Nannizzia incurvata. [Descriptions of Fungi and Bacteria].

1965 ◽  
Author(s):  
P. M. Stockdale
Keyword(s):  
Great Britain ◽  
Geographical Distribution ◽  
Guinea Pigs ◽  
Tinea Capitis ◽  
World Wide ◽  
Skin Lesions ◽  
Wide Distribution ◽  
Tinea Corporis ◽  
Soil P ◽  
Infected Skin

Abstract A description is provided for Nannizzia incurvata. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Recorded only from man and dog (but see NOTES). Guinea-pigs have been experimentally infected. DISEASE: Ringworm (dermatophytosis, tinea). Nannizzia incurvata is present in soil and apparently only rarely a cause of disease. In man the scalp (tinea capitis) and glabrous skin (tinea corporis) may be infected. Skin lesions are inflammatory but details of known scalp infections are not available. In experimental inoculations of guineapigs (Rdzanek, pers. comm.) N. incurvata was intermediate between N. gypsea and N. fulva in virulence, the reaction varying from negative to strongly inflammatory. Ectothrix hyphae breaking up into large arthrospores were seen on some hairs, and infected hairs did not fluoresce under Wood's light. GEOGRAPHICAL DISTRIBUTION: Asia (India), Europe (Czechoslovakia, Great Britain and Ireland, Germany, Netherlands, Poland); U.S.A. (Tenn.); N. incurvata is probably of world-wide distribution in the soil.

Nannizzia gypsea. [Descriptions of Fungi and Bacteria].

1965 ◽  
Author(s):  
P. M. Stockdale
Keyword(s):  
Great Britain ◽  
Geographical Distribution ◽  
Tinea Capitis ◽  
New South ◽  
Skin Lesions ◽  
Wide Distribution ◽  
Soil P ◽  
Cook Island ◽  
South Wales ◽  
Infected Skin

Abstract A description is provided for Nannizzia gypsea. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Recorded only from man (but see NOTES). Guinea-pigs have been experimentally infected. DISEASE: Ringworm (dermatophytosis, tinea). Nannizzia gypsea is apparently widespread in soil and only rarely pathogenic. The scalp (tinea capitis) and glabrous skin (tinea corporis) may be infected. Skin lesions are inflammatory, but details of only two scalp infections are known. In both the lesions were inflammatory; in one favus-like crusts were also found and in the other hair invasion was of the large-spored ectothrix type. In experimental inoculations of guineapigs (Rdzanek, pers. comm.) N. gypsea was slightly more virulent than N. incurvata and N. fulva, the reaction varying from negative to strongly inflammatory, a few ectothrix hyphae breaking up into arthrospores were seen on some hairs, and infected hairs did not fluoresce under Wood's light. GEOGRAPHICAL DISTRIBUTION: Asia (Japan), Australasia and Oceania (Australia (New South Wales), New Zealand, Raratonga, Cook Island), Europe (France, Great Britain, Italy); North America (Tenn.). Probably of world-wide distribution in the soil.

Nannizzia fulva. [Descriptions of Fungi and Bacteria].

1965 ◽  
Author(s):  
P. M. Stockdale
Keyword(s):  
Great Britain ◽  
South America ◽  
Geographical Distribution ◽  
Inflammatory Reaction ◽  
Tinea Capitis ◽  
World Wide ◽  
Skin Lesions ◽  
Limiting Factors ◽  
Proved Case ◽  
Infected Skin

