scholarly journals Cytotoxicity, oxidative stress, and inflammatory response of smokeless tobacco extracts and cytotoxicity of combustible cigarette whole smoke in a 3D oral organotypic buccal cell model

2021 ◽  
pp. 1-10
Author(s):  
Brian M. Keyser
Keyword(s):  
Oxidative Stress ◽  
Inflammatory Response ◽  
Smokeless Tobacco ◽  
Cell Model ◽  
Buccal Cell ◽  
Whole Smoke

Toxicological Impact of Air Pollution Particulate Matter (PM2.5) Collected under Urban, Industrial or Rural Influence: Occurrence of Oxidative Stress and Inflammatory Reaction in BEAS-2B Human Bronchial Epithelial Cells (Corrected Version)

2011 ◽  
Vol 324 ◽  
pp. 489-492 ◽  
Author(s):  
Mona Dergham ◽  
Sylvain Billet ◽  
Anthony Verdin ◽  
Dominique Courcot ◽  
Fabrice Cazier ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Air Pollution ◽  
Particulate Matter ◽  
Inflammatory Response ◽  
Cell Model ◽  
Chemical Characterization ◽  
Vascular Diseases ◽  
Bronchial Epithelial Cell ◽  
Bronchial Epithelial ◽  
Physico Chemical

Exposure to air pollution Particulate Matter (PM) is one of the risk factors involved in the high incidence of respiratory and cardio-vascular diseases. In this work, to integrate inter-seasonal and inter-site variations, fine particle (PM2.5) samples have been collected in spring-summer 2008) and autumn 2008-winter 2009, in Dunkerque (France) under urban or industrial influence, and in Rubrouck (France), under rural influence. Attention was paid to characterize their physico-chemical characteristics, and to determine their ability to induce oxidative stress and inflammatory response in a human bronchial epithelial cell model (BEAS-2B cell line). Physico-chemical characterization of the six PM samples showed their heterogeneities and complexities depending upon their respective natural and/or anthropogenic emission sources. Lung cytotoxicity of these air pollution PM2.5samples, as shown in BEAS-2B cells, might rely on the induction of oxidative stress conditions and particularly on the excessive inflammatory response.

Naringin Inhibits TNF-α Induced Oxidative Stress and Inflammatory Response in HUVECs via Nox4/NF-κ B and PI3K/Akt Pathways

2014 ◽  
Vol 15 (12) ◽  
pp. 1173-1182 ◽  
Author(s):  
Wenshuang Li ◽  
Changyuan Wang ◽  
Jinyong Peng ◽  
Jing Liang ◽  
Yue Jin ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Inflammatory Response

scholarly journals E-cigarette fluids and aerosol residues cause oxidative stress and an inflammatory response in human keratinocytes and 3D skin models

2021 ◽  
pp. 105234
Author(s):  
Careen Khachatoorian ◽  
Wentai Luo ◽  
Kevin J. McWhirter ◽  
James F. Pankow ◽  
Prue Talbot
Keyword(s):  
Oxidative Stress ◽  
Inflammatory Response ◽  
Human Keratinocytes ◽  
3D Skin

Oxidative stress and inflammatory response to printer toner particles in human epithelial A549 lung cells

2013 ◽  
Vol 216 (2-3) ◽  
pp. 171-180 ◽  
Author(s):  
Mathias Könczöl ◽  
Adilka Weiß ◽  
Richard Gminski ◽  
Irmgard Merfort ◽  
Volker Mersch-Sundermann
Keyword(s):  
Oxidative Stress ◽  
Inflammatory Response ◽  
Lung Cells ◽  
A549 Lung

scholarly journals Why so little effort to study anti-oxidant therapy in burns?

2016 ◽  
Vol 4 ◽  
pp. 1-2 ◽  
Author(s):  
Gordon L. Klein
Keyword(s):  
Oxidative Stress ◽  
Stress Response ◽  
Inflammatory Response ◽  
Burn Injury ◽  
Burn Treatment ◽  
Gamma Tocopherol ◽  
Limited Success ◽  
Anti Oxidant

Abstract Given that oxidative stress is an inherent response to burn injury, it is puzzling as to why investigation into anti-oxidant therapy as an adjunct to burn treatment has been limited. Both the inflammatory response and the stress response to burn injury involve oxidative stress, and there has been some limited success in studies using gamma tocopherol and selenium to improve certain consequences of burns. Much remains to be done to investigate the number, doses and combinations of anti-oxidants, their efficacy, and limitations in improving defined outcomes after burn injury.

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