scholarly journals Interactions and Effects on Cysteine Synthase Activity of Aminooxyacetate and Boc-Aminooxyacetate on the Bioherbicides <i>Colletotrichum truncatum</i> and <i>Alternaria cassia</i> and Their Weed Hosts

2021 ◽  
Vol 12 (05) ◽  
pp. 759-770
Author(s):  
Robert E. Hoagland ◽  
Kangetsu Hirase ◽  
C. Douglas Boyette
Nucleus ◽  
2015 ◽  
Vol 12 (1) ◽  
Author(s):  
Gabrielle de Lima MASSON ◽  
Bruno Agostini COLMAN ◽  
Paulo Rogério Beltramin da FONSECA ◽  
Alexandre Dinnys ROESE

2021 ◽  
Vol 12 (1) ◽  
Author(s):  
Sheng-Kai Sun ◽  
Xuejie Xu ◽  
Zhong Tang ◽  
Zhu Tang ◽  
Xin-Yuan Huang ◽  
...  

AbstractRice grains typically contain high levels of toxic arsenic but low levels of the essential micronutrient selenium. Anthropogenic arsenic contamination of paddy soils exacerbates arsenic toxicity in rice crops resulting in substantial yield losses. Here, we report the identification of the gain-of-function arsenite tolerant 1 (astol1) mutant of rice that benefits from enhanced sulfur and selenium assimilation, arsenic tolerance, and decreased arsenic accumulation in grains. The astol1 mutation promotes the physical interaction of the chloroplast-localized O-acetylserine (thiol) lyase protein with its interaction partner serine-acetyltransferase in the cysteine synthase complex. Activation of the serine-acetyltransferase in this complex promotes the uptake of sulfate and selenium and enhances the production of cysteine, glutathione, and phytochelatins, resulting in increased tolerance and decreased translocation of arsenic to grains. Our findings uncover the pivotal sensing-function of the cysteine synthase complex in plastids for optimizing stress resilience and grain quality by regulating a fundamental macronutrient assimilation pathway.


1988 ◽  
Vol 78 (4) ◽  
pp. 673-682 ◽  
Author(s):  
Garrick McDonald ◽  
A. Mark Smith

AbstractPopulations of Nysius vinitor Bergroth were studied from 1979 to 1982 in two weed hosts, Arctotheca calendula and Polygonum aviculare, and eight irrigated sunflower crops in a summer cropping area of northern Victoria, Australia. The spring generation began with the adults colonizing flowering A.calendula plants in September and concluded with the rapid development of late stage nymphs and an exodus of adults from these plants from mid-November to December. Gradual invasion of sunflowers occurred mostly in late December and reached a peak at flowering, after which nymphs appeared. P. aviculare attracted adults from February and hosted a number of overlapping generations until winter. The weed sustained diminishing numbers of adults through the winter, except in 1982, when a further generation produced an early spring peak. Immigrant populations were regarded as a common source of adults for initiating the spring and summer generations. The rate of development of N. vinitor in spring was more rapid than that predicted by phenological simulation based on ambient temperatures and laboratory-derived day-degree estimates. This was attributed to increased body temperatures through absorption of solar radiation, and the simulation model was adjusted by increasing daily minimum and maximum temperatures by 1·3 and 5·5°C for young and older instars, respectively. This suggested that older nymphs have lower developmental thresholds or are better able to optimize body temperatures.


2012 ◽  
Vol 7 (1) ◽  
pp. 193-195 ◽  
Author(s):  
M. J. Fuhlbohm ◽  
M. J. Ryley ◽  
E. A. B. Aitken

Plant Disease ◽  
2015 ◽  
Vol 99 (1) ◽  
pp. 14-20 ◽  
Author(s):  
C. A. Ellison ◽  
A. Sawadogo ◽  
S. Braman ◽  
S. Nacro

A new disease was identified on the biofuel crop Jatropha curcas in 2012 in Burkina Faso that is causing serious yield losses. The disease was found to be widespread in both Sissili and Comoé Provinces. It causes characteristic leaf lesions, fruit necrosis, and cankers on young stems and branches. There was evidence of multiple infections on plants over the growing season, with regrowth evident from below old cankers, but there was little fruit production from infected branches. A detailed monitoring and assessment was undertaken of the disease progress in a severely infected field, over a 7-week period. The disease symptoms progressed from chlorosis through a necrotic phase and, in approximately 83% of replicates, stem cankers developed that resulted in dieback and lodging of branches. Colletotrichum truncatum and a member of the species complex C. gloeosporioides sensu lato were consistently isolated from fresh stem samples showing early symptoms (chlorosis). Koch's postulates were undertaken, to establish the pathogenicity of the two species. No symptoms were observed on plants inoculated with C. gloeosporioides; however, leaf and stem lesions developed after inoculation with C. truncatum, which was reisolated from the diseased tissue, confirming it as the disease-causing agent. Preliminary management practices for the disease are proposed.