Abstract A description is provided for Nannizzia fulva. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Recorded only from man (but see NOTES). Guinea-pigs have been experimentally infected. DISEASE: Ringworm (dermatophytosis, tinea). Nannizzia fulva is present in the soil and apparently only rarely pathogenic. The scalp (tinea capitis) and glabrous skin (tinea corporis) may be infected. Skin lesions are inflammatory. The only proved case of scalp infection is that recorded by Uriburu (1909); according to Sabouraud (Les Teignes, 1910, p. 241) in this case there was a light inflammatory reaction and infected hairs were indistinguishable from those in M. audouinii infections. In experimental inoculations of guineapigs (Rdzanek, pers. comm.) N. fulva was slightly less virulent than N. gypsea and N. incurvata, the reaction varying from negative to strongly inflammatory. A few ectothrix hyphae breaking up into arthrospores were seen on some hairs, and infected hairs did not fluoresce under Wood's light. GEOGRAPHICAL DISTRIBUTION: Africa (Tanzania), Europe (Great Britain, Hungary, Yugoslavia), South America (Argentina). Probably world-wide in distribution in the soil, but there may be local limiting factors (e.g., in Great Britain it has been recorded only for Bristol, Somerset, and Wiltshire).

The Output of First Stage Larvae by Cats Infested with Aelurostrongylus abstrusus

1968 ◽  
Vol 42 (3-4) ◽  
pp. 295-298 ◽  
Author(s):  
J. M. Hamilton ◽  
A. W. McCaw
Keyword(s):  
Life Cycle ◽  
Great Britain ◽  
Intermediate Host ◽  
World Wide ◽  
Wide Distribution ◽  
Clinical Disease ◽  
Complex Life Cycle ◽  
Patent Period ◽  
Aelurostrongylus Abstrusus

Aelurostrongylus abstrusus, the lungworm of the cat, has a world wide distribution and has been reported from countries as far apart as America, Great Britain and Palestine. It has a complex life cycle insofar as a molluscan intermediate host is essential and it is possible that auxiliary hosts also play an important part. In Britain, the incidence of active infestation of cats with the parasite has been recorded as 19·4% (Lewis, 1927) and 6·6% (Hamilton, 1966) but the latter author found that, generally, the clinical disease produced by the parasite was of a mild nature. It is known that the average patent period of the infestation in the cat is 8–13 weeks and it seems likely that, in that time, a considerable number of first stage larvae would be evacuated. Information on that point is not available and the object of the following experiment was to ascertain the number of larvae produced by cats during the course of a typical infestation.

Nannizzia obtusa. [Descriptions of Fungi and Bacteria].

1965 ◽  
Author(s):  
P. M. Stockdale
Keyword(s):  
New Zealand ◽  
North America ◽  
Central America ◽  
Geographical Distribution ◽  
West Indies ◽  
Tinea Capitis ◽  
World Wide ◽  
Whole Body ◽  
Scalp Lesions ◽  
Light Green

Abstract A description is provided for Nannizzia obtusa. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Primarily a pathogen of the pig, occasionally transmitted to man. Guinea-pigs have been experimentally infected. DISEASE: Ringworm (dermatophytosis, tinea). In pigs lesions are circular and circumscribed to irregular in outline, sometimes involving almost the whole body. They are reddish in colour and are frequently covered with brownish crusts. Alopecia and pruritis do not occur (RMVM 5, 226, 491-492). In man the scalp (tinea capitis) and glabrous skin (tinea corporis) may be infected. Scalp lesions reported have shown a kerion type of reaction with endothrix hair invasion; in some cases infected hairs have fluoresced light green under Wood's light. GEOGRAPHICAL DISTRIBUTION: Reported from Africa (Kenya), Australasia (Australia, New Zealand); North America (Canada, U.S.A. (Ind., Kans., Ky, Miss., N.J., Pa)), Central America and West Indies (Mexico, Cuba). Possibly world-wide in distribution.

scholarly journals Molecular typing of Salmonella serotype Thompson strains isolated from human and animal sources

1999 ◽  
Vol 122 (1) ◽  
pp. 33-39 ◽  
Author(s):  
S. A. CHISHOLM ◽  
P. B. CRICHTON ◽  
H. I. KNIGHT ◽  
D. C. OLD
Keyword(s):  
Geographical Distribution ◽  
Molecular Typing ◽  
World Wide ◽  
Wide Distribution ◽  
Human Infection ◽  
Major Clone ◽  
Salmonella Serotype

