Influence of passive humidification on respiratory heat loss in tracheotomized patients

Head & Neck ◽  
2006 ◽  
Vol 28 (7) ◽  
pp. 609-613 ◽  
Author(s):  
Ajnacska Rozsasi ◽  
Richard Leiacker ◽  
Yvonne Fischer ◽  
Tilman Keck

1971 ◽  
Vol 49 (5) ◽  
pp. 767-774 ◽  
Author(s):  
M. Berger ◽  
J. S. Hart ◽  
O. Z. Roy

Pulmonary ventilation and temperature of expired air and of the respiratory passages has been measured by telemetry during flight in the black duck (Anas rubripes) and the respiratory water and heat loss has been calculated.During flight, temperature of expired air was higher than at rest and decreased with decreasing ambient temperatures. Accordingly, respiratory water loss as well as evaporative heat loss decreased at low ambient temperatures, whereas heat loss by warming of the inspired air increased. The data indicated respiratory water loss exceeded metabolic water production except at very low ambient temperatures. In the range between −16 °C to +19 °C, the total respiratory heat loss was fairly constant and amounted to 19% of the heat production. Evidence for the independence of total heat loss and production from changes in ambient temperature during flight is discussed.



1989 ◽  
Vol 66 (6) ◽  
pp. 2788-2798 ◽  
Author(s):  
D. W. Ray ◽  
E. P. Ingenito ◽  
M. Strek ◽  
P. T. Schumacker ◽  
J. Solway

We assessed the longitudinal distribution of intra-airway heat and water exchanges and their effects on airway wall temperature by directly measuring respiratory fluctuations in airstream temperature and humidity, as well as airway wall temperature, at multiple sites along the airways of endotracheally intubated dogs. By comparing these axial thermal and water profiles, we have demonstrated that increasing minute ventilation of cold or warm dry air leads to 1) further penetration of unconditioned air into the lung, 2) a shift of the principal site of total respiratory heat loss from the trachea to the bronchi, and 3) alteration of the relative contributions of conductive and evaporative heat losses to local total (conductive plus evaporative) heat loss. These changes were not accurately reflected in global measurements of respiratory heat and water exchange made at the free end of the endotracheal tube. Raising the temperature of inspired dry air from frigid to near body temperature principally altered the mechanism of airway cooling but did not influence airway mucosal temperature substantially. When local heat loss was increased from both trachea and bronchi (by increasing minute ventilation), only the tracheal mucosal temperature fell appreciably (up to 4.0 degrees C), even though the rise in heat loss from the bronchi about doubled that in the trachea. Thus it appears that the bronchi are better able to resist changes in airway wall temperature than is the trachea. These data indicate that the sites, magnitudes, and mechanisms of respiratory heat loss vary appreciably with breathing pattern and inspired gas temperature and that these changes cannot be predicted from measurements made at the mouth. In addition, they demonstrate that local heat (and presumably, water) sources that replenish mucosal heat and water lost to the airstream are important in determining the degree of local airway cooling (and presumably, drying).



1961 ◽  
Vol 16 (4) ◽  
pp. 633-638 ◽  
Author(s):  
Robert W. Bullard ◽  
John R. Crise

Human subjects were exposed to an ambient temperature of 5 C for 75-min periods. Subjects breathed 2.5%—6% carbon dioxide for selected time periods during the exposure. Carbon dioxide appeared to inhibit shivering. After carbon dioxide inhalation, shivering and metabolism were greatly increased. When 6% carbon dioxide was inhaled for 30 min, the inhibition was overcome and shivering and metabolism approached high levels. The increased respiratory heat loss associated with carbon dioxide breathing may be one factor causing the breakthrough of the inhibition. Submitted on November 14, 1960



Thorax ◽  
1983 ◽  
Vol 38 (4) ◽  
pp. 320-320 ◽  
Author(s):  
D. Godden ◽  
S. Jamieson ◽  
T. Higenbottam




1995 ◽  
Vol 20 (6) ◽  
pp. 489-496 ◽  
Author(s):  
Matthew D. White ◽  
Michel Cabanac


2002 ◽  
Vol 46 (3) ◽  
pp. 136-140 ◽  
Author(s):  
Roberto Gomes da Silva ◽  
Newton LaScala ◽  
Alvaro Lima Filho ◽  
Marcelo Catharin


Lung ◽  
1990 ◽  
Vol 168 (1) ◽  
pp. 23-34 ◽  
Author(s):  
E. P. Ingenito ◽  
B. M. Pichurko ◽  
J. Lafleur ◽  
J. M. Drazen ◽  
R. H. Ingram ◽  
...  


1981 ◽  
Vol 50 (4) ◽  
pp. 829-834 ◽  
Author(s):  
C. A. Piantadosi ◽  
E. D. Thalmann ◽  
W. H. Spaur

To study the phenomenon of isolated core cooling, four resting men breathed cooled helium-oxygen (T in = 14 +/- 2 degrees C, 40-60% relative humidity) in a warm hyperbaric chamber at pressures equivalent to 640, 1,000, and 1,400, and 1,800 ft seawater (fsw). Rectal temperature (T re) fell by 0.43 +/- 0.13 degrees C at 640 fsw to 0.98 +/- 0.15 degrees C at 1,800 fsw after 60 min. The rate at which T re fell was linearly related to the product of inspired gas density times specific heat. The metabolic response (VO2) to this isolated core cooling was more closely related to the rate of fall in T re than to the magnitude of this fall. A distinct threshold temperature, below which a rise in VO2 would occur, was not demonstrable. However, when the rate of fall of T re exceeded 0.70 degrees C . h-1, VO2 increased above base line, in spite of high skin temperatures that may have blunted the VO2 response. When VO2 did increase, its net benefit on thermal homeostasis was negated by the associated rise in pulmonary ventilation and its attendant increase in respiratory heat loss. Breathing cool helium-oxygen under hyperbaric conditions can rapidly lower deep body temperature, even in the presence of a warm body surface.



Sign in / Sign up

Export Citation Format

Share Document