Multicycle Performance of CaTiO3 Decorated CaO-Based CO2 Adsorbent Prepared by a Versatile Aerosol Assisted Self-Assembly Method

Calcium oxide (CaO) is a promising adsorbent to separate CO2 from flue gas. However, with cycling of carbonation/decarbonation at high temperature, the serious sintering problem causes its capture capacity to decrease dramatically. A CaTiO3-decorated CaO-based CO2 adsorbent was prepared by a continuous and simple aerosol-assisted self-assembly process in this work. Results indicated that CaTiO3 and CaO formed in the adsorbent, whereas CaO gradually showed a good crystalline structure with increased calcium loading. Owing to the high thermal stability of CaTiO3, it played a role in suppressing the sintering effect and maintaining repeated high-temperature carbonation and decarbonation processes. When the calcium and titanium ratio was 3, the CO2 capture capacity was as large as 7 mmol/g with fast kinetics. After 20 cycles under mild regeneration conditions (700 °C, N2), the performance of CO2 capture of CaTiO3-decorated CaO-based adsorbent nearly unchanged. Even after 10 cycles under severe regeneration conditions (920 °C, CO2), the performance of CO2 capture still remained nearly 70% compared to the first cycle. The addition of CaTiO3 induced good and firm CaO dispersion on its surface. Excellent kinetics and stability were evident.

An Early Calcium Loading during Cherry Tree Dormancy Improves Fruit Quality Features at Harvest

The possible role of an early calcium application via sprays (0.25, 0.5 and 1M CaCl2) on dormant buds to improve sweet cherry (cv. Ferrovia) fruit quality at harvest was investigated. Fruit quality characteristics were also investigated in response to the age of spurs, the ripening stage, and their interactions. Results indicate that calcium enters the dormant flower buds and the phloem but not to the dormant vegetative buds. At harvest, the levels of Zn, Mn, and Cu were declined in fruits by increasing CaCl2 doses of sprays. Fruit respiratory activity was higher and on–tree fruit cracking was lower in red-colour (unripe) cherries as well as in fruit that was produced by 2-year-old short spurs or by Ca-treated buds. Differences in the sweet cherry skin metabolic profiles were identified. Fruit produced from Ca-exposed spurs exhibited lower levels of ribose and other cell-wall-related sugars and higher sucrose, maltose, and quininic acid levels. Nutrient shift was increased in red cherries, while anthocyanins were boosted in the black ones. PCA analysis was performed between the high dose of calcium spray and a control for mineral element content and cherry quality traits. This study illustrates that the high dose of calcium application during bud dormancy can effectively improve sweet cherry fruit characteristics, in terms of calcium content, cracking incidence, and fruit set. Overall, the present study contributes to a better understanding of the impact of calcium nutrition in fruit crops, which will provide references for alternative nutrient management and quality control in sweet cherry production.

Early Cardiac Mitochondrial Molecular and Functional Responses to Acute Anthracycline Treatment in Wistar Rats

Doxorubicin (DOX) is an anticancer drug widely used to treat human and nonhuman tumors but the late and persistent cardio-toxicity reduces the therapeutic utility of the drug. The full mechanism(s) of DOX-induced acute, subchronic and delayed toxicity, which has a preponderant mitochondrial component, remains unclear; therefore, it is clinically relevant to identify early markers to identify patients who are predisposed to DOX-related cardiovascular toxicity. To address this, Wistar rats (16 weeks old) were treated with a single DOX dose (20 mg/kg, i.p.); then, mRNA, protein levels and functional analysis of mitochondrial endpoints were assessed 24 h later in the heart, liver, and kidney. Using an exploratory data analysis, we observed cardiac-specific alterations after DOX treatment for mitochondrial complexes III, IV, and preferentially for complex I. Conversely, the same analysis revealed complex II alterations are associated with DOX response in the liver and kidney. Interestingly, H2O2 production by the mitochondrial respiratory chain as well as loss of calcium-loading capacity, markers of subchronic toxicity, were not reliable indicators of acute DOX cardiotoxicity in this animal model. By using sequential principal component analysis and feature correlation analysis, we demonstrated for the first time alterations in sets of transcripts and proteins, but not functional measurements, that might serve as potential early acute markers of cardiac-specific mitochondrial toxicity, contributing to explain the trajectory of DOX cardiac toxicity and to develop novel interventions to minimize DOX cardiac liabilities.