Cost-of-illness for non-underweight binge-eating disorders

Eating and Weight Disorders - Studies on Anorexia Bulimia and Obesity ◽

10.1007/s40519-021-01277-3 ◽

2021 ◽

Author(s):

Paul E. Jenkins

Keyword(s):

Eating Disorders ◽

Binge Eating ◽

Economic Impact ◽

Indirect Costs ◽

Cost Effective ◽

Level Of Evidence ◽

Academic Impairment ◽

The United Kingdom ◽

Binge Eating Disorders ◽

Low Weight

Abstract Purpose This study examined economic costs associated with untreated eating disorders (EDs) characterised by regular binge eating in the absence of low weight. Both direct and indirect costs were assessed, reporting a limited societal perspective of economic impact as some costs were not included. Methods One hundred and twenty six adults seeking treatment for recurrent binge eating were asked to report impairment associated with an ED. Costs were calculated using 2017 prices, including an examination of variables associated with costs. Results Estimated societal costs for the year preceding assessment were £3268.47 (€3758.54) per person. In multivariate analyses, no reliable baseline associates of cost were identified. Conclusion The economic burden of EDs characterised by regular binge eating is significant, and underscores the need for efficacious and cost-effective treatments. Individuals with binge-eating disorders report work impairment and healthcare use that may cost the United Kingdom economy upwards of £3.5 billion (€4bn) per annum. Further studies should consider academic impairment and the economic impact of EDs on families. Level of evidence III: evidence obtained from well-designed cohort or case–control analytic studies.

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Cost-of-illness for Non-underweight Eating Disorders

10.21203/rs.3.rs-543732/v1 ◽

2021 ◽

Author(s):

Paul Jenkins

Keyword(s):

Eating Disorders ◽

Binge Eating ◽

Economic Impact ◽

Indirect Costs ◽

Economic Cost ◽

Cost Effective ◽

Level Of Evidence ◽

Productivity Costs ◽

Academic Impairment ◽

The Uk

Abstract Purpose: This study examined economic costs associated with untreated eating disorders characterised by regular binge eating in the absence of low weight. Both direct and indirect costs were assessed, taking a partial societal perspective of economic impact.Methods: One-hundred and twenty-six adults seeking treatment for recurrent binge eating were asked to report impairment associated with an eating disorder. Total direct, indirect, and out-of-pocket costs were calculated using 2017 prices. Overall costs per individual were estimated, including an examination of variables correlated with costs.Results: Estimated costs for the year preceding assessment were £2,428.59 (€2,792.88) per person. Frequency of binge eating was associated with several elements of cost, and BMI was associated with productivity costs.Conclusion: The economic cost of eating disorders characterised by regular binge eating is significant, and underscores the need for efficacious and cost-effective treatments. Individuals with eating disorders report work impairment and healthcare usage that may cost the UK economy upwards of £2.6–3.5 billion (€3–4bn). Further studies should consider academic impairment and the economic impact on families.Level of evidenceLevel III: Evidence obtained from well-designed cohort or case-control analytic studies

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The GABAergic control of the hungry. Topiramate in the treatment of obesity without binge eating disorders (BED). Preliminary data

Experimental and Clinical Endocrinology & Diabetes ◽

10.1055/s-2003-817591 ◽

2003 ◽

Vol 111 (06) ◽

Author(s):

G Messina ◽

R Proietti ◽

JV Faroni

Keyword(s):

Eating Disorders ◽

Binge Eating ◽

Preliminary Data ◽

Binge Eating Disorders ◽

Treatment Of Obesity

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Dietary patterns of patients with binge eating disorders with and without night eating

Eating and Weight Disorders - Studies on Anorexia Bulimia and Obesity ◽

10.1007/s40519-018-0590-2 ◽

2018 ◽

Vol 25 (2) ◽

pp. 321-328

Author(s):

Yael Latzer ◽

Adi Elron Yutal ◽

Miri Givon ◽

Orna Kabakov ◽

Sigal Alon ◽

...

Keyword(s):

Eating Disorders ◽

Binge Eating ◽

Dietary Patterns ◽

Night Eating ◽

Binge Eating Disorders

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¿Se relaciona el trastorno por atracón con la adherencia a la dieta mediterránea en estudiantes universitarios?

