Arterial oxygen saturation during Cheyne-Stokes respiration in heart failure patients: does measurement site matter?

2019 ◽  
Vol 55 ◽  
pp. 6-13 ◽  
Author(s):  
Roberto Maestri ◽  
Elena Robbi ◽  
Marta Lovagnini ◽  
Claudio Bruschi ◽  
Maria Teresa La Rovere ◽  
...  
2011 ◽  
Vol 2 (1) ◽  
pp. 81-86
Author(s):  
Ol'ga Vladimirovna Polyakova ◽  
Grigoriy Guramovich Arabidze

Summary. In the first part of this review both domestic and foreign data from clinical and experimental researches relating to modern views on the definition, classification, clinical and morphological picture, as well as approaches to the treatment of chronic pulmonary heart (CPH) are presented. The CPH classification in stages and functional classes depending on the level of pulmonary failure, arterial oxygen saturation, right ventricular hypertrophy and heart failure are given. Modern methods of diagnosing CPH are introduced as well.


2021 ◽  
Vol 16 ◽  
Author(s):  
Alaa Thabet Hassan ◽  
Soher Mostafa Ahmed ◽  
Azza Salah AbdelHaffeez ◽  
Sherif A.A. Mohamed

Background: Despite its wide use in clinical practice, few studies had assessed the role of pulse oximetry in patients with heart failure. We aimed to evaluate the accuracy and precision of the pulse oximeter in patients with heart failure and to determine this accuracy at three different sensor locations.Methods: Comparison of pulse oximetry reading (SpO2) with arterial oxygen saturation (SaO2) was reported in 3 groups of patients with heart failure (HF); those with ejection fraction (EF) >40%, those with EF <40%, and those with acute HF (AHF) with ST and non-ST segment elevation acute myocardial infarction (STEMI and non-STEMI).Results: A total of 235 patients and 90 control subjects were enrolled. There were significant differences in O2 saturation between control and patients’ groups when O2 saturation is measured at the finger and toe, but not the ear probes; p=0.029, p=0.049, and 0.051, respectively. In HF with EF>40% and AHF with O2 saturations >90%, finger oximetry is the most accurate and reliable, while in HF with EF<40% and in patients with AHF with O2 saturations <90%, ear oximetry is the most accurate.Conclusion: Pulse oximetry is a reliable tool in assessing oxygen saturation in patients with heart failure of different severity. In HF with EF>40% and in AHF with O2 saturations >90%, finger oximetry is the most accurate and reliable, while in HF with EF<40% and in patients with AHF with O2 saturations <90%, ear oximetry is the most accurate. Further studies are warranted.


1996 ◽  
Vol 91 (s1) ◽  
pp. 72-74 ◽  
Author(s):  
Andrea Mortara ◽  
Luciano Bernardi ◽  
GianDomenico Pinna ◽  
Giammario Spadacini ◽  
Roberto Maestri ◽  
...  

1963 ◽  
Vol 205 (5) ◽  
pp. 963-970 ◽  
Author(s):  
Cecil E. Cross ◽  
P. Andre Rieben ◽  
Charles I. Barron ◽  
Peter F. Salisbury

An integrative series of investigations progressed from simplified systems, where cause-and-effect relations were relatively clear, to the virtually intact animal with separately perfused carotid arteries. Arterial pO2 below 40 mm Hg (about 75% oxygen saturation) caused edema of the heart muscle; however, the contractile strength and performance of isolated hearts were compromised severely only when the arterial pO2 had fallen below 15 mm Hg (about 25% saturation). The ‘acute circulatory crisis" which is known to occur when the arterial oxygen saturation falls below 80% was not caused by weakness of the heart muscle but by reflexes from the carotid artery territory: even when the oxygen saturation of systemic arterial blood had fallen as low as 50% this did not cause heart failure as long as the carotids were perfused with blood of normal pO2. Severe heart failure occurred when the blood in the carotid arteries was moderately hypoxic (pO2 below 50, sat. below 80%) while the rest of the circulation was fully oxygenated.


