scholarly journals The prevalence of clinical signs of ankle instability in club rugby players

2013 ◽  
Vol 25 (1) ◽  
Author(s):  
Aimee Vivienne Stewart ◽  
Eloize Mellet
Keyword(s):  
Ankle Instability ◽  
Clinical Signs ◽  
Rugby Players

scholarly journals The prevalence of clinical signs of ankle instability in club rugby players

2013 ◽  
Vol 25 (1) ◽  
pp. 23
Author(s):  
E Mellet ◽  
A Stewart
Keyword(s):  
Ankle Instability ◽  
Clinical Signs ◽  
Ankle Injuries ◽  
Mechanical Instability ◽  
Demographic Questionnaire ◽  
High Prevalence ◽  
History Of ◽  
Rugby Players ◽  
Balance Error ◽  
Ethical Clearance

Background. Ankle injuries are one of the most common injuries in sport and have a high recurrence rate.Aim. To determine the prevalence of clinical signs of ankle injuries in club rugby players in South Gauteng.Methods. Institutional ethical clearance was obtained for the study. Of the 180 players from 9 clubs who were eligible for participation in thestudy, 76% (n=137) were recuited. Informed consent was obtained before players were asked to complete a battery of tests. Each player wasasked to complete a demographic questionnaire and the Olerud and Molander questionnaire to determine the prevalence of clinical signs ofperceived instability. The prevalence of clinical signs of mechanical instability was determined by the anterior drawer test (ADT) and talartilt test (TTT). Balance and proprioception were assessed by the Balance Error Scoring System (BESS) and this was used to determine theprevalence of clinical signs of functional instability.Results. The prevalence of perceived instability was 44%. The prevalence of clinical signs of mechanical ankle instability was 33%. There was anincreased prevalence of mechanical instability in players who had a history of previous ankle injuries: ADT left (p=0.003); ADT right (p=0.01);TTT left (p=0.001); TTT right (p=0.08), both tests positive left (p=0.001) and both tests positive right (p=0.03). The prevalence of clinical signsof functional ankle instability depended on the surface and visual input, and was greater as the challenge or perturbation increased.Conclusion. There was a high prevalence of clinical signs of ankle instability in club rugby players for perceived, mechanical and functionalinstability. Those with previously injured ankles were more likely to have unstable ankles.

scholarly journals The prevalence of clinical signs of ankle instability in club rugby players

2013 ◽  
Vol 25 (1) ◽  
pp. 23
Author(s):  
E Mellet ◽  
A Stewart
Keyword(s):  
Ankle Instability ◽  
Clinical Signs ◽  
Ankle Injuries ◽  
Mechanical Instability ◽  
Demographic Questionnaire ◽  
High Prevalence ◽  
History Of ◽  
Rugby Players ◽  
Balance Error ◽  
Ethical Clearance

Background. Ankle injuries are one of the most common injuries in sport and have a high recurrence rate.Aim. To determine the prevalence of clinical signs of ankle injuries in club rugby players in South Gauteng.Methods. Institutional ethical clearance was obtained for the study. Of the 180 players from 9 clubs who were eligible for participation in thestudy, 76% (n=137) were recuited. Informed consent was obtained before players were asked to complete a battery of tests. Each player wasasked to complete a demographic questionnaire and the Olerud and Molander questionnaire to determine the prevalence of clinical signs ofperceived instability. The prevalence of clinical signs of mechanical instability was determined by the anterior drawer test (ADT) and talartilt test (TTT). Balance and proprioception were assessed by the Balance Error Scoring System (BESS) and this was used to determine theprevalence of clinical signs of functional instability.Results. The prevalence of perceived instability was 44%. The prevalence of clinical signs of mechanical ankle instability was 33%. There was anincreased prevalence of mechanical instability in players who had a history of previous ankle injuries: ADT left (p=0.003); ADT right (p=0.01);TTT left (p=0.001); TTT right (p=0.08), both tests positive left (p=0.001) and both tests positive right (p=0.03). The prevalence of clinical signsof functional ankle instability depended on the surface and visual input, and was greater as the challenge or perturbation increased.Conclusion. There was a high prevalence of clinical signs of ankle instability in club rugby players for perceived, mechanical and functionalinstability. Those with previously injured ankles were more likely to have unstable ankles.

A rapid method for the direct identification of paramyxovirus in canine CSF by ultracentrifugation and negative staining as a rule out for distemper

1990 ◽  
Vol 48 (3) ◽  
pp. 594-595
Author(s):  
W.L. Steffens ◽  
M.B. Ard ◽  
C.E. Greene ◽  
A. Jaggy
Keyword(s):  
Subacute Sclerosing Panencephalitis ◽  
Clinical Signs ◽  
Viral Disease ◽  
Etiologic Agent ◽  
Mucosal Inflammation ◽  
Worldwide Distribution ◽  
Negative Stain ◽  
Viral Particles ◽  
Systemic Manifestations ◽  
Rule Out

Canine distemper is a multisystemic contagious viral disease having a worldwide distribution, a high mortality rate, and significant central neurologic system (CNS) complications. In its systemic manifestations, it is often presumptively diagnosed on the basis of clinical signs and history. Few definitive antemortem diagnostic tests exist, and most are limited to the detection of viral antigen by immunofluorescence techniques on tissues or cytologic specimens or high immunoglobulin levels in CSF (cerebrospinal fluid). Diagnosis of CNS distemper is often unreliable due to the relatively low cell count in CSF (<50 cells/μl) and the binding of blocking immunoglobulins in CSF to cell surfaces. A more reliable and definitive test might be possible utilizing direct morphologic detection of the etiologic agent. Distemper is the canine equivalent of human measles, in that both involve a closely related member of the Paramyxoviridae, both produce mucosal inflammation, and may produce CNS complications. In humans, diagnosis of measles-induced subacute sclerosing panencephalitis is through negative stain identification of whole or incomplete viral particles in patient CSF.

