Histopathological changes in Labeo rohita (Hamilton) fingerlings to various acclimation temperatures

2011 ◽  
Vol 2 (1) ◽  
pp. 29 ◽  
Author(s):  
Mukti Chanda ◽  
Monjit Paul ◽  
Gadadhar Dash ◽  
Prathistha Yonzone
Keyword(s):  
Labeo Rohita ◽  
Histopathological Changes

scholarly journals Biochemical and haematological responses to thermal stress in Labeo rohita (Hamilton, 1822) fingerlings

2019 ◽  
Vol 66 (2) ◽  
Author(s):  
Prem Kumar ◽  
Asim K. Pal ◽  
Narottam P. Sahu ◽  
L. Christina ◽  
Ashish Kumar Jha
Keyword(s):  
Thermal Stress ◽  
Thermal Shock ◽  
Normal Condition ◽  
Biochemical Parameters ◽  
Labeo Rohita ◽  
Histopathological Changes ◽  
Respiratory Burst Activity ◽  
Serum Total Protein ◽  
Glucose 6 Phosphate Dehydrogenase ◽  
Stress Parameters

An experiment was conducted to understand the effect of thermal shock on stress and recovery in Labeo rohita (Hamilton, 1822) fingerlings. L. rohita fingerlings were exposed to a temperature of 380C for  2 h and permitted to recuperate afterwards for 48 h. Primary and secondary stress parameters were analysed after 2, 4, 8, 12, 24 and 48 h of recovery. The activity of glucose-6-phosphate dehydrogenase and acetylcholine esterase reduced significantly (p<0.05). Erythrocytes, haemoglobin, respiratory burst activity and lysozyme levels increased significantly (p<0.05) by 7.31, 23.91, 109.09 and 70.13% after thermal shock, whereas leucocytes, serum protein, albumin and globulin showed a reverse trend. During recovery, biochemical parameters normalised in 48 h. However, immunological variables such as serum total protein, albumin and globulin, and histopathological changes in gill and kidney tissues did not return to normal condition.

scholarly journals Histopathological changes in the gill architecture of Labeo rohita from the pond adjacent to thermal power station, Koradi, Nagpur, India

2011 ◽  
Vol 3 (2) ◽  
pp. 284-286
Author(s):  
A. S. Deshpande ◽  
S. B. Zade ◽  
S. R. Sitre
Keyword(s):  
Power Plant ◽  
Freshwater Fish ◽  
Thermal Power Plant ◽  
Fish Assemblages ◽  
Labeo Rohita ◽  
Thermal Power ◽  
Chloride Cells ◽  
Thermal Plume ◽  
Histopathological Changes ◽  
Power Plant Effluent

The hazardous effect of water pollution on the histopathology of selected organs of the freshwater fish Labeo rohita was studied with respect to thermal power plant effluent. The purpose of this research paper is to study whether the thermal plume can affect fish assemblages in the waters around the outlet area of the thermal power plant located at Koradi near Nagpur city. Both experimental and control fishes were monitored for a year from March 2010 to Feb. 2011. Histopathology serve as a tool to assess the extent of damage caused to the organism.Gill tissue showed fusion of primary lamellae, congestion of blood vessels and hyperplasia of bronchial plates. The histopathological changes in the gill tissue include epithelial hyperplasia, curling of secondary lamellae and changes in chloride cells. Besides these changes pyknotic nuclei, vacuolization, degradation of epithelial and pillar cells, were noticed, which point out towards deleterious effect of thermal power plant effluent on the freshwater fish L. rohita.

scholarly journals UJI TOKSISITAS SODIUM SIANIDA (NaCN) PADA BEBERAPA SPESIES IKAN AIR TAWAR: REVIEW (The Toxicity Test of Sodium Cyanide (NaCN) to Some Species of Freshwater Fish: A Review)

2020 ◽  
Vol 25 (1) ◽  
pp. 1
Author(s):  
Siti Muyassaroh ◽  
Indah Rahmatiah Siti Salami
Keyword(s):  
Carboxylic Acid ◽  
Freshwater Fish ◽  
Enzyme System ◽  
Labeo Rohita ◽  
Aquatic Organisms ◽  
Sodium Cyanide ◽  
Histopathological Changes ◽  
Free Medium ◽  
Fish Kills ◽  
Cyanide Toxicity

