Structural Changes of Small Intestine Under Compression by Laparoscopic Stapler1

2016 ◽  
Vol 10 (3) ◽  
Author(s):  
Yu Zhou ◽  
Jingjing Xu ◽  
Boting Li ◽  
Binbin Ren ◽  
Yiyun Jin ◽  
...  
Keyword(s):  
Small Intestine ◽  
Structural Changes

STRUCTURAL CHANGES IN THE SMALL INTESTINE WALL AT THE MECHANICAL OBSTRUCTION OF THE DIGESTIVE CHANNEL IN THE EXPERIMENT

2019 ◽  
Vol 15 (67) ◽  
pp. 142
Author(s):  
I. Ya. Dzyubanovskyi ◽  
V. V. Benedykt ◽  
S. O. Nesteruk ◽  
K. S. Volkov
Keyword(s):  
Small Intestine ◽  
Structural Changes ◽  
Mechanical Obstruction

scholarly journals Functional and structural changes in the small intestine in children with hookworm infection.

1968 ◽  
Vol 43 (228) ◽  
pp. 235-238 ◽  
Author(s):  
D. K. Guha ◽  
B. N. Walia ◽  
B. N. Tandon ◽  
O. P. Ghai
Keyword(s):  
Small Intestine ◽  
Structural Changes ◽  
Hookworm Infection

scholarly journals Functional and structural changes of the human proximal small intestine after cytotoxic therapy.

1985 ◽  
Vol 38 (3) ◽  
pp. 265-270 ◽  
Author(s):  
D Cunningham ◽  
R J Morgan ◽  
P R Mills ◽  
L M Nelson ◽  
P G Toner ◽  
...  
Keyword(s):  
Small Intestine ◽  
Structural Changes ◽  
Cytotoxic Therapy ◽  
Proximal Small Intestine

The effect of litter origin upon the structure of the small intestine of piglets at weaning

2005 ◽  
Vol 2005 ◽  
pp. 95-95
Author(s):  
S. M. Carroll ◽  
H. M. Miller
Keyword(s):  
Small Intestine ◽  
Significant Influence ◽  
Null Hypothesis ◽  
Structural Changes ◽  
Sampling Strategies ◽  
Weaned Piglets ◽  
Villus Height ◽  
Weaned Piglet ◽  
Metabolite Concentrations ◽  
Experimental Sampling

The purpose of this study was to determine whether litter origin had an effect upon the structure of the small intestine in newly weaned piglets. The gut of the newly weaned piglet undergoes significant structural changes such as a decrease in villus height and an increase in crypt depth following commercial weaning. Litter origin has been shown to influence piglet performance immediately post weaning (Slade and Miller, 1999) and to have a significant influence upon blood metabolite concentrations in the newborn piglet (Ilsley and Miller, 2003). Litter origin therefore may also influence the structure of the small intestine in the piglet, which in turn would affect the experimental sampling strategies used in trials investigating effects on gut structure. This study was carried out to test the null hypothesis that litter origin would not have an effect upon the structure of the small intestine in the newly weaned piglet.

scholarly journals Influence of morning and evening melatonin administration on the mucosa and crypts of a small intestine in rats with obesity

2019 ◽  
Vol 78 (2) ◽  
pp. 50-53
Author(s):  
I. Vareniuk ◽  
N. Shevchuk ◽  
N. Roslova ◽  
M. Dzerzhynsky
Keyword(s):  
Small Intestine ◽  
Structural Changes ◽  
Goblet Cells ◽  
Functional Changes ◽  
Melatonin Administration ◽  
Calorie Diet ◽  
Mucosal Edema ◽  
Morning Administration ◽  
Evening Administration ◽  
High Calorie

The aim of this work was to determine structural and functional changes in a small intestine of rats after morning and evening administration of melatonin in obese animals during the spring-autumn photoperiod (12L:12D). The obesity was caused with a high-calorie diet for 6 weeks. After that, morning or evening melatonin administrations were given to normal and obese animals at a dose of 30 mg/kg for 7 weeks. After that, paraffin sections of the small intestine were made, on which a state of the mucosa, enterocytes and goblet cells in crypts was morphometrically and visually examined under a microscope. It has been shown, that obesity causes swelling and an increase of thickness of a mucosa, reduction of crypts, a decrease of activity of enterocytes and goblet cells of the small intestine. Introduction of melatonin to animals without obesity causes an increase in thickness of mucosa and a decrease in area of goblet cells. Additionally, after morning melatonin administration a depth of crypts and a height of enterocytes increases. Morning administration of melatonin to obese animals partially recovers crypts and their goblet cells, but doesn't prevent mucosal edema and worsens a state of enterocytes. The evening administration of melatonin partially normalizes all structural changes, caused by obesity. It was concluded, that melatonin may partially correct morpho-functional changes in the small intestine, caused by obesity in the spring and autumn seasons. The evening administration of melatonin to animals with obesity is more effective, than morning administrations. Also, the evening administration of melatonin causes fewer changes in the small intestine of animals without obesity, compared with morning administration.

scholarly journals Tumor Necrosis Factor Induces Developmental Stage-Dependent Structural Changes in the Immature Small Intestine

2014 ◽  
Vol 2014 ◽  
pp. 1-11 ◽  
Author(s):  
Kathryn S. Brown ◽  
Huiyu Gong ◽  
Mark R. Frey ◽  
Brock Pope ◽  
Matthew Golden ◽  
...  
Keyword(s):  
Small Intestine ◽  
Tumor Necrosis Factor ◽  
Premature Infants ◽  
Tumor Necrosis ◽  
Intestinal Tract ◽  
Structural Changes ◽  
Intestinal Inflammation ◽  
Inflammatory Diseases ◽  
Unique Challenge ◽  
Necrosis Factor

Background. Premature infants are commonly subject to intestinal inflammation. Since the human small intestine does not reach maturity until term gestation, premature infants have a unique challenge, as either acute or chronic inflammation may alter the normal development of the intestinal tract. Tumor necrosis factor (TNF) has been shown to acutely alter goblet cell numbers and villus length in adult mice. In this study we tested the effects of TNF on villus architecture and epithelial cells at different stages of development of the immature small intestine.Methods. To examine the effects of TNF-induced inflammation, we injected acute, brief, or chronic exposures of TNF in neonatal and juvenile mice.Results. TNF induced significant villus blunting through a TNF receptor-1 (TNFR1) mediated mechanism, leading to loss of villus area. This response to TNFR1 signaling was altered during intestinal development, despite constant TNFR1 protein expression. Acute TNF-mediated signaling also significantly decreased Paneth cells.Conclusions. Taken together, the morphologic changes caused by TNF provide insight as to the effects of inflammation on the developing intestinal tract. Additionally, they suggest a mechanism which, coupled with an immature immune system, may help to explain the unique susceptibility of the immature intestine to inflammatory diseases such as NEC.

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