One-hundred-and-thirteen isolates of Salmonella serotype Thompson from diverse sources in seven countries were characterized by PvuII ribotyping and IS200 fingerprinting. Ten PvuII ribotypes were observed. The predominant PvuII ribotype 1 represented a major clone of world-wide distribution but was not found in Australia; PvuII ribotypes 2 and 3 represented minor clones. HincII ribotyping discriminated subtypes within PvuII ribotype 1: HincII ribotype 1 was distributed widely but HincII ribotype 2 was found mainly in Scottish isolates. None of 101 isolates of PvuII ribotypes 1–3 contained copies of IS200. All 12 isolates of PvuII ribotypes 4–10 were from Australia and 7 of them contained copies of IS200 of 5 different profiles. These results suggest the existence of at least two lineages of Salmonella Thompson with a different geographical distribution. The finding that most isolates from man and poultry in Scotland belonged to the same ribotype (PvuII 1/HincII 2) and were IS200-negative suggests that poultry is an important source of human infection in Scotland.

ON THE NEMATODE GENUS RHABDOCHONA RAILLIET, 1916 (NEMATODA: SPIRUROIDEA)

1951 ◽  
Vol 29 (1) ◽  
pp. 1-16 ◽  
Author(s):  
L. P. E. Choquette
Keyword(s):  
New Species ◽  
North America ◽  
Geographical Distribution ◽  
Fish Species ◽  
World Wide ◽  
Wide Distribution ◽  
Systematic Position ◽  
Nematode Genus ◽  
A New Species ◽  
Host List

The systematic position of the genus Rhabdochona Railliet, 1916 and its diagnosis is reviewed. The genus comprising 33 species has a world-wide distribution and has been found, with one exception, in fish. Species from fish in North America are discussed and R. cascadilla Wigdor, 1918 is redescribed; R. laurentiana Lyster, 1940 is considered to be identical with the latter. A new species, R. milleri, is described. A host list and the geographical distribution of the 33 species of Rhabdochona so far recorded are given.

Microsporum audouinii. [Descriptions of Fungi and Bacteria].

1965 ◽  
Author(s):  
P. M. Stockdale
Keyword(s):  
Geographical Distribution ◽  
Tinea Capitis ◽  
Skin Lesions ◽  
Glabrous Skin ◽  
French West Africa ◽  
South Wales ◽  
Microsporum Audouinii ◽  
Bright Green ◽  
Scalp Lesions

Abstract A description is provided for Microsporum audouinii. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Primarily a pathogen of man. Also recorded on the dog, gibbon, guineapig and monkey. Most strains are not pathogenic for experimental animals. DISEASE: Ringworm (dermatophytosis, tinea). Infected hairs usually fluoresce bright green under Wood's light and are surrounded by an ectothrix sheath of small spores in a mosaic arrangement. In man, the scalp (tinea capitis) and less frequently the glabrous skin (tinea corporis) may be infected. Children are most susceptible, and in adults infection is usually confined to the glabrous skin, although a few scalp infections have been reported. Rothman et al. (RMVM 1, 614) noted that fatty acids from adult hair fat inhibited M. audouinii in vitro and suggested that this was the reason for lower incidence of infection in adults, but Kligman & Grinsberg (RMVM 1, 1800) found that adult sebum was not more fungistatic than that of children. Scalp lesions are usually circular, scaling areas of alopecia and, in contrast to M. cants infections, there is generally very little inflammatory reaction. Skin lesions are usually circulate scaling areas with clearing centres. Kligman (RMVM 1, 2510; 2, 2484) studied the pathogenesis of tinea capitis due to M. audouinii. GEOGRAPHICAL DISTRIBUTION: Africa (Algeria, Chad Republic, Cameroons, Congo, Egypt, French West Africa, Guinea Republic, Madagascar, Morocco, Mozambique, Nigeria, South Africa, Tunisia); Asia (India, Java, Palestine, Persia, Turkey), Australasia & Oceania (Australia (New South Wales, Western Australia), New Zealand), Europe (Austria, Belgium, Czechoslovakia, Denmark, France, Germany, Great Britain and Ireland, Hungary, Italy, Netherlands, Poland, Portugal, Romania, Spain, Sweden, Switzerland, U.S.S.R., Yugoslavia), North America (Canada, U.S.A.), Central America and West Indies (Cuba, Jamaica, Puerto Rico); South America (Argentina, Brazil, Chile, Ecuador, Venezuela).

