Protective effect of white tea extract against acute oxidative injury caused by adriamycin in different tissues

2012 ◽  
Vol 134 (4) ◽  
pp. 1780-1785 ◽  
Author(s):  
Cristóbal Espinosa ◽  
José Ángel López-Jiménez ◽  
Lorena Cabrera ◽  
Elvira Larqué ◽  
María Pilar Almajano ◽  
...  
2016 ◽  
Vol 83 ◽  
pp. 1422-1427 ◽  
Author(s):  
Débora Delwing-Dal Magro ◽  
Roberto Roecker ◽  
Gustavo M. Junges ◽  
André F. Rodrigues ◽  
Daniela Delwing-de Lima ◽  
...  

2012 ◽  
Vol 32 (4) ◽  
pp. 733-741 ◽  
Author(s):  
Lílian Gonçalves Teixeira ◽  
Priscilla Ceci Lages ◽  
Tatianna Lemos Jascolka ◽  
Edenil Costa Aguilar ◽  
Fabíola Lacerda Pires Soares ◽  
...  

White tea is an unfermented tea made from young shoots of Camellia sinensis protected from sunlight to avoid polyphenol degradation. Although its levels of catechins are higher than those of green tea (derived from the same plant), there are no studies addressing the relationship between this tea and obesity associated with oxidative stress.The objective of this study was to evaluate the effect of white tea on obesity and its complications using a diet induced obesity model. Forty male C57BL/6 mice were fed a high-fat diet to induce obesity (Obese group) or the same diet supplemented with 0.5% white tea extract (Obese + WTE) for 8 weeks. Adipose tissue, serum lipid profile, and oxidative stress were studied. White tea supplementation was not able to reduce food intake, body weight, or visceral adiposity. Similarly, there were no changes in cholesterol rich lipoprotein profile between the groups. A reduction in blood triacylglycerols associated with increased cecal lipids was observed in the group fed the diet supplemented with white tea. White tea supplementation also reduced oxidative stress in liver and adipose tissue. In conclusion, white tea extract supplementation (0.5%) does not influence body weight or adiposity in obese mice. Its benefits are restricted to the reduction in oxidative stress associated with obesity and improvement of hypertriacylglycerolemia.


2018 ◽  
Vol 32 (12) ◽  
pp. 2480-2486 ◽  
Author(s):  
Yuan Li ◽  
Suming Fu ◽  
Enlai Li ◽  
Xianchao Sun ◽  
Haie Xu ◽  
...  

Author(s):  
Elham Ahmadi ◽  
Amir Hossein Elhamirad ◽  
Nasrin Mollania ◽  
Mohammad Reza Saeidi Asl ◽  
Ahmad Pedramnia
Keyword(s):  

2015 ◽  
Vol 63 (7) ◽  
pp. 2026-2032 ◽  
Author(s):  
Vanna Sanna ◽  
Giuseppe Lubinu ◽  
Pierluigi Madau ◽  
Nicolino Pala ◽  
Salvatore Nurra ◽  
...  

RSC Advances ◽  
2020 ◽  
Vol 10 (17) ◽  
pp. 9924-9933 ◽  
Author(s):  
Xiaoyu Peng ◽  
Tingjun Hu ◽  
Yuxue Zhang ◽  
Anran Zhao ◽  
Bharathi Natarajan ◽  
...  

Synthesized caffeic acid derivatives exhibit protective effect on H2O2 induced oxidative injury in A549 cells via Nrf2 pathway.


1989 ◽  
Vol 44 (8) ◽  
pp. 955-958 ◽  
Author(s):  
George Dreyfus ◽  
Leopoldo de Meis

Mitochondrial F1 ATPase is inactivated by urea. Protection against urea inactivation is obtained when betaine, a methylamine found in different tissues, is added to the assay medium. Protection is also obtained upon the addition of either glycerol or dimethyl sulfoxide to the assay medium. The F, ATPase is rapidly inactivated at 4 °C. Inactivation by low temperature is prevented by betaine, glycerol and dimethyl sulfoxide. The protective effect of organic solvents and betaine against cold inactivation is prevented by urea.


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