Hormone Replacement Therapy and Voice

2019 ◽  
Vol 4 (4) ◽  
pp. 607-614
Author(s):  
Jean Abitbol
Keyword(s):  
Replacement Therapy ◽  
Symptom Severity ◽  
Low Dose ◽  
Hormone Replacement ◽  
Clinical Signs ◽  
Female Voice ◽  
Fat Cells ◽  
High Pitch ◽  
Endocrine Effects ◽  
The Voice

The purpose of this article is to update the management of the treatment of the female voice at perimenopause and menopause. Voice and hormones—these are 2 words that clash, meet, and harmonize. If we are to solve this inquiry, we shall inevitably have to understand the hormones, their impact, and the scars of time. The endocrine effects on laryngeal structures are numerous: The actions of estrogens and progesterone produce modification of glandular secretions. Low dose of androgens are secreted principally by the adrenal cortex, but they are also secreted by the ovaries. Their effect may increase the low pitch and decease the high pitch of the voice at menopause due to important diminution of estrogens and the privation of progesterone. The menopausal voice syndrome presents clinical signs, which we will describe. I consider menopausal patients to fit into 2 broad types: the “Modigliani” types, rather thin and slender with little adipose tissue, and the “Rubens” types, with a rounded figure with more fat cells. Androgen derivatives are transformed to estrogens in fat cells. Hormonal replacement therapy should be carefully considered in the context of premenopausal symptom severity as alternative medicine. Hippocrates: “Your diet is your first medicine.”

Die Aplasie der Vena cava inferior als Ursache rezidivierender Bein- und Beckenvenenthrombosen

VASA ◽  
1999 ◽  
Vol 28 (4) ◽  
pp. 289-292 ◽  
Author(s):  
Tiesenhausen ◽  
Amann ◽  
Thalhammer ◽  
Aschauer
Keyword(s):  
Inferior Vena Cava ◽  
Inferior Vena ◽  
Heart Defects ◽  
Vena Cava ◽  
Clinical Signs ◽  
Vena Cava Inferior ◽  
Caval Vein ◽  
Rare Finding ◽  
Thrombotic Complications ◽  
Asplenia Syndrome

Congenital anomalies of the caval vein are often associated with other abnormities such as heart defects, situs inversus or a polysplenia-asplenia-syndrome. An isolated, congenital malformation like aplasia of the inferior vena cava is a rare finding. A review of the embryology and abnormities, diagnostics, clinical signs and treatment is given together with the histories of two patients having thrombosis of the lower extremities and pelvic veins, caused by aplasia of the inferior vena cava. After thrombotic complications caused by vena cava aplasia there is high risk of recurrence. Those patients should be anticoagulated for lifetime.

SelenoproteinN1-related myopathy: From clinical signs to diagnosis

2011 ◽  
Vol 42 (S 01) ◽  
Author(s):  
E Alberg ◽  
S Lutz ◽  
W Kress ◽  
U Schara
Keyword(s):  
Clinical Signs

Sonographically guided, transcatheter foam sclerotherapy of the great saphenous vein

Phlebologie ◽  
2007 ◽  
Vol 36 (06) ◽  
pp. 309-312 ◽  
Author(s):  
T. Schulz ◽  
M. Jünger ◽  
M. Hahn
Keyword(s):  
Saphenous Vein ◽  
Great Saphenous Vein ◽  
Clinical Stage ◽  
Clinical Signs ◽  
Cost Effective ◽  
Duplex Sonography ◽  
Foam Sclerotherapy ◽  
Average Loss ◽  
One Year ◽  
The Cost

Summary Objective: The goal of the study was to assess the effectiveness and patient tolerability of single-session, sonographically guided, transcatheter foam sclerotherapy and to evaluate its economic impact. Patients, methods: We treated 20 patients with a total of 22 varicoses of the great saphenous vein (GSV) in Hach stage III-IV, clinical stage C2-C5 and a mean GSV diameter of 9 mm (range: 7 to 13 mm). We used 10 ml 3% Aethoxysklerol®. Additional varicoses of the auxiliary veins of the GSV were sclerosed immediately afterwards. Results: The occlusion rate in the treated GSVs was 100% one week after therapy as demonstrated with duplex sonography. The cost of the procedure was 207.91 E including follow-up visit, with an average loss of working time of 0.6 days. After one year one patient showed clinical signs of recurrent varicosis in the GSV; duplex sonography showed reflux in the region of the saphenofemoral junction in a total of seven patients (32% of the treated GSVs). Conclusion: Transcatheter foam sclerotherapy of the GSV is a cost-effective, safe method of treating varicoses of GSV and broadens the spectrum of therapeutic options. Relapses can be re-treated inexpensively with sclerotherapy.

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