present, high measles antibody titers are found in the sera and inflammatory lesions are prominent in the brains. Measles virus specific immunoglobulin G (IgG) is present in the brain,and IgG/ albumin ratios indicate that the antibodies are synthesized within the CNS.


1980 ◽  
Vol 43 (02) ◽  
pp. 137-140 ◽  
Author(s):  
Jan Erikssen ◽  
Erik Thaulow ◽  
Helge Stormorken ◽  
Ole Brendemoen ◽  
Arvid Hellem

SummaryThe view based on epidemiological and laboratory data that blood group A subjects (=A) have clinically significant higher thrombotic potential than blood group 0 subjects (= O), is supported by the present finding of a significantly higher platelet retention in A than 0.The completely normal ABO distribution found among 71 cases of proven latent CHD, and the disproportionate excess of 0 vs. A in a consecutive series of 191 coronary artery bypass candidates apparently conflict with epidemiological data indicating a higher risk of achieving CHD in A than 0. The conflict may be solved by suggestinga) that the »thrombotic proneness« in A compared with 0 causes a poorer prognosis in CHD among the former, leaving a disproportionate excess of 0 among longterm CHD survivors, and b) that AB0-related factors have had an insignificant, independent impact on the evolution of preclinical coronary artery disease in our 71 men with latent CHD.


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