2001 ◽  
Vol 158 (5) ◽  
pp. 655-661 ◽  
Author(s):  
Emiko Harada ◽  
Yong-Eui Choi ◽  
Atsunari Tsuchisaka ◽  
Hitoshi Obata ◽  
Hiroshi Sano

Author(s):  
M. B. Ellis

Abstract A description is provided for Pyrenophora tritici-repentis. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Wheat, rye, barley, Agropyrons repens, Cynodon dactylon, Elymus glaucus. DISEASES: Causes yellow leaf spot of cereals and grasses; oval to lanceolate. yellow to grey brown lesions often with a yellow halo. The lesions can be distinguished from those caused by Cochliobolus sativus because of their lighter colour. The disease results in premature death of leaves. It can also cause a seedling blight and root rot. Common and widespread on Agropyron repens[Elymus repens] and wheat, occasionally on barley and rye and recorded on many other grasses. Sometimes causes severe leaf wilt and spotting especially on durum wheat. Leaves of Agropyron repens[Elymus repens] when attacked gradually lose their colour and wither from the tips backwards; they become at first pale yellow, later grey. On wheat fusiform, oval or lanceolate spots, 0.5-2 cm long, 2-4 mm wide are formed. These are at first yellow but later turn brown or greyish brown often with a yellow halo. The leaves die prematurely from the tip backwards. GEOGRAPHICAL DISTRIBUTION: Australia; Asia (Japan, India, Nepal); Africa (Uganda, Kenya, Tanzania, Ethiopia); Europe (Britain, Germany, Czechoslovakia, Sweden, Cyprus); S. America (Bolivia); and N. America (Canada, USA). TRANSMISSION: Air-borne spores (51, 1045p), seed-borne by both external contamination and internal infection (34, 24), secondary grass weed hosts (11, 695), carryover on stubble and other crop debris (43, 1225f; 52, 685).


Author(s):  
J. F. Bradbury

Abstract A description is provided for Xanthomonas oryzae. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: On Oryza sativa. Natural infection of Leersia oryzaides, Leersia oryzaides var. japonica and Zizania latifolia[Zizania aquatica] is known (Mizukami & Wakimoto, 1959). Natural infection of Cyperus rotundus and C. difformis was reported in India (48, 460), but inoculation of these hosts in the Philippines failed (48, 1689). By inoculation many wild species of Oryza (45, 1789), Leptochloa filiformis (48, 1689), L. chinensis, L. panacea and Zizania aquatica (48, 1683) have all been found susceptible. DISEASE: Bacterial blight of rice and kresek disease of rice. Blight most commonly appears on leaves of young plants, after planting out, as water-soaked stripes at the margins. These enlarge and coalesce to give the characteristic yellowish lesions with wavy edges that occur mainly along the margins of the upper parts of the leaves. These lesions may later expand to cover much of the leaf, which turns whitish or greyish and dies. Leaf sheaths of the more susceptible varieties may be affected. Kresek, which occurs in tropical regions, is a strong systemic infection in which leaves or whole young plants wither and die. In older plants the leaves become pale yellow. GEOGRAPHICAL DISTRIBUTION: Limited to Asia (CMI Map 304, ed. 2, 1964). Occurrences not shown on this map include Korea, Taiwan and Indonesia (Mizukami & Wakimoto, 1969). TRANSMISSION: Spread locally by wind and rain and also by flood and irrigation waters; gains entry to the field in infected planting material, to the nursery in seed, and to both nursery and field from volunteer rice plants and weed hosts usually via the irrigation water. Overwintering may occur on volunteers, on or in the rhizospheres of weed hosts, in stored infected straw and in seed, but it is unlikely in soil and plant debris exposed to the weather, at least under Japanese conditions (Mizukami & Wakimoto, 1969). Infection is through hydathodes and wounds. Penetration through stomata results in a build-up of bacteria in the intercellular spaces, but it is not until they have been exuded on to the leaf surface and re-admitted through the hydathodes and thence into the vascular system, that symptoms of the disease appear (46, 2720).


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