1994 ◽  
Vol 73 (2) ◽  
pp. 180-185 ◽  
Author(s):  
Mark A. Munger ◽  
Eric J. Stanek ◽  
Andrew R. Nara ◽  
Kingman P. Strohl ◽  
Michael J. Decker ◽  
...  

2021 ◽  
Vol 11 (1) ◽  
Author(s):  
Shinshu Katayama ◽  
Jun Shima ◽  
Ken Tonai ◽  
Kansuke Koyama ◽  
Shin Nunomiya

AbstractRecently, maintaining a certain oxygen saturation measured by pulse oximetry (SpO2) range in mechanically ventilated patients was recommended; attaching the INTELLiVENT-ASV to ventilators might be beneficial. We evaluated the SpO2 measurement accuracy of a Nihon Kohden and a Masimo monitor compared to actual arterial oxygen saturation (SaO2). SpO2 was simultaneously measured by a Nihon Kohden and Masimo monitor in patients consecutively admitted to a general intensive care unit and mechanically ventilated. Bland–Altman plots were used to compare measured SpO2 with actual SaO2. One hundred mechanically ventilated patients and 1497 arterial blood gas results were reviewed. Mean SaO2 values, Nihon Kohden SpO2 measurements, and Masimo SpO2 measurements were 95.7%, 96.4%, and 96.9%, respectively. The Nihon Kohden SpO2 measurements were less biased than Masimo measurements; their precision was not significantly different. Nihon Kohden and Masimo SpO2 measurements were not significantly different in the “SaO2 < 94%” group (P = 0.083). In the “94% ≤ SaO2 < 98%” and “SaO2 ≥ 98%” groups, there were significant differences between the Nihon Kohden and Masimo SpO2 measurements (P < 0.0001; P = 0.006; respectively). Therefore, when using automatically controlling oxygenation with INTELLiVENT-ASV in mechanically ventilated patients, the Nihon Kohden SpO2 sensor is preferable.Trial registration UMIN000027671. Registered 7 June 2017.


1944 ◽  
Vol 79 (1) ◽  
pp. 9-22 ◽  
Author(s):  
Frank L. Engel ◽  
Helen C. Harrison ◽  
C. N. H. Long

1. In a series of rats subjected to hemorrhage and shock a high negative correlation was found between the portal and peripheral venous oxygen saturations and the arterial blood pressure on the one hand, and the blood amino nitrogen levels on the other, and a high positive correlation between the portal and the peripheral oxygen saturations and between each of these and the blood pressure. 2. In five cats subjected to hemorrhage and shock the rise in plasma amino nitrogen and the fall in peripheral and portal venous oxygen saturations were confirmed. Further it was shown that the hepatic vein oxygen saturation falls early in shock while the arterial oxygen saturation showed no alteration except terminally, when it may fall also. 3. Ligation of the hepatic artery in rats did not affect the liver's ability to deaminate amino acids. Hemorrhage in a series of hepatic artery ligated rats did not produce any greater rise in the blood amino nitrogen than a similar hemorrhage in normal rats. The hepatic artery probably cannot compensate to any degree for the decrease in portal blood flow in shock. 4. An operation was devised whereby the viscera and portal circulation of the rat were eliminated and the liver maintained only on its arterial circulation. The ability of such a liver to metabolize amino acids was found to be less than either the normal or the hepatic artery ligated liver and to have very little reserve. 5. On complete occlusion of the circulation to the rat liver this organ was found to resist anoxia up to 45 minutes. With further anoxia irreversible damage to this organ's ability to handle amino acids occurred. 6. It is concluded that the blood amino nitrogen rise during shock results from an increased breakdown of protein in the peripheral tissues, the products of which accumulate either because they do not circulate through the liver at a sufficiently rapid rate or because with continued anoxia intrinsic damage may occur to the hepatic parenchyma so that it cannot dispose of amino acids.


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