Nutrición Hospitalaria ◽

10.20960/nh.799 ◽

2016 ◽

Vol 33 (6) ◽

Author(s):

Ana Zaragoza-Martí ◽

Miriam Sánchez-SanSegundo ◽

Rosario Ferrer-Cascales

Keyword(s):

Eating Disorders ◽

Binge Eating ◽

Odds Ratios ◽

Binge Eating Disorders ◽

Dieta Mediterranea ◽

Dieta Mediterránea ◽

Estudiantes Universitarios

Introducción: el trastorno por atracón o “Binge Eating Disorders” (BED) se caracteriza por el consumo de grandes cantidades de comida en un breve periodo de tiempo acompañado de una sensación de falta de control sobre la ingesta. El BED se relaciona con una alimentación poco equilibrada, contribuyendo a la aparición de diversas patologías. La alimentación y los estilos de vida desempeñan un factor clave en la prevención de estas patologías, siendo la dieta mediterránea (DM) uno de los estándares de la alimentación saludable.Objetivo: analizar la relación entre la DM y el BED en una muestra de estudiantes universitarios.Método: se realizó un estudio descriptivo transversal en una muestra de 120 estudiantes de la Universidad de Alicante. Para establecer la relación entre el patrón de atracón y el grado de adherencia a la DM se determinó el coefi ciente de correlación r de Pearson. Los resultados se expresaron en odds ratios (OR) e intervalo de confi anza (IC) al 95%.Resultados: los sujetos con un patrón de BED presentaron menor adherencia a la DM (OR = 4,03; IC 95%, 1,80-18,69), mayores índices de exceso de peso (OR = 5,31; IC 95%, 1,81-15,64), y vidas más sedentarias (OR = 1,4; IC 95%, 0,52-4,12). El BED correlacionó negativamente con el grado de adherencia DM (r = -0,3, p < 0,01).Conclusión: el patrón de DM se asocia de manera inversa con el riesgo de sufrir trastorno por atracón y además dicho patrón de atracón se asocia significativamente con el riesgo de sufrir exceso de peso en estudiantes universitarios. Son necesarios estudios que confi rmen que la DM reduce el riesgo de sufrir trastorno por atracón.

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Characterizing cerebral metabolite profiles in anorexia and bulimia nervosa and their associations with habitual behavior

10.1101/2021.09.12.21263466 ◽

2021 ◽

Author(s):

Margaret L. Westwater ◽

Alexander G. Murley ◽

Kelly M.J. Diederen ◽

T. Adrian Carpenter ◽

Hisham Ziauddeen ◽

...

Keyword(s):

Eating Disorders ◽

Bulimia Nervosa ◽

Binge Eating ◽

Instrumental Learning ◽

Learning Task ◽

Resonance Spectroscopy ◽

Binge Eating Disorders ◽

Habitual Behavior ◽

Neurochemical Changes ◽

The Right

AbstractBackgroundAnorexia nervosa (AN) and bulimia nervosa (BN) are associated with altered brain structure and function, as well as increased habitual behavior. This neurobehavioral profile may implicate neurochemical changes in the pathogenesis of these illnesses. Altered glutamate, myo-inositol and N-acetyl aspartate (NAA) concentrations are reported in restrictive AN, yet whether these extend to binge-eating disorders, or relate to habitual traits in affected individuals, remains unknown.MethodsUsing single-voxel proton magnetic resonance spectroscopy, we measured glutamate, myo-inositol and NAA in 85 women [n=22 AN (binge-eating/purging subtype; AN-BP), n=33 BN, n=30 controls]. Spectra were acquired from the right inferior lateral prefrontal cortex and the right occipital cortex. To index habitual behavior, participants performed an instrumental learning task and completed the Creature of Habit Scale. Exploratory analyses examined associations between metabolites and habitual behavior.ResultsWomen with AN-BP, but not BN, had reduced myo-inositol and NAA concentrations relative to controls in both voxels. Patient groups had intact performance on the instrumental learning task; however, both groups reported increased routine behaviors compared to controls. Women with BN also reported greater automatic behaviors, and automaticity was related to reduced prefrontal glutamate and NAA in the AN-BP group.DiscussionFindings extend previous reports of reduced myo-inositol and NAA levels in AN to AN-BP, which may reflect disrupted axonal-glial signaling. Although we found inconsistent support for increased habitual behavior in AN-BP and BN, we identified preliminary associations between prefrontal metabolites and automaticity in AN-BP. These results provide further evidence of unique neurobiological profiles across binge-eating disorders.

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