Trichophyton violaceum. [Descriptions of Fungi and Bacteria].

1980 ◽  
Author(s):  
P. M. Stockdale
Keyword(s):  
Middle East ◽  
Lymph Nodes ◽  
Geographical Distribution ◽  
Tinea Capitis ◽  
Tinea Corporis ◽  
Tinea Unguium ◽  
Inflammatory Lesions ◽  
Trichophyton Violaceum ◽  
Tinea Barbae ◽  
Subcutaneous Tissues

Abstract A description is provided for Trichophyton violaceum. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Primarily a pathogen of man (causing mycoses). Also recorded from buffalo, cat, cattle, dog and mouse (RMVM 6, 3187; 10, 505). The cat, dog, guineapig, monkey and mouse have been infected experimentally. The horse, fowl and pigeon have been implicated as hosts of T. violaceum. DISEASE: Ringworm (dermatophytosis, tinea). The scalp (tinea capitis), beard (tinea barbae), body (tinea corporis) and nails (tinea unguium, onychomycosis) may be infected. Infected hairs show an endothrix type of invasion and do not fluoresce under Wood's light. The hairs may break and curl producing 'black dots' on the scalp (also sometimes produced by other endothrix Trichophyton species). Inflammatory lesions (occasionally with the development of kerion), scarring and alopecia and, occasionally, favus-like lesions may occur. Infections of the subcutaneous tissues, lymph nodes, brain, bones and other internal tissues have been reported (RMVM 3, 1441; 4, 716; 9, 677; 11, 708; 12, 1847; 13, 961, 1572). GEOGRAPHICAL DISTRIBUTION: Worldwide. A dominant cause of scalp ringworm in S. and E. Europe, the Middle East, N. Africa, parts of Central and E. Africa, S. Africa and Asia (RMVM 10, 602; 13, 1558).

Microsporum ferrugineum. [Descriptions of Fungi and Bacteria].

1965 ◽  
Author(s):  
P. M. Stockdale
Keyword(s):  
Geographical Distribution ◽  
Tinea Capitis ◽  
Skin Lesions ◽  
Cape Verde ◽  
French West Africa ◽  
Experimental Animals ◽  
Microsporum Ferrugineum ◽  
The Face ◽  
Infection Disease ◽  
Scalp Lesions

Abstract A description is provided for Microsporum ferrugineum. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Recorded only from man. Experimental animals are not susceptible to infection. DISEASE: Ringworm (dermatophytosis, tinea). Primarily affecting the scalp (tinea capitis) and less frequently the glabrous skin, particularly of the face (tinea corporis) of children. Scalp lesions are usually non-inflammatory, scaling patches of alopecia. Infected hairs are surrounded by sheaths of small spores in a mosaic arrangement, and usually fluoresce under Wood's light, but Germeraad & Klokke (E. Afr. med. J. 39: 515-519, 1962) saw no distinct fluorescence in 22 cases in Uganda. Skin lesions are usually scaly, with little or no inflammation. GEOGRAPHICAL DISTRIBUTION: Africa (Angola, Cameroons, Cape Verde Islands, Congo, French West Africa, Mozambique, Nigeria, Ruanda-Burundi, Uganda); Asia (China, Formosa, Hawaii, India, Indo-China, Israel, Japan, U.S.S.R.); Europe (Bulgaria, Czechoslovakia, Hungary, France, Italy, Poland, Romania, U.S.S.R., Yugoslavia (Macedonia)); North America (? Texas); South America (? Brazil,? Uruguay).

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