scholarly journals Morphophysiological changes in the intact surface of rat skin under the application of silicon and gold nanoparticles

2019 ◽  
Vol 1399 ◽  
pp. 022039
Author(s):  
E I Kondratenko ◽  
N A Lomteva ◽  
V Yu Timoshenko ◽  
A F Alykova ◽  
S K Kasimova ◽  
...  
2008 ◽  
Vol 65 (1) ◽  
pp. 1-10 ◽  
Author(s):  
Ganeshchandra Sonavane ◽  
Keishiro Tomoda ◽  
Akira Sano ◽  
Hiroyuki Ohshima ◽  
Hiroshi Terada ◽  
...  

2020 ◽  
Vol 2020 (1) ◽  
pp. 23-32
Author(s):  
S. P. Luhovskyi ◽  
◽  
M. M. Didenko ◽  
N. M Dmytrukha ◽  
L. V. Bakalo ◽  
...  

Author(s):  
M. John Hicks ◽  
Leon M. Silverstone ◽  
David G. Gantt ◽  
Catherine M. Flaitz

Although fluoride levels become elevated in sound enamel following a topical fluoride treatment, the caries-preventive effect of fluoride is thought to be due primarily to the role of fluoride in remineralization of clinically undetectable enamel lesions and hypomineralized enamel. During lesion formation, redistribution of fluoride from the enamel surface to the subsurface demineralized enamel occurs. This results in a surface zone with a relatively low fluoride content. In order to maintain an intact surface zone over a carious lesion, it may be necessary to replenish the fluoride levels with an exogenous fluoride source. By acid-etching the lesion surface, a more reactive surface is made available for fluoride interaction. In addition, porosities and etching patterns may be created, allowing for bonding of a caries-resistant resin material to the lesion surface. The purpose of this study was to determine the integrity of the caries-like lesion surface following acid-etching and subsequent stannous fluoride treatment (SnF2).


2013 ◽  
Vol 51 (01) ◽  
Author(s):  
N Fekete-Drimusz ◽  
J de la Roche ◽  
F Vondran ◽  
CL Sajti ◽  
MP Manns ◽  
...  

1973 ◽  
Vol 30 (02) ◽  
pp. 363-370
Author(s):  
D Thilo ◽  
E Böhm

SummaryExperiments with injury of the abdominal rat skin were carried out to examine the haemostatic system mechanism in vivo after zero to 30 seconds bleeding time. In the bleeding area only a few platelet aggregates could be found with no primary platelet thrombus. After 3.5 second bleeding time the first fibrin strands have been observed at the site of injury. The hypothesis is put forward that there is a very fast reacting haemostatic mechanism which results in the fibrin formation already at 3.5 seconds.


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