AbstrakSianida telah digunakan sebagai senyawa toksik selama beberapa dekade untuk penangkapan ikan. Pengetahuan para nelayan yang rendah dapat menyebabkan kelebihan dosis penggunaan senyawa sianida. Hal ini dapat menyebabkan kematian ikan dan bahkan kerusakan terumbu karang. Sodium sianida tidak bersifat bioakumulasi dan biomagnifikasi berdasarkan nilai Kow-nya dan sebagian besar metabolit dikeluarkan melalui urin dalam bentuk thiosianat, SCN- (60-80%), 2-aminothiazoline-4-carboxylic acid (ATCA) atau 2-iminothiazolidine-4-carboxylic acid, ITCA (15%), serta gas HCN dan CO2. Biomarker organisme yang terpapar sianida yaitu kandungan CN-, SCN-,ATCA atau ITCA pada urin dan darah; perubahan histopatologis di limpa, hepato-renal dan ginjal; penurunan aktivitas enzim katalase pada jaringan hati, insang, otak, dan otot ikan; perubahan aktivitas laktat dehydrogenase (LDH) dan suksinat dehydrogenase (SDH), tingkah laku, laju respirasi, dan metabolit (asam piruvat dan asam laktat). Akan tetapi, aktivitas enzim dan struktur histopatologis kembali normal setelah pemulihan selama 14 hari di medium bebas NaCN. Paparan NaCN juga dapat menyebabkan perubahan kecepatan renang dan durasi surfacing behavior. Konsentrasi sianida di lingkungan tidak bisa diabaikan, sebab dimungkinkan adanya efek sinergis dan juga penghambatan sistem enzim katalase, yang pada akhirnya terjadi kerentanan organisme akuatik terhadap toksisitas sianida. Labeo rohita merupakan spesies ikan air tawar yang paling sensitif terhadap paparan NaCN, dengan nilai LC50 sebesar 0,32 mg/L (tanpa melihat jenis aliran yang digunakan).AbstractCyanide has been used as a toxic compound for decades for fishing. Lack of knowledge of fishermen can lead to overdosing use of a compound. This can lead to fish kills and even damage the coral reefs. Sodium cyanide is not bioaccumulative and biomagnificative based on the value of Kow and most metabolites are excreted in the urine in the form of SCN- (60-80%), 2-aminothiazoline-4-carboxylic acid, ATCA or 2-iminothiazolidine-4-carboxylic acid, ITCA (15%), as well as HCN gas and CO2. Biomarkers organisms are exposed cyanide content of CN-, SCN-, ATCA or ITCA on urine and blood; histopathological changes in spleen, hepato-renal and kidney; decreased activity of catalase enzyme in the liver, gills, brain, and muscles of fish; lactate dehydrogenase (LDH) and succinate dehydrogenase (SDH) activity changes, behavioral, respiratory rate, and metabolites (pyruvic acid and lactic acid). However, the enzyme activity and histopathological structure back to normal after recovery for 14 days at NaCN-free medium. NaCN exposure can also cause changes in swimming speed and duration of surfacing behavior. The concentration of cyanide in the environment can not be ignored, because it is possible the existence of a synergistic effect and also the inhibition of catalase enzyme system, which eventually happened vulnerability of aquatic organisms to cyanide toxicity. Labeo rohita is a freshwater fish species are most sensitive to exposure to NaCN, with LC50 values of 0.32 mg / L (regardless of the type of flow used).

Fine structure of early degenerating myofibers in the tibialis anterior of young ‘MDX’ mouse

1988 ◽  
Vol 46 ◽  
pp. 322-323
Author(s):  
H.D. Geissinger ◽  
C.K. McDonald-Taylor
Keyword(s):  
Fine Structure ◽  
Muscle Regeneration ◽  
Tibialis Anterior ◽  
Genetic Defect ◽  
Mdx Mouse ◽  
Mdx Mice ◽  
Histopathological Changes ◽  
Electron Microscopic ◽  
Dystrophic Mouse ◽  
Preliminary Study

A new strain of mice, which had arisen by mutation from a dystrophic mouse colony was designated ‘mdx’, because the genetic defect, which manifests itself in brief periods of muscle destruction followed by episodes of muscle regeneration appears to be X-linked. Further studies of histopathological changes in muscle from ‘mdx’ mice at the light microscopic or electron microscopic levels have been published, but only one preliminary study has been on the tibialis anterior (TA) of ‘mdx’ mice less than four weeks old. Lesions in the ‘mdx’ mice vary between different muscles, and centronucleation of fibers in all muscles studied so far appears to be especially prominent in older mice. Lesions in young ‘mdx’ mice have not been studied extensively, and the results appear to be at variance with one another. The degenerative and regenerative aspects of the lesions in the TA of 23 to 26-day-old ‘mdx’ mice appear to vary quantitatively.

The Fine Structure of Irradiated Mouse Heart

1976 ◽  
Vol 34 ◽  
pp. 184-185
Author(s):  
Vivian V. Yang ◽  
S. Phyllis Stearner
Keyword(s):  
Microscopic Level ◽  
Ultrastructural Changes ◽  
Mouse Heart ◽  
Blood Vessel Wall ◽  
Histopathological Changes ◽  
Light Microscopic Level ◽  
Γ Rays ◽  
Fission Neutrons ◽  
Total Body

The heart is generally considered a radioresistant organ, and has received relatively little study after total-body irradiation with doses below the acutely lethal range. Some late damage in the irradiated heart has been described at the light microscopic level. However, since the dimensions of many important structures of the blood vessel wall are submicroscopic, investigators have turned to the electron microscope for adequate visualization of histopathological changes. Our studies are designed to evaluate ultrastructural changes in the mouse heart, particularly in the capillaries and muscle fibers, for 18 months after total-body exposure, and to compare the effects of 240 rad fission neutrons and 788 rad 60Co γ-rays.Three animals from each irradiated group and three control mice were sacrificed by ether inhalation at 4 days, and at 1, 3, 6, 12, and 18 months after irradiation. The thorax was opened and the heart was fixed briefly in situwith Karnofsky's fixative.

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